Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Jing Yan Meng"'
Autor:
Naomi Kitamura, Masashi Koono, Hiroaki Kataoka, Jing Yan Meng, Hiroshi Itoh, Ryouichi Hamasuna
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1519:92-95
Hepatocyte growth factor (HGF) activator inhibitor type 1 (HAI-1) and type 2 (HAI-2) were recently discovered as specific inhibitors of HGF activator. Each of them contains two Kunitz-type serine protease inhibitor domains and a transmembrane domain,
Publikováno v:
International Journal of Cancer. 92:31-39
Amyloid beta protein precursor (APP) is a membrane-bound protein ubiquitously expressed in a variety of types of cells. However, its biological functions remain largely uncertain, particularly in non-neural cells and tumors. Our previous studies reve
Publikováno v:
Biochemical and Biophysical Research Communications. 280:1262-1268
Previously we have demonstrated that in MDCK epithelial cells not only transforming growth factor-beta (TGF-beta) but also hepatocyte growth factor/scatter factor (HGF/SF) regulates fibronectin (FN) splicing by increasing the ratio of EDA-containing
Publikováno v:
Chinese journal of integrative medicine. 20(10)
To investigate the antiproliferative and anti-metastasis effect of Xihuang Pill (, XP) on human colorectal cancer cell and to explore the molecular mechanism by which it produces the effects.Highly metastatic human colorectal cancer cell line LoVo wa
Autor:
MIAO WANG, SU-FEI HE, LEI-LEI LIU, XIAO-XIA SUN, FAN YANG, QIAN GE, WEI-KANG WONG, JING-YAN MENG
Publikováno v:
Oncology Reports; Oct2017, Vol. 38 Issue 4, p1941-1948, 8p, 5 Graphs
Autor:
Takeshi Shimomura, Ryouichi Hamasuna, Jing-Yan Meng, Hiroshi Itoh, Hiroaki Kataoka, Tatsuo Suganuma, Masashi Koono
Publikováno v:
Histochemistry and cell biology. 114(6)
Activation of hepatocyte growth factor (HGF) is a crucial limiting step in HGF-induced signaling pathway. The HGF activator inhibitor type 1 (HAI-1) was identified as a potent inhibitor of HGF activator (HGFA), a serine proteinase that is responsible