Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Joanna Wisniewska-Kruk"'
Autor:
Minerva Xueting Li, Benoit Vanhollebeke, Julianna Kele, Jonas Frisén, Raoul Freitas Vale Germano, Dirk Hubmacher, Rickard Sandberg, Ralf H. Adams, Suneel S. Apte, Chika Yokota, Mike Hupe, Belma Hot, Daniel Nyqvist, Marta Trusohamn, Agnieszka Martowicz, Daniel Ramsköld, Joanna Wisniewska-Kruk, Sarantis Giatrellis, Thomas D. Arnold, Volker M. Lauschke, Lasse Jensen, Jan M. Stenman
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, 39 (7
Arteriosclerosis, Thrombosis, and Vascular Biology
Arteriosclerosis, Thrombosis, and Vascular Biology
Objective- The Wnt/β-catenin pathway orchestrates development of the blood-brain barrier, but the downstream mechanisms involved at different developmental windows and in different central nervous system (CNS) tissues have remained elusive. Approach
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::06033b6ff3ba417f3df4c5de13c061af
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/289166
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/289166
Autor:
Marta Trusohamn, Monica Corada, Julianna Kele, Daniel Nyqvist, Agnieszka Martowicz, Frank Chenfei Ning, Nina Jensen, Joanna Wisniewska-Kruk, Elisabetta Dejana
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology
Objective: Activation of endothelial β-catenin signaling by neural cell-derived Norrin or Wnt ligands is vital for the vascularization of the retina and brain. Mutations in members of the Norrin/β-catenin pathway contribute to inherited blinding di
Autor:
Anne-Eva van der Wijk, Henk A. van Veen, Ilse M. C. Vogels, Theo G. M. F. Gorgels, Cornelis J.F. Van Noorden, Ingeborg Klaassen, Joanna Wisniewska-Kruk, Danielle Versteeg, Reinier O. Schlingemann
Publikováno v:
American Journal of Pathology, 186(4), 1044-1054. Elsevier Science
American journal of pathology, 186(4), 1044-1054. Elsevier Inc.
American journal of pathology, 186(4), 1044-1054. Elsevier Inc.
Loss of blood-retinal barrier (BRB) properties induced by vascular endothelial growth factor (VEGF) and other factors is an important cause of diabetic macular edema. Previously, we found that the presence of plasmalemma vesicle associated protein (P
Autor:
Reinier O. Schlingemann, Ingeborg Klaassen, Pieter J. Gaillard, Cornelis J.F. Van Noorden, Joanna Wisniewska-Kruk, Ilse M. C. Vogels, Kees A. Hoeben
Publikováno v:
Experimental eye research, 96(1), 181-190. Academic Press Inc.
Experimental Eye Research, 96, 181-190. Academic Press
Experimental Eye Research, 96, 181-190. Academic Press
Loss of blood-retinal barrier (BRB) properties is an important feature in the pathology of diabetic macular edema (DME), but cellular mechanisms underlying BRB dysfunction are poorly understood. Therefore, we developed and characterized a novel in vi
Autor:
Cornelis J.F. Van Noorden, Ilse M. C. Vogels, Ingeborg Klaassen, Joanna Wisniewska-Kruk, Aaron L. Magno, Reinier O. Schlingemann, Chooi-May Lai, Elizabeth Rakoczy
Publikováno v:
Experimental eye research, 122, 123-131. Academic Press Inc.
Experimental Eye Research, 122, 123-31. Academic Press
Experimental Eye Research, 122, 123-31. Academic Press
The molecular mechanisms of vascular leakage in diabetic macular edema and proliferative retinopathy are poorly understood, mainly due to the lack of reliable in vivo models. The Akimba (Ins2(Akita)VEGF(+/-)) mouse model combines retinal neovasculari
Publikováno v:
Acta Ophthalmologica. 90
Purpose Loss of blood-retinal barrier (BRB) is an important cause of diabetic macular edema (DME), but cellular mechanisms underlying BRB dysfunction are poorly understood. Therefore, we developed and characterized a novel in vitro BRB model. Methods
Autor:
Wlodzimierz J. Krzyzosiak, Pawel M. Switonski, Agnieszka Mykowska, Agnieszka Fiszer, Joanna P. Wroblewska, Marta Olejniczak, Joanna Wisniewska-Kruk
Publikováno v:
BMC Molecular Biology, Vol 13, Iss 1, p 6 (2012)
BMC Molecular Biology
BMC Molecular Biology
Background RNA interference (RNAi) and antisense strategies provide experimental therapeutic agents for numerous diseases, including polyglutamine (polyQ) disorders caused by CAG repeat expansion. We compared the potential of different oligonucleotid
Autor:
Pieter J. Gaillard, Cornelis J.F. Van Noorden, Kees A. Hoeben, Reinier O. Schlingemann, Joanna Wisniewska-Kruk, Ilse M. C. Vogels, Ingeborg Klaassen
Publikováno v:
Experimental Eye Research. 138:167