Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Jing-Na Fan"'
Autor:
Xiao-Huan Liu, Jin-Ting Zhou, Chun-xia Yan, Cheng Cheng, Jing-Na Fan, Jing Xu, Qiangsun Zheng, Qiang Bai, Zongfang Li, Shengbin Li, Xiaoming Li
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
The liver immune microenvironment is a key element in the development of hepatic inflammation in NAFLD. ApoA4 deficiency increases the hepatic lipid burden, insulin resistance, and metabolic inflammation. However, the effect of ApoA4 on liver immune
Externí odkaz:
https://doaj.org/article/3748d6ce81e34f3dab0e2820ee8ca446
Autor:
Cheng Cheng, Xiao‐Huan Liu, Jing He, Jing Gao, Jin‐Ting Zhou, Jing‐Na Fan, Xi Jin, Jianbo Zhang, Liao Chang, Zijun Xiong, Jun Yu, Shengbin Li, Xiaoming Li
Publikováno v:
Molecular nutritionfood research. 66(18)
Hepatic steatosis and insulin resistance (IR) are risk factors for many metabolic syndromes such as NAFLD and T2DM. ApoA4 improves glucose hemostasis by increasing glucose-stimulated insulin secretion and glucose uptake via PI3K-Akt activation in adi
Autor:
Malgorzata A. Garstka, Jing-Na Fan, Yupeng Zhang, Zongfang Li, Xiaoming Li, Jinting Zhou, Xiao-Huan Liu, Cheng Cheng
Publikováno v:
Biochemical and Biophysical Research Communications. 534:659-665
Apolipoprotein A4 (ApoA4) regulates lipid and glucose metabolism and exerts anti-inflammatory effects in atherogenesis and colitis. The present study explored the presumed protective role of ApoA4 in carbon tetrachloride (CCl4)-induced acute liver in
Publikováno v:
Diabetes. 69
Lipogenesis and lipolysis largely determining adipose tissues and liver lipid content, and the imbalance between them contributes to fatty liver disease and metabolic syndromes. Lipogenesis and lipolysis are regulated by nutritional signals and metab