Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Joel P. French"'
Publikováno v:
RUA. Repositorio Institucional de la Universidad de Alicante
Universidad de Alicante (UA)
Journal of Human Sport and Exercise, Vol 15, Iss 3, Pp 692-705 (2020)
Universidad de Alicante (UA)
Journal of Human Sport and Exercise, Vol 15, Iss 3, Pp 692-705 (2020)
Inactivity leads to morbidity and mortality, while novel and engaging approaches to fitness improve health outcomes. The current study examined an 8-week commercial group exercise regimen for high intensity interval training (HIIT) in order to examin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::57342971e9536077f7998c78d6cb9364
http://hdl.handle.net/10045/98108
http://hdl.handle.net/10045/98108
Autor:
Joel P. French, Scott K. Powers, Youngil Lee, Karyn L. Hamilton, John C. Quindry, Josh Selsby
Publikováno v:
The Journal of Physiological Sciences. 60:181-186
Increased myocardial cyclooxygenase-2 (COX-2) activity is essential for late phase ischemic preconditioning (IPC). Currently unknown is whether cardioprotection elicited by exercise also involves elevated myocardial COX-2 activity. This investigation
Autor:
Patrick A. Upchurch, Scott K. Powers, Joel P. French, Karyn L. Hamilton, John C. Quindry, Youngil Lee
Publikováno v:
The FASEB Journal. 22:2862-2871
Exercise provides protection against myocardial ischemia-reperfusion (IR) injury. Understanding the mechanisms of this protection may lead to new interventions for the prevention and/or treatment of heart disease. Although presently these mechanisms
Autor:
Scott K. Powers, Joel P. French, John C. Quindry, Darin J. Falk, Youngil Lee, Kevin K.W. Wang, Jessica L. Staib
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 290:H128-H136
The Ca2+-activated protease calpain has been shown to play a deleterious role in the heart during ischemia-reperfusion (I/R). We tested the hypothesis that exercise training would minimize I/R-induced calpain activation and provide cardioprotection a
Autor:
Jawahar L. Mehta, Jess Staib, Joel P. French, Karyn L. Hamilton, Scott K. Powers, John C. Quindry, Jeffrey A. Hughes
Publikováno v:
Free Radical Biology and Medicine. 37:1360-1368
Exercise provides protection against ischemia–reperfusion (I-R)-induced arrhythmias, myocardial stunning, and infarction. An exercise-induced increase in myocardial manganese superoxide dismutase (MnSOD) activity has been reported to be vital for p
Autor:
Scott K. Powers, Shannon L. Lennon, Joel P. French, Jessica L. Staib, Karyn L. Hamilton, Jawahar L. Mehta, John C. Quindry
Publikováno v:
Journal of Applied Physiology. 96:1299-1305
Endurance exercise provides cardioprotection against ischemia-reperfusion (I/R) injury. Exercise-induced cardioprotection is associated with increases in cytoprotective proteins, including heat shock protein 72 (HSP72) and increases in antioxidant en
There is evidence that endogenous extracellular adenosine reduces cardiac hypertrophy and heart failure in mice subjected to chronic pressure overload, but the mechanism by which adenosine exerts these protective effects is unknown. Here, we identifi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3ad4eed13e8f1f38af53108633d1ba78
https://europepmc.org/articles/PMC2724212/
https://europepmc.org/articles/PMC2724212/
Autor:
Xinli Hu, Xin Xu, Joel P. French, Guangshuo Zhu, Robert J. Bache, Elza D. van Deel, Yingjie Chen, Zhongbing Lu, Ping Zhang, John Fassett, Tim D. Oury
Publikováno v:
Hypertension, 51(1), 19-25. Lippincott Williams & Wilkins
Extracellular superoxide dismutase (SOD) contributes only a small fraction to total SOD activity in the normal heart but is strategically located to scavenge free radicals in the extracellular compartment. To examine the physiological significance of
Autor:
Youngil Lee, Karyn L. Hamilton, John C. Quindry, Zsolt Murlasits, Joel P. French, Nihal Tümer, Scott K. Powers
Publikováno v:
Journal of applied physiology (Bethesda, Md. : 1985). 103(3)
Successive bouts of endurance exercise are associated with both increased cardiac levels of heat shock protein-72 (HSP-72) and improved cardioprotection against ischemia-reperfusion (I/R)-induced cardiac cell death. Although overexpression of HSP-72
Publikováno v:
American journal of physiology. Regulatory, integrative and comparative physiology. 292(1)
The expression of myocardial heat shock protein 72 (HSP72) postexercise is initiated by the activation of heat shock transcription factor 1 (HSF1). However, it remains unknown which physiological stimuli govern myocardial HSF1 activation during exerc