Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Jevi Septyani, Latief"'
Publikováno v:
Stem Cell Research & Therapy, Vol 12, Iss 1, Pp 1-14 (2021)
Abstract Background In spite of clinical progress, cardiovascular disease (CVD) remains the predominant cause of mortality worldwide. Overexpression studies in animals have proven miR-424-5p to have anti-angiogenic properties. As type 1 diabetes mell
Externí odkaz:
https://doaj.org/article/75373b152ef8406faf625e47b6edd4d9
Publikováno v:
Journal of Translational Medicine, Vol 19, Iss 1, Pp 1-12 (2021)
Abstract Background Type 1 diabetes (T1DM) is associated with premature cardiovascular disease (CVD) and a pro-inflammatory state whilst the proangiogenic miR-126-3p/-5p may play a role in CVD. Animal studies established miR-126 to be pro-angiogenic.
Externí odkaz:
https://doaj.org/article/233bbf10098b47bc8c6e37ee3fad737f
Publikováno v:
Stem Cell Research & Therapy, Vol 12, Iss 1, Pp 1-14 (2021)
Stem Cell Research & Therapy
Stem Cell Research & Therapy
Background In spite of clinical progress, cardiovascular disease (CVD) remains the predominant cause of mortality worldwide. Overexpression studies in animals have proven miR-424-5p to have anti-angiogenic properties. As type 1 diabetes mellitus (T1D
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::83d97288c53ba3521f5272f27b9f416f
https://doi.org/10.1186/s13287-021-02332-7
https://doi.org/10.1186/s13287-021-02332-7
Publikováno v:
Journal of Translational Medicine
Journal of Translational Medicine, Vol 19, Iss 1, Pp 1-12 (2021)
Journal of Translational Medicine, Vol 19, Iss 1, Pp 1-12 (2021)
Background Type 1 diabetes (T1DM) is associated with premature cardiovascular disease (CVD) and a pro-inflammatory state whilst the proangiogenic miR-126-3p/-5p may play a role in CVD. Animal studies established miR-126 to be pro-angiogenic. We hypot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50eef287620fccc45ffa6b79eebfca11
https://doi.org/10.1186/s12967-021-02785-7
https://doi.org/10.1186/s12967-021-02785-7
Autor:
Alice Tamara, Sabina L Ray, Jolanta U. Weaver, Sherin Bakhashab, Megan Li Yuen Yeoh, Jevi Septyani Latief, David J. Coulson
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 7217, p 7217 (2020)
International Journal of Molecular Sciences
Volume 21
Issue 19
International Journal of Molecular Sciences
Volume 21
Issue 19
Cardiovascular disease (CVD) correlates with inflammation and a reduction in circulating endothelial progenitor cells (cEPCs). Recently, CVD was shown to be the main cause of mortality in individuals with type 1 diabetes (T1DM). In animals, miR-342 w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4e19921715c8fb0212cd5a3334415438
https://www.mdpi.com/1422-0067/21/19/7217
https://www.mdpi.com/1422-0067/21/19/7217
Autor:
Sabina L, Ray, David J, Coulson, Megan Li Yuen, Yeoh, Alice, Tamara, Jevi Septyani, Latief, Sherin, Bakhashab, Jolanta U, Weaver
Publikováno v:
International Journal of Molecular Sciences
Cardiovascular disease (CVD) correlates with inflammation and a reduction in circulating endothelial progenitor cells (cEPCs). Recently, CVD was shown to be the main cause of mortality in individuals with type 1 diabetes (T1DM). In animals, miR-342 w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e91d36c5de2ea5e2e490cf3213bff147
https://pubmed.ncbi.nlm.nih.gov/33003647
https://pubmed.ncbi.nlm.nih.gov/33003647
Autor:
Megan Ly Yeoh, Alice Tamara, Sabina L Ray, David J. Coulson, Sherin Bakhashab, Jevi Septyani Latief, Jolanta U. Weaver
BACKGROUND Cardiovascular disease (CVD), the leading causes of death worldwide, correlates with inflammation and shortens life span in type 1 diabetes (T1DM). In animals, miR-342 were shown to be anti-inflammatory in CVD. We hypothesise: proangiogeni
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f069ffb64d08a23c801304f9f272191
https://doi.org/10.21203/rs.3.rs-31267/v2
https://doi.org/10.21203/rs.3.rs-31267/v2
