Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Jianming Lee"'
Autor:
Xiao Cui, Tingyan Mi, Hong Zhang, Ping Gao, Xue Xiao, Jianming Lee, Guelakis, Marian, Xuelan Gu
Publikováno v:
Journal of the European Academy of Dermatology & Venereology; 2024 Supplement, Vol. 38, p12-20, 9p
Autor:
Xiao Cui, Tingyan Mi, Xue Xiao, Hong Zhang, Yiying Dong, Nan Huang, Ping Gao, Jianming Lee, Guelakis, Marian, Xuelan Gu
Publikováno v:
Journal of the European Academy of Dermatology & Venereology; 2024 Supplement, Vol. 38, p3-11, 9p
Autor:
Luis A. Garza, Mary Sheu, Noori Kim, Jerry Tsai, Sabrina S. Alessi Cesar, Jianming Lee, Stacy S. Hawkins, Anna L. Chien, Sewon Kang
Publikováno v:
J Invest Dermatol
Laser resurfacing treatments for photoaged skin have improved dramatically over the past decades, but few studies have examined the molecular mechanisms underlying differences in clinical response. Seventeen white female participants with moderate-to
Publikováno v:
International journal of cosmetic science. 39(6)
Objective To evaluate a combination of retinyl propionate and climbazole (RPC) compared to 0.1% retinol for its efficacy, tolerance and ageing appearance. Method Forty-five healthy Caucasian females, ages 40–70, with moderately photodamaged facial
Publikováno v:
International journal of cosmetic science. 39(4)
Objective To explore if climbazole enhances retinoid-associated biological activities in vitro and in vivo. Methods Primary human dermal fibroblasts (HDFs) were treated from six to 48 hours with either retinoids (retinol, retinyl propionate, retinyl
Publikováno v:
Journal of Investigative Dermatology. 132:2376-2385
It is well known that profilaggrin, after its release from keratohyalin granules through dephosphorylation, becomes enzymatically processed into individual filaggrin monomers. The roles for filaggrin monomers in aggregating keratin filaments, as a co
Publikováno v:
The Journal of Investigative Dermatology
TO THE EDITOR Senescence is a state wherein cells are metabolically active but are unable to replicate due to the increased expression of cell cycle check point proteins, such as p16INK4A, that prevent passage of cells through the cycle. p16INK4A exp