Zobrazeno 1 - 10
of 44
pro vyhledávání: '"Jia-xin Xing"'
Publikováno v:
BMC Psychiatry, Vol 20, Iss 1, Pp 1-10 (2020)
Abstract Background The 5-hydroxytryptamine 1B receptor (5-HT1B) plays an essential role in the serotonin (5-HT) system and is widely involved in a variety of brain activities. HTR1B is the gene encoding 5-HT1B. Genome-wide association studies have s
Externí odkaz:
https://doaj.org/article/3b60c86bc71d49e5b9e3689c0251d37d
Publikováno v:
BMC Genetics, Vol 21, Iss 1, Pp 1-8 (2020)
Abstract Background The HTR1B gene encodes the 5-hydroxytryptamine (5-HT1B) receptor, which is involved in a variety of brain activities and mental disorders. The regulatory effects of non-coding regions on genomic DNA are one of many reasons for the
Externí odkaz:
https://doaj.org/article/9286bbda86ab42058ced89149fba77ed
Autor:
Yong-ping Liu, Mei Ding, Xi-cen Zhang, Yi Liu, Jin-feng Xuan, Jia-xin Xing, Xi Xia, Jun Yao, Bao-jie Wang
Publikováno v:
BMC Medical Genetics, Vol 20, Iss 1, Pp 1-7 (2019)
Abstract Background Schizophrenia is a severe neurodevelopmental disorder with a complex genetic and environmental etiology. Abnormal glutamate ionotropic N-methyl-D-aspartate receptor (NMDA) type subunit 1 (NR1) may be a potential cause of schizophr
Externí odkaz:
https://doaj.org/article/21eaef9f44d747669452b6dae9d7bcd4
Publikováno v:
BMC Psychiatry, Vol 18, Iss 1, Pp 1-11 (2018)
Abstract Background Schizophrenia is associated with multiple neurotransmitter disorders, including serotonin (5-hydroxytryptamine, 5-HT). The neuromodulatory action of serotonin on brain function largely depends on the action of specific subtypes of
Externí odkaz:
https://doaj.org/article/2fb50f372f8e40f49a87285222439da3
Autor:
Feng-Ling Xu, Mei Ding, Jun Yao, Zhang-Sen Shi, Xue Wu, Jing-Jing Zhang, Hao Pang, Jia-Xin Xing, Jin-Feng Xuan, Bao-Jie Wang
Publikováno v:
PLoS ONE, Vol 12, Iss 8, p e0182769 (2017)
To determine whether mitochondrial DNA (mtDNA) variations are associated with schizophrenia, 313 patients with schizophrenia and 326 unaffected participants of the northern Chinese Han population were included in a prospective study. Single-nucleotid
Externí odkaz:
https://doaj.org/article/b599d0c636624b5e963e62e37d899307
Autor:
Jia-xin Xing, Bao-jie Wang, Yong-ping Liu, Feng-ling Xu, Ya Li, Meng Gao, Kuo Zeng, Jin-feng Xuan, Jun Yao, Xi Xia
Publikováno v:
Neuropsychiatric Disease and Treatment. 16:1519-1532
Background Epidemiological studies have shown that genetic factors are among the causes of schizophrenia. Galanin receptor 1 is an inhibitory receptor of galanin that is widely distributed in the central nervous system. This study mainly explored the
Autor:
Xue Wu, Feng-ling Xu, Jun Yao, Jia-xin Xing, Xi Xia, Bao-jie Wang, Yong-ping Liu, Jin-feng Xuan
Publikováno v:
Neuropsychiatric Disease and Treatment. 16:985-992
Background Abnormal RGS4 gene expression may cause neurotransmitter disorders, resulting in schizophrenia. The association between RGS4 and the risk of schizophrenia is controversial, and there has been little research on the SNPs in the promoter reg
Publikováno v:
Forensic Science International: Genetics Supplement Series. 7:823-825
In forensic medicine, mixed stains usually include body fluids, secretions of two or more individuals. The application of STR profiling of single cell capture or mixed samples requires that the existence of intact nucleated cells or the appropriate p
Autor:
Jia-xin Xing, Jun Yao, Jin-feng Xuan, Xi Xia, Jing-hua Meng, Xue Wu, Bao-jie Wang, Yong-ping Liu
Publikováno v:
Gene. 808
Introduction Abnormal expression of ionotropic glutamate receptor NMDA type subunit 1, the key subunit of the NMDA receptor, may be related to many neuropsychiatric disorders. In this study, we explored the functional sequence of the 5′ regulatory
Background: In order to detect function of three SNPs (rs12041948, rs6678136 and rs7515900) in 5′ regulatory region of the human RGS4 gene, fragments of 5′ regulatory region of RGS4 (-1112–+365, TSS+1) was cloned into pGL3-Basic vector, and dua
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::78b6555d56a492c21ff4681cb4d6bd97
https://doi.org/10.21203/rs.3.rs-147918/v1
https://doi.org/10.21203/rs.3.rs-147918/v1