Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Jian-Long Miao"'
Publikováno v:
Archives of Medical Science, Vol 16, Iss 1, Pp 16-26 (2019)
Introduction Identifying target oncogenic alterations in lung cancer represents a major development in disease management. We examined the association of fibroblast growth factor receptor 1 (FGFR1) gene amplification with pathological characteristics
Externí odkaz:
https://doaj.org/article/c40562fc219545e3a48d1209b6cabfc4
Publikováno v:
Chinese Medical Journal, Vol 129, Iss 23, Pp 2868-2872 (2016)
Objective: To review the prevalence and prognostic significance of fibroblast growth factor receptor 1 (FGFR1) amplification and to establish an association between FGFR1 amplification and the clinical characteristics of nonsmall cell lung cancer (NS
Externí odkaz:
https://doaj.org/article/224b8f3d11e14ec89dbf927cba4813e5
Autor:
Jian-Long Miao1, Jin-Hua Zhou1, Jing-Jing Cai1, Rui-Juan Liu1 mqb_6@163.com, Miao, Jian-Long1 (AUTHOR), Zhou, Jin-Hua1 (AUTHOR), Cai, Jing-Jing1 (AUTHOR), Liu, Rui-Juan1 (AUTHOR)
Publikováno v:
Archives of Medical Science. 2020, Vol. 16 Issue 1, p16-26. 11p.
Publikováno v:
Chinese Medical Journal, Vol 129, Iss 23, Pp 2868-2872 (2016)
Chinese Medical Journal
Chinese Medical Journal
Objective: To review the prevalence and prognostic significance of fibroblast growth factor receptor 1 (FGFR1) amplification and to establish an association between FGFR1 amplification and the clinical characteristics of nonsmall cell lung cancer (NS
Publikováno v:
BioMed Research International, Vol 2018 (2018)
BioMed Research International
BioMed Research International
Objective. To investigate the clinicopathological characteristics and risk factors in patients with lung cancer and COPD. Materials and Methods. We retrospectively reviewed the clinical data of 282 patients with lung cancer, including 174 and 108 pat
Publikováno v:
Tumor Biology. 37:291-298
Previous studies demonstrate that microRNA-138 (miR-138) is critical in non-small cell lung cancer (NSCLC) regulation. We further explored the molecular mechanism of miR-138 in NSCLC. Lentivirus was used to upregulate miR-138 in NSCLC cell lines H460