Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Jian Rong Sheng"'
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
The IG20 gene undergoes alternative splicing resulting in the differential expression of six putative splice variants. Four of these (IG20pa, MADD, IG20-SV2, and DENN-SV) are expressed in virtually all human tissues. However, investigations examining
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::39e33a568a863e83e261e4b8132f978b
https://doi.org/10.1158/0008-5472.c.6497570
https://doi.org/10.1158/0008-5472.c.6497570
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Figure 3 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00cafff4ac316215c2cf70df6f165a31
https://doi.org/10.1158/0008-5472.22374773
https://doi.org/10.1158/0008-5472.22374773
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Figure 1 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::280991d58721b93a22fd8512e34e853d
https://doi.org/10.1158/0008-5472.22374779
https://doi.org/10.1158/0008-5472.22374779
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Legends 1-5 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f3710ba87c13cb71efcac01622a0be9
https://doi.org/10.1158/0008-5472.22374764.v1
https://doi.org/10.1158/0008-5472.22374764.v1
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Figure 4 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8049131232eb9b728282bc2417f366be
https://doi.org/10.1158/0008-5472.22374770
https://doi.org/10.1158/0008-5472.22374770
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Figure 2 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::033ba87c1920fa0639de7ac8327183a5
https://doi.org/10.1158/0008-5472.22374776.v1
https://doi.org/10.1158/0008-5472.22374776.v1
Autor:
Matthew N. Meriggioli, Bellur S. Prabhakar, Nirupama Mulherkar, Jian Rong Sheng, Liang Cheng Li
Supplementary Figure 5 from Regulation of Apoptosis and Caspase-8 Expression in Neuroblastoma Cells by Isoforms of the IG20 Gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f0ca6dcfe1de277047ca45767df9cb7
https://doi.org/10.1158/0008-5472.22374767.v1
https://doi.org/10.1158/0008-5472.22374767.v1
Publikováno v:
Journal of Neuroimmunology. 297:38-45
Regulatory B cells (Bregs) attenuate the severity of experimental autoimmune myasthenia gravis (EAMG) in an interleukin-10 (IL-10)-dependent manner. The goal of this study was to investigate the role of human Bregs in MG focusing on CD19(+)CD1d(hi) C
Publikováno v:
Clinical and Experimental Immunology. 184:50-61
Summary B7-2–/– non-obese diabetic (NOD) mice develop a spontaneous autoimmune polyneuropathy (SAP) that mimics the progressive form of chronic inflammatory demyelinating polyradiculoneuropathy (CIDP). In this study, we focused on the role of reg
Recombinant IgG2a Fc (M045) multimers effectively suppress experimental autoimmune myasthenia gravis
Autor:
Jian Rong Sheng, Liang Cheng Li, Bellur S. Prabhakar, Muthusamy Thiruppathi, Matthew N. Meriggioli
Publikováno v:
Journal of Autoimmunity. 52:64-73
Myasthenia gravis (MG) is an autoimmune disorder caused by target-specific pathogenic antibodies directed toward postsynaptic neuromuscular junction (NMJ) proteins, most commonly the skeletal muscle nicotinic acetylcholine receptor (AChR). In MG, hig