Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Joe Nemoto"'
Autor:
Joe Nemoto, Hideaki Nishihara, Taro Yasuhi, Ryota Sato, Michiaki Koga, Takashi Kanda, Masayuki Nakamori
Publikováno v:
Frontiers in Neurology, Vol 14 (2023)
IntroductionAntineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) frequently leads to mononeuritis multiplex, which are characterized by distal weakness associated with sensory disturbances. Although AAV has also been reported to be
Externí odkaz:
https://doaj.org/article/bcf601b777d144e2b2be25c101856713
Publikováno v:
Internal Medicine; 2024, Vol. 63 Issue 19, p2621-2625, 5p
Publikováno v:
Internal Medicine; 2024, Vol. 63 Issue 13, p1933-1938, 6p
Autor:
Joe Nemoto, Fumitaka Shimizu, Toshihiko Maeda, Hideaki Nishihara, Michiaki Koga, Takashi Kanda
Publikováno v:
Rinsho Shinkeigaku. 63:209-213
Autor:
Joe, Nemoto, Takashi, Kanda
Publikováno v:
Brain and nerve = Shinkei kenkyu no shinpo. 74(10)
Varicella-zoster virus (VZV) is known to causes various inflammatory disorders of the central nervous system (CNS) such as encephalitis, meningitis, and myelitis. Similar to the varicella rashes, these conditions are associated with the reactivation
Autor:
Miwako Fujisawa, Yasuteru Sano, Joe Nemoto, Susumu Fujikawa, Yukio Takeshita, Shota Miyake, Kenichi Serizawa, Takashi Kanda, Fumitaka Shimizu, Kinya Matsuo, Richard M. Ransohoff, Haruna Tomizawa-Shinohara
Publikováno v:
Neurology® Neuroimmunology & Neuroinflammation
article-version (Version of Record) 3
article-version (Version of Record) 3
Background and ObjectivesTo evaluate the pathophysiology of neuromyelitis optica spectrum disorder (NMOSD) and the therapeutic mechanism and levels of interleukin-6 (IL-6) blockade (satralizumab), especially with respect to blood-brain barrier (BBB)
Autor:
Yukio Takeshita, Yasuteru Sano, Takashi Kanda, Haruna Tomizawa-Shinohara, Kinya Matsuo, Miwako Fujisawa, Fumitaka Shimizu, Richard M. Ransohoff, Shota Miyake, Susumu Fujikawa, Kenichi Serizawa, Joe Nemoto
Neuromyelitis optica spectrum disorder (NMOSD) is an autoimmune astrocytopathy caused by antibodies against the aquaporin 4(AQP4) in end-feet of astrocytes. Breakdown of the blood–brain barrier (BBB) allowing ingress of AQP4 antibodies into the cen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9750e0897fa27e2fc9ab595fb1191ec8
https://doi.org/10.1101/2021.01.28.428564
https://doi.org/10.1101/2021.01.28.428564
Autor:
Akie Yanai, Md. Nabiul Islam, Ryutaro Fujinaga, Amami Imagawa, Yukio Takeshita, Joe Nemoto, Mir Rubayet Jahan, Koh Shinoda, Greggory Wroblewski
Publikováno v:
Neuroscience. 340
Huntingtin-associated protein 1 (HAP1) is a neuronal interactor with causatively polyglutamine (polyQ)-expanded huntingtin in Huntington's disease and also associated with pathologically polyQ-expanded androgen receptor (AR) in spinobulbar muscular a