Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Jo Q. Hill"'
Autor:
Alexandria P. Lassetter, Megan M. Corty, Romina Barria, Amy E. Sheehan, Jo Q. Hill, Sue A. Aicher, A. Nicole Fox, Marc R. Freeman
Publikováno v:
The Journal of cell biology. 222(1)
Maintaining long, energetically demanding axons throughout the life of an animal is a major challenge for the nervous system. Specialized glia ensheathe axons and support their function and integrity throughout life, but glial support mechanisms rema
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aea9b45173db549ed0de6275578b95e7
https://pubmed.ncbi.nlm.nih.gov/36399182
https://pubmed.ncbi.nlm.nih.gov/36399182
Autor:
Anna E. Mammel, Alec F. Condon, Katherine C. Delgado, Andrea L. Chin, Yingming Wang, Fred L. Robinson, Sue A. Aicher, Jo Q. Hill, Lev M. Fedorov
Publikováno v:
Hum Mol Genet
The form of Charcot–Marie–Tooth type 4B (CMT4B) disease caused by mutations in myotubularin-related 5 (MTMR5; also called SET binding factor 1, SBF1) shows a spectrum of axonal and demyelinating nerve phenotypes. This contrasts with the CMT4B sub
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ee911fb37a1280625faffa0a24cabf7
https://europepmc.org/articles/PMC9190308/
https://europepmc.org/articles/PMC9190308/
Autor:
Amy E. Sheehan, Megan M. Corty, Jo Q. Hill, Sue A. Aicher, Marc R. Freeman, Alexandria P. Lassetter, Romina Barria, A. Nicole Fox
Axons can represent the majority of the volume of a neuron and are energetically very demanding. Specialized glia ensheathe axons and are believed to support axon function and maintenance throughout life, but molecular details of glia-neuron support
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::77f30491146c28fcb3b62e8e5916b5e7
https://doi.org/10.1101/2021.09.02.458753
https://doi.org/10.1101/2021.09.02.458753
Autor:
Andrea L. Chin, Yingming Wang, Sue A. Aicher, Fred L. Robinson, Katherine C. Delgado, Lev M. Fedorov, Anna E. Mammel, Jo Q. Hill, Alec F. Condon
The form of Charcot-Marie-Tooth type 4B (CMT4B) disease caused by mutations in myotubularin-related 5 (MTMR5; also called SET Binding Factor 1; SBF1) shows a spectrum of axonal and demyelinating nerve phenotypes. This contrasts with the CMT4B subtype
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::722fc0b8a9779b01f2d121923805e8e6
https://doi.org/10.1101/843219
https://doi.org/10.1101/843219