Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jessica Hadwin"'
Autor:
Angela Kalil, Pat Tanzi, Anna V. Savos, Kevin C. Cain, J. Michael Gee, Jessica Hadwin, Kelly T. Carter, Dean Shibata, Dannielle Zierath, Kyra J. Becker
Publikováno v:
Neurocritical Care. 14:244-252
Infection is common following stroke and is independently associated with worse outcome. Clinical studies suggest that infections occur more frequently in those individuals with stroke-induced immunologic dysfunction. This study sought to explore the
Autor:
Kyra J. Becker, Kelly T. Carter, Dannielle Zierath, Allison Kunze, Anna V. Savos, Jessica Hadwin
Publikováno v:
Stroke. 41:2653-2660
Background and Purpose— Anamnestic recall of stroke-related deficits is a common clinical observation, especially during periods of systemic infection. The pathophysiology of this transient re-emergence of neurological dysfunction is unknown. Metho
Autor:
Jessica Hadwin, J. Michael Gee, Anna V. Savos, Matthew Thullbery, Dannielle Zierath, Angela Kalil, Kyra J. Becker
Publikováno v:
Neurocritical Care. 12:274-284
Animals subjected to an inflammatory insult with lipopolysaccharide (LPS) at the time of stroke are predisposed to develop a detrimental autoimmune response to myelin basic protein (MBP). In this study, we sought to determine whether other inflammato
Autor:
Kevin C. Cain, J. Michael Gee, Dannielle Zierath, Dean Shibata, Angela Kalil, Kelly T. Carter, Jessica Hadwin, Kyra J. Becker, Pat Tanzi, Anna V. Savos
Publikováno v:
Stroke. 42(10)
Background and Purpose— Immune responses to brain antigens occur after stroke, and experimental studies show that the likelihood of developing a detrimental autoimmune response to these antigens is increased by systemic inflammation at the time of
Autor:
J. Michael Gee, Angela Kalil, Anna V. Savos, Matthew Thullbery, Jessica Hadwin, Kyra J. Becker, Dannielle Zierath
Publikováno v:
Experimental & Translational Stroke Medicine
BackgroundAn inflammatory insult following middle cerebral artery occlusion (MCAO) is associated with a predisposition to develop a deleterious autoimmune response to the brain antigen myelin basic protein (MBP). Induction of immunologic tolerance to