Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Jin-xu Zheng"'
Publikováno v:
Molecular Medicine Reports. 2015, Vol. 11 Issue 4, p2703-2710. 8p.
Autor:
PING SONG1, JIN-XU ZHENG1 jinxuzheng@126.com, JI-ZHU LIU2, JIAO XU1, LI-YAN WU1, CHAO LIU1, QIN ZHU1, YANG WANG1
Publikováno v:
Molecular Medicine Reports. 2014, Vol. 10 Issue 2, p1030-1036. 7p.
Publikováno v:
Tropical Journal of Pharmaceutical Research; Vol 16, No 3 (2017); 581-588
Purpose : To investigate the therapeutic effects of saikosapoin D (SSD) on bleomycin (BLM)-induced pulmonary fibrosis (PF) in mice and its probable mechanisms. Methods : PF mice were prepared by intraperitoneal (i.p.) injection of BLM (5 mg/kg). Twen
Publikováno v:
Molecular medicine reports. 11(4)
Idiopathic pulmonary fibrosis (IPF) is a fatal lung disease characterized by expansion of the fibroblast and myofibroblast population and extracellular matrix deposition. Although the pathogenic mechanisms of IPF remain to be fully elucidated, there
Publikováno v:
Chinese medical journal. 125(15)
Epithelial-mesenchymal transition is a cellular process characterized by the loss of cell adhesion, inhibition of E-cadherin expression, and increased cell mobility. Cells without Napsin A are susceptible to transition. Further studies are required t
Publikováno v:
Zhonghua yi xue za zhi. 90(46)
To study the in vitro effect and mechanism of Napsin A gene transfection into type II alveolar epithelial cells on pulmonary fibrosis.A recombinant lentiviral plasmid PLJM1-Napsin A was constructed and transfected into human type II alveolar epitheli
Publikováno v:
Zhonghua yi xue za zhi. 90(12)
To study the therapeutic effects and mechanism of saikosaponin-d (SSd) in mice with bleomycin (BLM)-induced pulmonary fibrosis.According to the random number table, 180 mice were randomly divided into 5 groups. Four groups were pulmonary fibrosis mod
Publikováno v:
Xi bao yu fen zi mian yi xue za zhi = Chinese journal of cellular and molecular immunology. 24(10)
To construct the eukaryotic expressing vector PCAGG -HuIFN-lambda1 and PCAGG-HuIFN-lambda2 and to study the biological activity of HuIFN-lambda1and HuIFN-lambda2.The cDNA fragment encodding HuIFN-lambda1 and HuIFN-lambda2 was amplified from the total
AIM: To determine whether Helicobacter pylori (H pylori) vacuolating cytotoxin (VacA) regulates release of pro-inflammatory cytokines (IL-1β, IL-8, TNF-α, and IL-6) or alters gastric epithelial cell viability and to determine whether NaCl affects t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::94a483e8e78bee9899658725eaac1f01
https://europepmc.org/articles/PMC4087642/
https://europepmc.org/articles/PMC4087642/
Autor:
Zhi-xin Yu, Mu-sen Ji, Jun Yan, Yan Cai, Jing Liu, Hong-feng Yang, Yong Li, Zhao-chen Jin, Jin-xu Zheng
Publikováno v:
Critical Care; 2015, Vol. 19 Issue 1, p1-11, 11p