Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Jing-Guo Wei"'
Autor:
Wen-Hu Zhang, Qian Yin, Hai-Yan Nan, Guangbin Cui, Lin-Feng Yan, Jing-Guo Wei, Song Zhang, Weihua Wang
Publikováno v:
Physiological Research. :305-315
Previous studies have suggested that the Notch signaling pathway plays a very important role in the proliferation and differentiation of pulmonary microvascular endothelial cells (PMVECs). Therefore, we aimed to investigate the expression level of No
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1e1728d2589da81434166837b11692d1
https://doi.org/10.33549/physiolres.933356
https://doi.org/10.33549/physiolres.933356
Autor:
Ya-Ning Wei, Ge Zhao, Jing-Guo Wei, Lin-Feng Yan, Hai-Yan Nan, Baoying Chen, Qian Yin, Guangbin Cui, Yue Qin, Xin Zhao
Publikováno v:
Experimental and Toxicologic Pathology. 66:61-71
The pathogenesis of idiopathic pulmonary fibrosis (IPF) is not very clear, with evidence for the involvement of both inflammation and aberrant vascular remodeling (associated with angiogenesis). Pulmonary microvascular endothelial cells (PMVECs), whi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a21d5b5d58faa9c2516f928a9e19920
https://doi.org/10.1016/j.etp.2013.08.004
https://doi.org/10.1016/j.etp.2013.08.004
Autor:
Xiao-Feng Huang, Wen-Hu Zhang, Lin-Feng Yan, Hai-Yan Nan, Qian Yin, Jing-guo Wei, Guangbin Cui
Publikováno v:
Journal of Cellular Physiology. 226:2091-2102
Accumulation and activation of myofibroblasts are the hallmark of progressive pulmonary fibrosis, and the resident fibroblasts are the major source of myofibroblasts. However, the key factors involved in the transformation of fibroblasts are unknown.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::572ce17b5a849f8d75e07657c39aa60d
https://doi.org/10.1002/jcp.22545
https://doi.org/10.1002/jcp.22545
Autor:
Ya-Ning Wei, Chun-Mei Wang, Pang Du, Bao-Ying Chen, Yue-Min Wang, Xin-Jiang Wang, Sha Wang, Wei Guo, Jing-Guo Wei, Ke-Jun Ma, Hai-Dong Huang, Xiao-Yong Zhang, Guang-Bin Cui, Jian-Hong Wang, Xiao-Feng Huang
Publikováno v:
European Journal of Gastroenterology & Hepatology. 20:202-208
Background and objectives The mechanisms that trigger gallbladder evacuation dysfunction, the key risk factor for gallstone formation, have not yet been fully elucidated. The sphincter of Oddi (SO) plays important roles in the regulation of gallbladd
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c52f3c3fbf2d510af8a982a71688a5e
https://doi.org/10.1097/meg.0b013e3282f1d6ee
https://doi.org/10.1097/meg.0b013e3282f1d6ee
Autor:
Wei Wang, Pang Du, Jing-Guo Wei, Yi Wan, Sha Wang, Zhen Yan, Xiao-Yong Zhang, Wei Han, Guang-bin Cui, Ke-Jun Ma, Yingqi Zhang
Publikováno v:
Molecular Biology Reports. 35:649-655
Large-conductance, voltage-dependent and Ca(2+)-sensitive K(+) (BK) channels are composed of pore-forming alpha subunits and the modulatory beta subunits. In smooth muscle, the modulatory beta1 subunits are vital in rendering BK channels function as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79d02d19827a0cb07cb90880dd22b424
https://doi.org/10.1007/s11033-007-9135-x
https://doi.org/10.1007/s11033-007-9135-x
Publikováno v:
Acta Biochimica et Biophysica Sinica. 38:893-899
Hypercholesterolemia, which is closely related to gallbladder bile stasis, can cause sphincter of Oddi dysfunction (SOD) by increasing the tension of sphincter of Oddi (SO). Intracellular calcium ion concentration ((Ca 2+ )i) could influence the tens
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::53b1900f01f90b4f9852ac7d37e6ff47
https://doi.org/10.1111/j.1745-7270.2006.00236.x
https://doi.org/10.1111/j.1745-7270.2006.00236.x
Alteration of tight junctions in pulmonary microvascular endothelial cells in bleomycin-treated rats
Publikováno v:
Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie. 64(1-2)
Macrophages and eosinophils that infiltrate the lung interstitium are active promoters of bleomycin (BLM)-induced pulmonary fibrosis. Leukocyte infiltration indicates disrupted barrier function in endothelial cells. The aims of this study were to inv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8d79c1a932d231bb0bee63377f405d9f
https://pubmed.ncbi.nlm.nih.gov/20663652
https://pubmed.ncbi.nlm.nih.gov/20663652
Autor:
Yufeng Zhao, Bao-Ying Chen, Guang-bin Cui, Wei Wang, Miao-Zhang Zhu, Jing-Guo Wei, Xiao-Xing Zhu, Jun Yu, Wei Guo
Publikováno v:
Archives of medical research. 40(4)
Background and Aims In obesity, oxidative stress is responsible for the aberrant production of adipokines such as adiponectin, plasminogen activator inhibitor (PAI)-1 and interleukin-6 (IL-6), which is causally associated with obesity-related inflamm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d5031a2c738ceff4997143fc61f7868
https://pubmed.ncbi.nlm.nih.gov/19608012
https://pubmed.ncbi.nlm.nih.gov/19608012
Autor:
Yingqi Zhang, Yi Wan, Sha Wang, Jing-Guo Wei, Zhen Yan, Xiao-Yong Zhang, Bin Zhang, Yubo Chai, Lifeng Wang
Publikováno v:
Gene. 438(1-2)
The beta1 subunit of the voltage-dependent and Ca(2+)-activated large-conductance K(+) channel (BK) in mammalian smooth muscle cells (SMCs) plays an important role in regulating smooth muscle tone and is closely linked with a series of smooth muscle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bfd2c5ae61b056b3ed628c7a9c9bcb59
https://pubmed.ncbi.nlm.nih.gov/19303925
https://pubmed.ncbi.nlm.nih.gov/19303925
Publikováno v:
Acta biochimica et biophysica Sinica. 38(12)
Hypercholesterolemia, which is closely related to gallbladder bile stasis, can cause sphincter of Oddi dysfunction (SOD) by increasing the tension of sphincter of Oddi (SO). Intracellular calcium ion concentration ([Ca(2+)](i)) could influence the te
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c769e64e7d0c1044a7444fad7109e311
https://pubmed.ncbi.nlm.nih.gov/17151783
https://pubmed.ncbi.nlm.nih.gov/17151783