Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Jiyoti Verma-Gaur"'
Autor:
Timothy M. Tucey, Jiyoti Verma-Gaur, Julie Nguyen, Victoria L. Hewitt, Tricia L. Lo, Miguel Shingu-Vazquez, Avril A. B. Robertson, James R. Hill, Filomena A. Pettolino, Travis Beddoe, Matthew A. Cooper, Thomas Naderer, Ana Traven
Publikováno v:
mSphere, Vol 1, Iss 3 (2016)
ABSTRACT The pathogenic yeast Candida albicans escapes macrophages by triggering NLRP3 inflammasome-dependent host cell death (pyroptosis). Pyroptosis is inflammatory and must be tightly regulated by host and microbe, but the mechanism is incompletel
Externí odkaz:
https://doaj.org/article/9250ecd36c7f4b649bfd1f5e5382377f
Autor:
Jiyoti Verma-Gaur, Yue Qu, Paul F Harrison, Tricia L Lo, Tara Quenault, Michael J Dagley, Matthew Bellousoff, David R Powell, Traude H Beilharz, Ana Traven
Publikováno v:
PLoS Genetics, Vol 11, Iss 10, p e1005590 (2015)
The yeast Candida albicans is a human commensal and opportunistic pathogen. Although both commensalism and pathogenesis depend on metabolic adaptation, the regulatory pathways that mediate metabolic processes in C. albicans are incompletely defined.
Externí odkaz:
https://doaj.org/article/26d369bd1bb9478b876a665e3593e08e
Autor:
Nathalie Uwamahoro, Jiyoti Verma-Gaur, Hsin-Hui Shen, Yue Qu, Rowena Lewis, Jingxiong Lu, Keith Bambery, Seth L. Masters, James E. Vince, Thomas Naderer, Ana Traven
Publikováno v:
mBio, Vol 5, Iss 2 (2014)
ABSTRACT The fungal pathogen Candida albicans causes macrophage death and escapes, but the molecular mechanisms remained unknown. Here we used live-cell imaging to monitor the interaction of C. albicans with macrophages and show that C. albicans kill
Externí odkaz:
https://doaj.org/article/563581c05a7c464a97fba2d6e6fe6809
Autor:
Ana Traven, Jiyoti Verma-Gaur
Publikováno v:
Cellular Microbiology
Summary In the human fungal pathogen Candida albicans, remodelling of gene expression drives host adaptation and virulence. Recent studies revealed that in addition to transcription, post‐transcriptional mRNA control plays important roles in virule
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f287c86b8f5599018f39c208493f5e80
http://europepmc.org/articles/PMC5074327
http://europepmc.org/articles/PMC5074327
Autor:
Stephen R Head, Nicholas J. Schork, Ali Torkamani, Lana Schaffer, Jiyoti Verma-Gaur, Ann J. Feeney
Publikováno v:
Proceedings of the National Academy of Sciences. 109:17004-17009
Noncoding sense and antisense germ-line transcription within the Ig heavy chain locus precedes V(D)J recombination and has been proposed to be associated with Igh locus accessibility, although its precise role remains elusive. However, no global anal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5cc5bd40573b9469d4a45c1ae8cda71
https://doi.org/10.1073/pnas.1208398109
https://doi.org/10.1073/pnas.1208398109
Autor:
Rabih Hassan, Winfried Edelman, Matthew D. Scharff, Ulrich Steidl, Jiyoti Verma-Gaur, Ann J. Feeney, Sabrina A. Volpi, Barbara K. Birshtein, Sanjukta Chatterjee, Zhongliang Ju, Britta Will, Harry Hou, Sergio Roa, Uwe Werling
Publikováno v:
The Journal of Immunology. 188:2556-2566
Regulatory elements located within an ∼28-kb region 3′ of the Igh gene cluster (3′ regulatory region) are required for class switch recombination and for high levels of IgH expression in plasma cells. We previously defined novel DNase I hyperse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::027550ff5093e51d36edfaaad2853c7a
https://doi.org/10.4049/jimmunol.1102763
https://doi.org/10.4049/jimmunol.1102763
Autor:
Jiyoti Verma-Gaur, Ann J. Feeney, Han-Yu Shih, Ali Torkamani, Michael S. Krangel, Niels Galjart
Publikováno v:
Proceedings of the National Academy of Sciences of the U.S.A., 109(50), E3493-E3502. National Academy of Sciences
Antigen receptor locus V(D)J recombination requires interactions between widely separated variable (V), diversity (D), and joining (J) gene segments, but the mechanisms that generate these interactions are not well understood. Here we assessed mechan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::541b394c7bb3905ceef68dc709c70a5b
https://doi.org/10.1073/pnas.1214131109
https://doi.org/10.1073/pnas.1214131109
Autor:
Ranjan Sen, Jiyoti Verma-Gaur, Changying Guo, Lindsey E. Montefiori, Amalendu Ghosh, Xiang Qiu, Nancy M. Choi, Ann J. Feeney, Tatiana I. Gerasimova
Publikováno v:
Genesdevelopment. 29(16)
Conformation of antigen receptor gene loci spatially juxtaposes rearranging gene segments in the appropriate cell lineage and developmental stage. We describe a three-step pathway that establishes the structure of the 2.8-Mb immunoglobulin heavy chai
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa45cb4a28c074f13473d2944b967fdb
https://pubmed.ncbi.nlm.nih.gov/26302788
https://pubmed.ncbi.nlm.nih.gov/26302788
Publikováno v:
The Journal of antimicrobial chemotherapy. 71(2)
OBJECTIVES Biofilm-related human infections have high mortality rates due to drug resistance. Cohabitation of diverse microbes in polymicrobial biofilms is common and these infections present additional challenges for treatment compared with monomicr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::822deec3503c7c84188e546624e35c09
https://pubmed.ncbi.nlm.nih.gov/26490013
https://pubmed.ncbi.nlm.nih.gov/26490013
Autor:
Tricia L. Lo, Traude H. Beilharz, Tara L Quenault, Matthew Bellousoff, Yue Qu, Michael J Dagley, Jiyoti Verma-Gaur, Ana Traven, David R. Powell, Paul Harrison
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 11, Iss 10, p e1005590 (2015)
PLoS Genetics, Vol 11, Iss 10, p e1005590 (2015)
The yeast Candida albicans is a human commensal and opportunistic pathogen. Although both commensalism and pathogenesis depend on metabolic adaptation, the regulatory pathways that mediate metabolic processes in C. albicans are incompletely defined.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8592cb1192ff2f4be596d5ef90f55fce
https://pubmed.ncbi.nlm.nih.gov/26474309
https://pubmed.ncbi.nlm.nih.gov/26474309