Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jiefen Lin"'
Publikováno v:
Frontiers in Neurology, Vol 12 (2021)
Background and Objective: The abnormal T1-weighted imaging of MRI can be used to characterize neonatal acute bilirubin encephalopathy (ABE) in newborns, but has limited use in evaluating the severity and prognosis of ABE. This study aims to assess th
Externí odkaz:
https://doaj.org/article/a68c883cc3b040b2aebfd17d6f717e06
Autor:
Peidong Chen, Zhiwei Shen, Qianqian Wang, Bingna Zhang, Zerui Zhuang, Jiefen Lin, Yuanyu Shen, Yanzhi Chen, Zhuozhi Dai, Renhua Wu
Publikováno v:
Frontiers in Aging Neuroscience, Vol 13 (2021)
A correlation between the abnormal cerebral glucose metabolism and the progression of Alzheimer’s disease (AD) has been found in previous studies, suggesting that glucose alterations may be used to predict the histopathological diagnosis in AD. In
Externí odkaz:
https://doaj.org/article/c8906f9c9f954819880626d9ca0d6c98
Autor:
Tianyu Luo, Xiaoyi Zhou, Minyan Qin, Yuqing Lin, Jiefen Lin, Guangpei Chen, Aijun Liu, Dongyun Ouyang, Dongfeng Chen, Hao Pan
Publikováno v:
Oxidative Medicine and Cellular Longevity.
Corilagin, a gallotannin, shows excellent antioxidant and anti-inflammatory effects. The NLRP3 inflammasome dysfunction has been implicated in a variety of inflammation diseases. However, it remains unclear how corilagin regulates the NLRP3 inflammas
Publikováno v:
Frontiers in Neurology
Frontiers in Neurology, Vol 12 (2021)
Frontiers in Neurology, Vol 12 (2021)
Background and Objective: The abnormal T1-weighted imaging of MRI can be used to characterize neonatal acute bilirubin encephalopathy (ABE) in newborns, but has limited use in evaluating the severity and prognosis of ABE. This study aims to assess th
Autor:
Qianqian Wang, Zhuozhi Dai, Bingna Zhang, Yuanyu Shen, Zhiwei Shen, Renhua Wu, Peidong Chen, Zerui Zhuang, Yanzhi Chen, Jiefen Lin
Publikováno v:
Frontiers in Aging Neuroscience
Frontiers in Aging Neuroscience, Vol 13 (2021)
Frontiers in Aging Neuroscience, Vol 13 (2021)
A correlation between the abnormal cerebral glucose metabolism and the progression of Alzheimer’s disease (AD) has been found in previous studies, suggesting that glucose alterations may be used to predict the histopathological diagnosis in AD. In