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Autor:
Ian F. G. King, Michael L. Wallace, Michael S. Sidorov, Benjamin D. Philpot, Mark J. Zylka, Ype Elgersma, Bin Gu, Geeske M. van Woerden, Richard J. Weinberg, Alain C. Burette, Ji Eun E. Han, Matthew C. Judson
Publikováno v:
Neuron, 90(1), 56-69. Cell Press
Loss of maternal UBE3A causes Angelman syndrome (AS), a neurodevelopmental disorder associated with severe epilepsy. We previously implicated GABAergic deficits onto layer (L) 2/3 pyramidal neurons in the pathogenesis of neocortical hyperexcitability