Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jochim Reinert"'
Autor:
Walter Fiedler, Stefan Schwartz, Jochim Reinert, Klaus Wethmar, Matthias Stelljes, Antonia Beitzen-Heineke
Publikováno v:
Annals of Hematology
Treatment options for relapsed or refractory B-lymphoblastic leukaemia (r/r B-ALL) are limited and the prognosis of these patients remains dismal, but novel immunotherapeutic options such as the anti-CD22 antibody–drug-conjugate Inotuzumab-Ozogamic
Autor:
Victoria Panagiota, Felicitas Thol, Jochim Reinert, Nora M Borchert, Michael Heuser, Walter Fiedler, Razif Gabdoulline, Nils W Engel
Publikováno v:
Annals of Hematology
Isolated myeloid sarcoma (MS) is a rare malignancy in which myeloid blast forms tumors at various locations while the bone marrow (BM) remains cytomorphologically free from disease. We analyzed isolated MS from four patients and their BMs at initial
Autor:
Jochim Reinert, Nils W Engel, Judith Dierlamm, Walter Fiedler, Claudia Langebrake, Caroline Krüger, Carsten Bokemeyer, Winfried H. Alsdorf
Publikováno v:
Pharmacotherapy: The Journal of Human Pharmacology and Drug Therapy. 40:479-483
Delayed methotrexate (MTX) elimination after treatment with high-dose (HD) MTX may result in life-threatening toxicities as well as acute kidney injury (AKI). Treatment includes administration of glucarpidase, an enzyme that rapidly inactivates MTX.
Autor:
Jochim Reinert, Auli Verkkoniemi-Ahola, Lars Lannfelt, Henrik Martens, Tekla Kolbow, Anders Paetau, Martin Ingelsson, Melanie Huettenrauch, Thomas A. Bayer, Oliver Wirths
Publikováno v:
Journal of Alzheimer's Disease. 39:871-881
The pathogenesis of Alzheimer's disease (AD) is believed to be closely dependent on deposits of neurotoxic amyloid-beta peptides (A beta), which become abundantly present throughout the central nervous system in advanced stages of the disease. The di
Autor:
Jochim Reinert, Richard, Bernhard, Klafki, Hans, Friedrich, Beate, Bayer, Thomas, Wiltfang, Jens, Gabor Kovacs, Ingelsson, Martin, Lannfelt, Lars, Paetau, Anders, Bergquist, Jonas, Wirths, Oliver
Deposition of C-terminally truncated Aβ species Aβ37 and Aβ39 in Alzheimer’s disease and transgenic mouse models. Figure S1. Immunohistochemical detection of Aβ37, Aβ38, Aβ39 and Aβ40 in a case with AD and CAA. While Aβ37 (a) and Aβ38 (b)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::588241683a894e0eb9f7eab106f240df