Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Jonas Peper"'
Autor:
Lukas Cyganek, Henning Urlaub, Niels Voigt, Peter Rehling, David Pacheu-Grau, Bernd Wollnik, Gunnar Weninger, Julius Ryan D. Pronto, F. Seibertz, Tobias Kohl, Stephan E. Lehnart, Michael Gotthardt, Eva A. Rog-Zielinska, Daniel Kownatzki-Danger, Henry Sutanto, Jonas Peper, Gerd Hasenfuss, Sören Brandenburg, Robin Hindmarsh, Jordi Heijman, Christof Lenz, Jörg Wegener
Publikováno v:
Circulation Research, 128(6). LIPPINCOTT WILLIAMS & WILKINS
Rationale: CAV3 (caveolin3) variants associated with arrhythmogenic cardiomyopathy and muscular dystrophy can disrupt post-Golgi surface trafficking. As CAV1 (caveolin1) was recently identified in cardiomyocytes, we hypothesize that conserved isoform
Autor:
Diones Bueno, Partha Narayan Dey, Teresa Schacht, Christina Wolf, Verena Wüllner, Elena Morpurgo, Liliana Rojas-Charry, Lena Sessinghaus, Petra Leukel, Clemens Sommer, Konstantin Radyushkin, Michael K.E. Schäfer, Luise Florin, Jan Baumgart, Paul Stamm, Andreas Daiber, Guilherme Horta, Leonardo Nardi, Verica Vasic, Michael J. Schmeisser, Andrea Hellwig, Angela Oskamp, Andreas Bauer, Ruchika Anand, Andreas S. Reichert, Sandra Ritz, Gianluigi Nocera, Claire Jacob, Jonas Peper, Marion Silies, Katrin B. M. Frauenknecht, Axel Methner
Publikováno v:
bioRxiv beta (2021). doi:10.1101/2021.02.15.431234
Synaptic signaling depends on ATP generated by mitochondria. Due to extensive connectivity, the striatum is especially vulnerable to mitochondrial dysfunction and thus requires efficient mitochondrial quality control. We found that the neuronal calci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::123703cc702ef41157238c13c03b5664
https://doi.org/10.1101/2021.02.15.431234
https://doi.org/10.1101/2021.02.15.431234
Autor:
Stephan E. Lehnart, Jonas Peper, Gyuzel Yu. Mitronova, Xander H.T. Wehrens, Tobias Kohl, Eva A. Rog-Zielinska, Jan Pawlowitz, Gerd Hasenfuss, Samuel Sossalla, Benjamin Eikenbusch, Peter Kohl, Sören Brandenburg
Publikováno v:
JCI Insight. 4
Atrial dysfunction is highly prevalent and associated with increased severity of heart failure. While rapid excitation-contraction coupling depends on axial junctions in atrial myocytes, the molecular basis of atrial loss of function remains unclear.