Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Ji Y. Jiang"'
Autor:
Thomas M. Reeves, Robert J. Hamm, Ji Y. Jiang, Bruce G. Lyeth, John T. Povlishock, Linda L. Phillips
Publikováno v:
Journal of Neuroscience Research. 49:197-206
We have used an animal model of traumatic brain injury (TBI) that incorporates both the neurotransmitter toxicity of fluid percussion TBI and deafferentation of bilateral entorhinal cortical (BEC) lesion to explore whether administration of muscarini
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b4db1cb79070fbdf5beb68bf10148f19
https://doi.org/10.1002/(sici)1097-4547(19970715)49:2<197::aid-jnr8>3.0.co
https://doi.org/10.1002/(sici)1097-4547(19970715)49:2<197::aid-jnr8>3.0.co
Publikováno v:
Brain Research. 640:240-245
Recent evidence suggests that excessive activation of muscarinic cholinergic receptors (mAChRs) contributes significantly to the pathophysiological consequences of traumatic brain injury (TBI). To examine possible alterations in mAChRs after TBI, the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d623a5e99163a1eec1e7d9a6504c9231
https://doi.org/10.1016/0006-8993(94)91879-1
https://doi.org/10.1016/0006-8993(94)91879-1
Autor:
Ronald L. Hayes, Robert J. Hamm, Bruce G. Lyeth, Larry W. Jenkins, Guy L. Clifton, Ji Y. Jiang
Publikováno v:
Journal of Neurosurgery. 74:492-496
Recent work has shown that mild to moderate levels of hypothermia may profoundly reduce the histological and biochemical sequelae of cerebral ischemic injury. In the present study, the authors examined the effect of fluid-percussion injury on brain t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7bc94e63de23af67dbc02e3a1190a53
https://doi.org/10.3171/jns.1991.74.3.0492
https://doi.org/10.3171/jns.1991.74.3.0492
Publikováno v:
Journal of Neurotrauma. 27:775-776
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b4cf928338c1dadca30015cf6b8786bd
https://doi.org/10.1089/neu.2009.1126
https://doi.org/10.1089/neu.2009.1126
Publikováno v:
Journal of neurotrauma. 12(6)
We have identified alterations in the responses of muscarinic and metabotropic receptors in rat hippocampus that persist for at least 15 days after central fluid percussion injury. This study compares the effect of lateral fluid percussion and centra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96b5306e6816d0abea2623f3362b6641
https://pubmed.ncbi.nlm.nih.gov/8742133
https://pubmed.ncbi.nlm.nih.gov/8742133
Publikováno v:
Neuropeptides. 29(1)
We examined the effects of an exogenous mu opioid agonist and antagonist on systemic physiology and neurological outcome following TBI in the rat. Experiment I: [D-Ala2,N-Me-Phe4,Gly5-ol]-enkephalin (DAMGO) (0.1 nMol or 0.3 nMol in 5 microliters) (n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac6e3699a647f71b3ee0d32e3405c0f4
https://pubmed.ncbi.nlm.nih.gov/7566508
https://pubmed.ncbi.nlm.nih.gov/7566508
Publikováno v:
Neurochemical research. 20(4)
In the fluid percussion model of traumatic brain injury (TBI), we examined muscarinic and metabotropic glutamate receptor-stimulated polyphosphoinositide (PPI) turnover in rat hippocampus. Moderate injury was obtained by displacement and deformation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21cb7490bda447e0d30965759a9525a4
https://pubmed.ncbi.nlm.nih.gov/7651577
https://pubmed.ncbi.nlm.nih.gov/7651577
Publikováno v:
Brain research. 651(1-2)
Laboratory studies indicate that activation of muscarinic cholinergic receptors (mAChRs) at or soon after traumatic brain injury (TBI) significantly contributes to behavioral morbidity. Recent research has demonstrated that pre-injury treatment with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33f8875e8925a76d6b206e3c7bd526be
https://pubmed.ncbi.nlm.nih.gov/7922558
https://pubmed.ncbi.nlm.nih.gov/7922558
Publikováno v:
Molecular and chemical neuropathology. 18(3)
Activation of muscarinic acetylcholine (ACh) receptors contributes to the pathophysiological consequences of moderate experimental traumatic brain injury (TBI). Hypothermia (30 degrees C) provides protection in experimental TBI. We measured ACh level
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a9923a0e3f86803d26eadbeff17fe50
https://pubmed.ncbi.nlm.nih.gov/8507303
https://pubmed.ncbi.nlm.nih.gov/8507303
Autor:
Bruce G. Lyeth, Ji Y. Jiang
Publikováno v:
The Role of Neurotransmitters in Brain Injury ISBN: 9781461365280
Neurochemical alterations related to abnormal agonistreceptor interactions mediate important components of brain pathophysiology associated with TBI (Hayes et al., 1986, in press) The mechanical forces applied to the brain by a traumatic insult may i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1c9fae3936ad0af940e5dbe3e5ed4684
https://doi.org/10.1007/978-1-4615-3452-5_38
https://doi.org/10.1007/978-1-4615-3452-5_38
