Zobrazeno 1 - 10
of 33
pro vyhledávání: '"John, Anagli"'
Publikováno v:
Frontiers in Neurology, Vol 13 (2022)
Externí odkaz:
https://doaj.org/article/07f6b3425e4340b7841c101da541aab7
Autor:
Firas H Kobeissy, Joy D Guingab-Cagmat, Zhiqun Zhang, Ahmed Moghieb, Olena Y Glushakova, Stefania Mondello, Angela M. Boutte, John Anagli, Richard Rubenstein, Hisham Bahmad, Amy K. Wagner, Ronald L Hayes, Kevin K W Wang
Publikováno v:
Frontiers in Neurology, Vol 7 (2016)
Traumatic brain injury (TBI) represents a critical health problem of which diagnosis, management and treatment remain challenging. TBI is a contributing factor in approximately 1/3 of all injury-related deaths in the United States. The Centers for Di
Externí odkaz:
https://doaj.org/article/7dbcdf19363b4e20a290f30c44c0b66a
Autor:
Meser M Ali, Branislava Janic, Abbas Babajani-Feremi, Nadimpalli R S Varma, A S M Iskander, John Anagli, Ali S Arbab
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8727 (2010)
Anti-angiogenic treatments of malignant tumors targeting vascular endothelial growth factor receptors (VEGFR) tyrosine kinase are being used in different early stages of clinical trials. Very recently, VEGFR tyrosine kinase inhibitor (Vetanalib, PTK7
Externí odkaz:
https://doaj.org/article/fee2ee3060bc40fd8ffae2c4fdaa7731
Autor:
Firas H. Kobeissy, Ming Cheng Liu, Zhihui Yang, Zhiqun Zhang, Wenrong Zheng, Olena Glushakova, Stefania Mondello, John Anagli, Ronald L. Hayes, Kevin K. W. Wang
Publikováno v:
Molecular Neurobiology. 55:898-900
Autor:
John Anagli, Susie Zoltewicz, Changhong Ren, Xiaokun Geng, Firas Kobeissy, Joy Guingab-Cagmat, Adam Hafeez, Xunming Ji, Stefania Mondello, Mingqing Gao, Ning Li, Yuchuan Ding, Ronald L. Hayes, Stephen F. Larner
Publikováno v:
Brain Research Bulletin. 102:46-56
Intracerebral hemorrhage (ICH) is a devastating form of stroke leading to a high rate of death and disability worldwide. Although it has been hypothesized that much of the IHC insult occurs in the subacute period mediated via a series of complex path
Autor:
Firas Kobeissy, Kevin K.W. Wang, Emilio B. Cagmat, Joy Guingab-Cagmat, John Anagli, Kimberly J. Newsom, Anatoliy V Vakulenko, Susie Zoltewicz
Publikováno v:
ELECTROPHORESIS. 33:3786-3797
MS-based proteomics has been the method of choice for biomarker discovery in the field of traumatic brain injury (TBI). Due to its high sensitivity and specificity, MS is now being explored for biomarker quantitative validation in tissue and biofluid
Publikováno v:
Journal of Neurosurgery. 114:1110-1116
Object This study investigates a potential novel application of a selective cathepsin B and L inhibitor in experimental intracerebral hemorrhage (ICH) in rats. Methods Forty adult male Wistar rats received an ICH by stereotactic injection of 100 μl
Autor:
Wenrong Zheng, Olena Glushakova, Firas Kobeissy, Ronald L. Hayes, John Anagli, Zhiqun Zhang, Ming Cheng Liu, Stefania Mondello, Kevin K.W. Wang, Zhihui Yang
A major consequence of traumatic brain injury (TBI) is the rapid proteolytic degradation of structural cytoskeletal proteins. This process is largely reflected by the interruption of axonal transport as a result of extensive axonal injury leading to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27b12bcf2b60be18f90abf50fa06d175
http://hdl.handle.net/11570/2843169
http://hdl.handle.net/11570/2843169
Autor:
Michael L. Cher, Hector Biliran, Jaron Lockett, Shuping Yin, Grant E. Blouse, Neelima Reddy, Shijie Sheng, Xinli Lin, Zimin Zhao, Yonghong Meng, John Anagli, Xiaohua Li
Publikováno v:
Cancer Research. 66:4173-4181
It is well documented that tumor suppressive maspin inhibits tumor cell invasion and extracellular matrix remodeling. Maspin is a cytosolic, cell surface–associated, and secreted protein in the serine protease inhibitor superfamily. Although severa
Autor:
John Anagli, Russell L. Betts
Publikováno v:
Biochemistry. 43:2596-2604
Uncontrolled activation of calpain has been linked to tissue damage after neuronal and cardiac ischemias, traumatic spine and brain injuries, and multiple sclerosis and Alzheimer's disease. In vivo, the activity of calpain is regulated by its endogen