Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Jing-Ni Ou"'
Autor:
Jing-ni Ou, 歐菁妮
90
This study aims to investigate the identification of Mandarin tone and the time course of this identification process in hearing-normal and hearing-impaired children. The gating paradigm proposed by Grosjean (1980) is adopted. Three groups of
This study aims to investigate the identification of Mandarin tone and the time course of this identification process in hearing-normal and hearing-impaired children. The gating paradigm proposed by Grosjean (1980) is adopted. Three groups of
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/52221879494623197778
Autor:
Julie Wang, Jing Ni Ou, J. Dixon Gray, Song Guo Zheng, Stephanie Q. Pan, Maogen Chen, David A. Horwitz
Publikováno v:
Clinical Immunology. 149:450-463
We report that polyclonal CD8regs generated in one week ex-vivo with anti-CD3/28 beads and cytokines rapidly developed suppressive activity in vitro sustained by TGF-β. In immunodeficient mice, these CD8regs demonstrated a markedly protective, IL-10
Autor:
Vivian H. Gersuk, Susan Shenoi, Anne M. Stevens, Chester Ni, Carol A. Wallace, Jing Ni Ou, Elizabeth D. Mellins, Claudia Macaubas
Publikováno v:
Pediatric research. 78(5)
Differentiation of systemic juvenile idiopathic arthritis (SJIA) fever from other childhood fevers is often delayed due to the lack of reliable, specific biomarkers. We hypothesized that PD-L1 expression is dysregulated in SJIA monocytes and compared
Publikováno v:
Scientific Reports
Monocytes in patients with systemic lupus erythematosus (SLE) are hyperstimulatory for T lymphocytes. We previously found that the normal program for expression of a negative costimulatory molecule programmed death ligand-1 (PD-L1) is defective in SL
Autor:
Kenichi Koike, Tomonari Shigemura, Anne M. Stevens, Takashi Yamazaki, Jing Ni Ou, Yosuke Hara, Hans D. Ochs, Kazunaga Agematsu
Publikováno v:
Pediatric Rheumatology Online Journal
Pediatric Rheumatology Online Journal, Vol 9, Iss 1, p 15 (2011)
Pediatric Rheumatology Online Journal, Vol 9, Iss 1, p 15 (2011)
Systemic juvenile idiopathic arthritis (sJIA) is a systemic inflammatory disease characterized by arthritis, spiking fever and a skin rash that is frequently complicated by macrophage activation syndrome (MAS), a life-threatening disorder. We report
Publikováno v:
DNA Methylation and Cancer Therapy ISBN: 9780306478482
Regional hypermethylation and global hypomethylation coexist in cancer cells. Under-standing the mechanisms responsible for global hypomethylation and regional hypermethylation in cancer is required for the proper design of therapeutic strategies tar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::81297096e3400292c617be580d57e063
https://doi.org/10.1007/0-387-27443-x_12
https://doi.org/10.1007/0-387-27443-x_12
Autor:
Keisuke Shikimi, Jerome Torrisani, Alexander Unterberger, Jing-Ni Ou, Nadine Provencal, Mohsen Karimi, Tomas J. Ekström, Moshe Szyf
Publikováno v:
Biochemical pharmacology. 73(9)
DNA methylation and chromatin structure are two modes of epigenetic control of genome function. Although it is now well established that chromatin silencing could lead to DNA methylation, the relation between chromatin activation and DNA demethylatio
Publikováno v:
Clinical Immunology. 135:S127-S128
Autor:
Jing-Ni Ou, Anne M. Stevens
Publikováno v:
Pediatric Rheumatology Online Journal
Pediatric Rheumatology Online Journal, Vol 10, Iss Suppl 1, p A2 (2012)
Pediatric Rheumatology Online Journal, Vol 10, Iss Suppl 1, p A2 (2012)
Purpose Programmed death ligand 1 (PD-L1) plays an important role in controlling autoreactive and follicular T helper lymphocytes, which can induce autoantibody production. Properly expressed PD-L1 thus prevents autoimmune disease. Induced by inflamm
Publikováno v:
The Journal of Immunology. 182:99.15-99.15
PD-L1 is an inhibitory protein expressed on monocytes (Mo) to maintain peripheral T cell tolerance. We previously reported deficient expression of PD-L1 on Mo from patients with active SLE. To investigate the mechanisms for dysregulated PD-L1 express