Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Joan H. Brown"'
Autor:
Kirsten T. Nijholt, Pablo I. Sánchez-Aguilera, Harmen G. Booij, Silke U. Oberdorf-Maass, Martin M. Dokter, Anouk H. G. Wolters, Ben N. G. Giepmans, Wiek H. van Gilst, Joan H. Brown, Rudolf A. de Boer, Herman H. W. Silljé, B. Daan Westenbrink
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-13 (2023)
Abstract A Kinase Interacting Protein 1 (AKIP1) is a signalling adaptor that promotes physiological hypertrophy in vitro. The purpose of this study is to determine if AKIP1 promotes physiological cardiomyocyte hypertrophy in vivo. Therefore, adult ma
Externí odkaz:
https://doaj.org/article/ed562cee66ed427ca8cecb89312bca87
Publikováno v:
The FASEB Journal. 36
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0c5af284096a082158ca856115ee241c
https://doi.org/10.1096/fasebj.2022.36.s1.r5964
https://doi.org/10.1096/fasebj.2022.36.s1.r5964
Publikováno v:
Circulation Research. 121
In cardiomyocytes, signaling through RhoA is protective against ischemia/reperfusion injury. Cardioprotective receptor agonists such as sphingosine-1-phosphate (S1P) provide protection through RhoA activation. Mitochondrial fission has been suggested
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::935afdab4cad73bb7a5938e051a6bd53
https://doi.org/10.1161/res.121.suppl_1.313
https://doi.org/10.1161/res.121.suppl_1.313
Autor:
Sophie, Burel, Fabien C, Coyan, Maxime, Lorenzini, Matthew R, Meyer, Cheryl F, Lichti, Joan H, Brown, Gildas, Loussouarn, Flavien, Charpentier, Jeanne M, Nerbonne, R Reid, Townsend, Lars S, Maier, Céline, Marionneau
Publikováno v:
The Journal of biological chemistry. 292(42)
Voltage-gated Na+ (NaV) channels are key regulators of myocardial excitability, and Ca2+/calmodulin-dependent protein kinase II (CaMKII)-dependent alterations in NaV1.5 channel inactivation are emerging as a critical determinant of arrhythmias in hea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1a2be5083a89bfb50b3ad3585c3121da
https://pubmed.ncbi.nlm.nih.gov/28882890
https://pubmed.ncbi.nlm.nih.gov/28882890