Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Joakim, Sandstedt"'
Autor:
Joakim Sandstedt, Kristina Vukusic, Göran Dellgren, Anders Jeppsson, Lillemor Mattsson Hultén, Victoria Rotter Sopasakis
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-11 (2023)
Abstract Cardiovascular disease (CVD) is strongly associated with chronic low-grade inflammation, involving activated Toll-like receptors and their downstream cellular machinery. Moreover, CVD and other related inflammatory conditions are associated
Externí odkaz:
https://doaj.org/article/ab32996cf1a94398ac971ef98cba9c53
Autor:
Kristina Vukusic, Annika Thorsell, Aida Muslimovic, Marianne Jonsson, Göran Dellgren, Anders Lindahl, Joakim Sandstedt, Ola Hammarsten
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-9 (2022)
Abstract Hospitalized patients who die from Covid-19 often have pre-existing heart disease. The SARS-CoV-2 virus is dependent on the ACE2 receptor to be able to infect cells. It is possible that the strong link between cardiovascular comorbidities an
Externí odkaz:
https://doaj.org/article/b5087f009ca34570a5c092d81ac76f06
Autor:
Mikael Sandstedt, Kristina Vukusic, Benjamin Ulfenborg, Marianne Jonsson, Lillemor Mattsson Hultén, Göran Dellgren, Anders Jeppsson, Jane Synnergren, Joakim Sandstedt
Publikováno v:
PLoS ONE, Vol 17, Iss 6, p e0269985 (2022)
Cardiomyocyte proliferation has emerged as the main source of new cardiomyocytes in the adult. Progenitor cell populations may on the other hand contribute to the renewal of other cell types, including endothelial and smooth muscle cells. The phenoty
Externí odkaz:
https://doaj.org/article/72a8707a9b5d4ba3856d2f372b9b66f5
Autor:
Mikael Sandstedt, Victoria Rotter Sopasakis, Annika Lundqvist, Kristina Vukusic, Anders Oldfors, Göran Dellgren, Joakim Sandstedt, Lillemor Mattsson Hultén
Publikováno v:
PLoS ONE, Vol 13, Iss 8, p e0202693 (2018)
A common denominator for patients with heart failure is the correlation between elevated serum levels of proinflammatory cytokines and adverse clinical outcomes. Furthermore, lipoxygenase-induced inflammation is reportedly involved in the pathology o
Externí odkaz:
https://doaj.org/article/d48bc516d26248b1947994b9bbea6a75
Autor:
Annika Lundqvist, Mikael Sandstedt, Joakim Sandstedt, Ruth Wickelgren, Göran I Hansson, Anders Jeppsson, Lillemor Mattsson Hultén
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0161629 (2016)
Ischemic heart disease is a major cause of death and morbidity and the search for novel therapeutic targets is still required. We have previously shown that the enzyme arachidonate 15 lipoxygenase (ALOX15), which catalyzes the conversion of arachidon
Externí odkaz:
https://doaj.org/article/65c94e63866d4bfebcb6d06085047fe2
Autor:
Niklas Forsgard, Bertil Lindahl, Ola Hammarsten, Susanne Nyström, Joakim Sandstedt, Göran Dellgren, Aida Muslimovic, Sven-Erik Ricksten, Karin Starnberg, Vincent Fridén, Kristina Vukusic
Publikováno v:
Clinical Chemistry. 66:333-341
BackgroundAlthough cardiac troponin I (cTnI) and troponin T (cTnT) form a complex in the human myocardium and bind to thin filaments in the sarcomere, cTnI often reaches higher concentrations and returns to normal concentrations faster than cTnT in p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1a6fe51c4155bc4f0a0e5baf5847b89
https://doi.org/10.1093/clinchem/hvz003
https://doi.org/10.1093/clinchem/hvz003
Autor:
Fredrik Noborn, Christer Thomsen, Egor Vorontsov, Emanuele Bobbio, Carina Sihlbom, Jonas Nilsson, Christian L. Polte, Entela Bollano, Kristina Vukusic, Joakim Sandstedt, Göran Dellgren, Kristjan Karason, Anders Oldfors, Göran Larson
Cardiac amyloidosis is a severe condition leading to restrictive cardiomyopathy and heart failure. Mass spectrometry-based methods for cardiac amyloid subtyping have become important diagnostic tools but are currently used only in a few reference lab
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee523f12015f7f529384448d97f9e5e0
Autor:
Göran Bergström, Michaela Johansson, Lillemor Mattsson Hultén, Annika Lundqvist, Joakim Sandstedt, Victoria Rotter Sopasakis, Anders Jeppsson
Publikováno v:
International Journal of Cardiology. 293:238-247
Background A sustained low grade inflammatory state is a recognized feature of various diseases, including cardiovascular disease. This state of chronic inflammation involves activation of Toll-like receptor (TLR) signaling. However, little is known
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a7e32f32697e11573cca4064158f55b
https://doi.org/10.1016/j.ijcard.2019.06.033
https://doi.org/10.1016/j.ijcard.2019.06.033
Autor:
Victoria Rotter Sopasakis, Anders Jeppsson, Lillemor Mattsson Hultén, Joakim Sandstedt, Kristina Vukusic, Göran Dellgren, Elham Rekabdar
Publikováno v:
International journal of cardiology. 344
Background Adverse cardiac remodeling and tissue damage following heart disease is strongly associated with chronic low grade inflammation. The mechanisms underlying persisting inflammatory signals are not fully understood, but may involve defective
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae566c5d640f9c1d6047dd3a27ed86c0
https://pubmed.ncbi.nlm.nih.gov/34856296
https://pubmed.ncbi.nlm.nih.gov/34856296
Autor:
Joakim, Sandstedt, Karin, Vargmar, Kristina, Björkman, Ulla, Ruetschi, Göran, Bergström, Lillemor Mattsson, Hultén, Eva, Skiöldebrand
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology
Supplemental Digital Content is available in the text.
Objective: COMP (cartilage oligomeric matrix protein) is abundantly expressed in the cardiovascular system, cartilage, and atherosclerotic plaques. We investigated if the total COMP (COMPtot
Objective: COMP (cartilage oligomeric matrix protein) is abundantly expressed in the cardiovascular system, cartilage, and atherosclerotic plaques. We investigated if the total COMP (COMPtot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1d02bae28d250b63b317a4ad6417bbe0
https://pubmed.ncbi.nlm.nih.gov/33472398
https://pubmed.ncbi.nlm.nih.gov/33472398