Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Johann‑Philipp Horstmann"'
Publikováno v:
Mediators of Inflammation, Vol 2018 (2018)
Objective. Traumatic injury or severe surgery leads to a profound immune response with a diminished functionality of monocytes and subsequently their IL-1β release. IL-1β plays an important role in host immunity and protection against infections. I
Externí odkaz:
https://doaj.org/article/3cde3cdf48344007932849e92f86b204
Publikováno v:
Mediators of Inflammation, Vol 2018 (2018)
Objective. Traumatic injury or severe surgery leads to a profound immune response with a diminished functionality of monocytes and subsequently their IL-1β release. IL-1β plays an important role in host immunity and protection against infections. I
Publikováno v:
Experientia supplementum (2012). 108
Traumatic injury as one of the world's most relevant but neglected health concerns results in modulated inflammasome activity, which is closely linked to the development of post-injury complications. Cytokine-producing capacity of cells is important
Autor:
Tamara Bonertz, Sarah von Grotthus, Albrecht Stroh, Benjamin Meyer, Gabriel Gonzalez-Escamilla, Venkata C. Chirumamilla, Sergiu Groppa, Oliver Tüscher, Johannes Vogt, Johann-Philipp Horstmann, Raffael Kalisch, Muthuraman Muthuraman
Publikováno v:
Scientific reports, 8:14506
Scientific Reports, Vol 8, Iss 1, Pp 1-12 (2018)
Scientific Reports
Scientific Reports, Vol 8, Iss 1, Pp 1-12 (2018)
Scientific Reports
Threat detection is essential for protecting individuals from adverse situations, in which a network of amygdala, limbic regions and dorsomedial prefrontal cortex (dmPFC) regions are involved in fear processing. Excitability regulation in the dmPFC m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe125b026d552278cc9a8e7c7b0100f3
https://www.nature.com/articles/s41598-018-32781-9#Sec17
https://www.nature.com/articles/s41598-018-32781-9#Sec17
Publikováno v:
Experimental and Therapeutic Medicine. 11:297-302
Prior to their release, interleukin (IL)-1β and IL-18 are cleaved to their bioactive forms by a multiprotein complex known as an inflammasome, which is comprised of a number of elements that are subject to nuclear factor-κB-dependent transcription.