Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Joaquim Duran-Vilaregut"'
Autor:
Mercè Pallàs, J. del Valle, Gemma Manich, Carme Pelegrí, Jordi Vilaplana, Joaquim Duran-Vilaregut, Antoni Camins
Publikováno v:
Neuropathology and Applied Neurobiology. 37:525-537
J. Duran-Vilaregut, J. del Valle, G. Manich, A. Camins, M. Pallas, J. Vilaplana and C. Pelegri (2011) Neuropathology and Applied Neurobiology37, 525–537 Role of matrix metalloproteinase (MMP)-9 in striatal blood–brain barrier disruption in a 3-ni
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::acb620fd9dc7087948608baf9bd031d2
https://doi.org/10.1111/j.1365-2990.2010.01157.x
https://doi.org/10.1111/j.1365-2990.2010.01157.x
Autor:
Jaume del Valle, Antoni Camins, Jordi Vilaplana, Gemma Manich, Mercè Pallàs, Clara Mercader, Carme Pelegrí, Joaquim Duran-Vilaregut
Publikováno v:
Journal of Alzheimer's Disease. 25:535-546
The senescence accelerated mouse-prone 8 (SAMP8) strain of mice is an experimental model of accelerated senescence that has also been proposed as a model of Alzheimer's disease as it shares several features with this dementia. We have recently report
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::78221db15d9ba27d58ca47b5a752e0c5
https://doi.org/10.3233/jad-2011-101713
https://doi.org/10.3233/jad-2011-101713
Autor:
Joaquim Duran-Vilaregut, Gemma Manich, Jordi Vilaplana, Antoni Camins, Mercè Pallàs, Carme Pelegrí, Jaume del Valle
Publikováno v:
Neurodegenerative Diseases. 8:421-429
Cerebrovascular dysfunction and β-amyloid peptide deposition on the walls of cerebral blood vessels might be an early event in the development of Alzheimer’s disease. Here we studied the time course of amyloid deposition in blood vessels and blood
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5dd1c9dfea2883a2f64193537f9d586
https://doi.org/10.1159/000324757
https://doi.org/10.1159/000324757
Autor:
Joaquim Duran-Vilaregut, Jordi Vilaplana, Antoni Camins, Mercè Pallàs, Jaume del Valle, Carme Pelegrí
Publikováno v:
NeuroToxicology. 30:136-143
3-Nitropropionic acid (3-NPA) is a natural toxin that is used to induce models of Huntington's disease (HD) in experimental animals. Here we injected 3-NPA into Sprague–Dawley rats in order to evaluate its effects on the blood-brain barrier (BBB).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae806763e128270a58ccac30ba5881de
https://doi.org/10.1016/j.neuro.2008.10.007
https://doi.org/10.1016/j.neuro.2008.10.007
Autor:
Jordi Vilaplana, Mercè Pallàs, Carme Pelegrí, Joaquim Duran-Vilaregut, Gemma Manich, Itsaso Cabezón, Antoni Camins, Jaume del Valle
Publikováno v:
European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences. 49(4)
One strategy used to transport pharmacologically active substances across the blood-brain barrier (BBB) is to link the substance to a molecule capable of crossing the BBB using a receptor-mediated transcellular transport system in brain capillary end
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8396f49eb2e5da1d24168e1db071877f
https://pubmed.ncbi.nlm.nih.gov/23748097
https://pubmed.ncbi.nlm.nih.gov/23748097
Autor:
Jordi Vilaplana, Joaquim Duran-Vilaregut, Gemma Manich, Jaume del Valle, Antoni Camins, Mercè Pallàs, Carme Pelegrí
Publikováno v:
Journal of neuroscience research. 90(9)
3-Nitropropionic acid (3-NPA) is a mitochondrial toxin used in the laboratory to replicate neurodegenerative conditions that are accompanied by degeneration of the caudate-putamen. 3-NPA induces depletion in ATP production, reactive oxygen species pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::614175e9263ffb2f5651382c7a18a8d8
https://pubmed.ncbi.nlm.nih.gov/22505033
https://pubmed.ncbi.nlm.nih.gov/22505033
Autor:
Jaume del Valle, Joaquim Duran-Vilaregut, Gemma Manich, Antoni Camins, Jordi Vilaplana, Carme Pelegrí, Mercè Pallàs
Publikováno v:
Neurotoxicology. 32(6)
Although terminally differentiated neurons lack the capacity to undergo cell division, they retain the capacity to reactivate the cell cycle. This reactivation, however, has been linked to the degeneration of neurons in many experimental models of ne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99b5bec99763877a7e5038b29786ad93
https://pubmed.ncbi.nlm.nih.gov/21827787
https://pubmed.ncbi.nlm.nih.gov/21827787
Autor:
Mark A. Smith, Joaquim Duran-Vilaregut, Jordi Vilaplana, Carme Pelegrí, Antoni Camins, Jaume del Valle, Gemma Casadesus, Gemma Manich, Mercè Pallàs
Publikováno v:
Journal of Alzheimer's disease : JAD. 19(4)
Late-onset Alzheimer's disease (AD) is the most common form of AD appearing after 65 years of age. To date, however, there are no non-genetically manipulated rodent models that develop a similar sporadic onset of AD with age-related amyloid-beta (Abe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d653ad375cb7f9ebcf9b1a4492a21ffa
https://pubmed.ncbi.nlm.nih.gov/20061609
https://pubmed.ncbi.nlm.nih.gov/20061609
Autor:
Jordi Vilaplana, Jaume del Valle, Carme Pelegrí, Felix Junyent, Antoni Camins, Gemma Manich, Mercè Pallàs, Joaquim Duran-Vilaregut
Publikováno v:
Neurochemistry international. 56(3)
The intraperitoneal administration of 3-nitropropionic acid, which is commonly used to induce toxicity models of Huntington's disease in experimental animals, produces severe brain injury in the lateral part of the striatum. We studied the presence o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61e5d440092e7ceb912ba749ad87ac8f
https://pubmed.ncbi.nlm.nih.gov/19969037
https://pubmed.ncbi.nlm.nih.gov/19969037
Autor:
Joaquim Duran-Vilaregut, Jordi Vilaplana, Jaume del Valle, Carme Pelegrí, Antoni Camins, Isidre Ferrer, Mercè Pallàs, Natalia Crespo-Biel
Publikováno v:
International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. 26(7)
This study was undertaken to investigate the potential role of cell cycle re-entry in an experimental model of Huntington's disease and in human brain samples. We found that after treatment of rats with the mitochondrial neurotoxin 3-nitropropionic a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88c5f4801f917f3e74f8d818312a4ec1
https://pubmed.ncbi.nlm.nih.gov/18768156
https://pubmed.ncbi.nlm.nih.gov/18768156