Zobrazeno 1 - 10 of 13 pro vyhledávání: '"Jessica González‐Sánchez"'
1
Akademický článek
Obestatin signalling counteracts glucocorticoid‐induced skeletal muscle atrophy via NEDD4/KLF15 axis
Autor: Tania Cid‐Díaz, Saúl Leal‐López, Fátima Fernández‐Barreiro, Jessica González‐Sánchez, Icía Santos‐Zas, Luis J. Andrade‐Bulos, Manuel E. Rodríguez‐Fuentes, Carlos S. Mosteiro, Vincent Mouly, Xesús Casabiell, Jose Luis Relova, Yolanda Pazos, Jesus P. Camiña
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle, Vol 12, Iss 2, Pp 493-505 (2021)
Abstract Background A therapeutic approach for the treatment of glucocorticoid‐induced skeletal muscle atrophy should be based on the knowledge of the molecular mechanisms determining the unbalance between anabolic and catabolic processes and how t
Externí odkaz: https://doaj.org/article/6aba5e89c5064f319ade19594e974fcf
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2
Akademický článek
Improvement of Duchenne muscular dystrophy phenotype following obestatin treatment
Autor: Jessica González‐Sánchez, Agustín Sánchez‐Temprano, Tania Cid‐Díaz, Regina Pabst‐Fernández, Carlos S. Mosteiro, Rosalía Gallego, Ruben Nogueiras, Xesús Casabiell, Gillian S. Butler‐Browne, Vincent Mouly, José Luis Relova, Yolanda Pazos, Jesús P. Camiña
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle, Vol 9, Iss 6, Pp 1063-1078 (2018)
Abstract Background This study was performed to test the therapeutic potential of obestatin, an autocrine anabolic factor regulating skeletal muscle repair, to ameliorate the Duchenne muscular dystrophy (DMD) phenotype. Methods and results Using a mu
Externí odkaz: https://doaj.org/article/ac85ac9944404db6b8a8d3a098d48356
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3
Obestatin signalling counteracts glucocorticoid‐induced skeletal muscle atrophy via NEDD4/KLF15 axis
Autor: Vincent Mouly, José Luis Relova, Saul Leal-lopez, Jesus P. Camiña, Manuel E. Rodríguez‐Fuentes, Icía Santos-Zas, Luis J. Andrade‐Bulos, Jessica González-Sánchez, Yolanda Pazos, Carlos S. Mosteiro, Xesús Casabiell, Tania Cid-Díaz, Fátima Fernández‐Barreiro
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle, Vol 12, Iss 2, Pp 493-505 (2021)
Journal of Cachexia, Sarcopenia and Muscle
Journal of Cachexia, Sarcopenia and Muscle, In press, ⟨10.1002/jcsm.12677⟩
Journal of Cachexia, Sarcopenia and Muscle, Wiley Open Access/Springer Verlag, In press, ⟨10.1002/jcsm.12677⟩
Journal of Cachexia, Sarcopenia and Muscle, Wiley Open Access/Springer Verlag, 2021, 12 (2), pp.493-505. ⟨10.1002/jcsm.12677⟩
International audience; Background: A therapeutic approach for the treatment of glucocorticoid‐induced skeletal muscle atrophy should be based on the knowledge of the molecular mechanisms determining the unbalance between anabolic and catabolic pro
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb3ea29729724ef9bd54613290241f11
https://doaj.org/article/6aba5e89c5064f319ade19594e974fcf
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4
Obestatin signaling controls Schwann cells and axonal transport to counteract neuromuscular synaptic loss in skeletal muscle
Autor: Saul Leal-lopez, Agustin Sanchez-Temprano, Rosalia Gallego, Carlos S. Mosteiro, Jessica González-Sánchez, Jesus P. Camiña, Yolanda Pazos, Jose L. Relova
Background. Injuries to the peripheral nerve system are common conditions, with broad spectrum of symptoms depending on the impaired nerves and severity of damage. Although peripheral nervous system retains a remarkable ability for regeneration, it i
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b971f927936e65701858a685665eb46c
https://doi.org/10.21203/rs.3.rs-55898/v1
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5
Improvement of Duchenne muscular dystrophy phenotype following obestatin treatment
Autor: José Luis Relova, Xesús Casabiell, Jesus P. Camiña, Carlos S. Mosteiro, Jessica González-Sánchez, Gillian Butler-Browne, Yolanda Pazos, Rosalía Gallego, Tania Cid-Díaz, Rubén Nogueiras, Agustin Sanchez-Temprano, Vincent Mouly, Regina Pabst‐Fernández
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle. 9:1063-1078
BACKGROUND This study was performed to test the therapeutic potential of obestatin, an autocrine anabolic factor regulating skeletal muscle repair, to ameliorate the Duchenne muscular dystrophy (DMD) phenotype. METHODS AND RESULTS Using a multidiscip
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::7ea7e5408774589453e9318215455706
https://doi.org/10.1002/jcsm.12338
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6
Obestatin controls the ubiquitin-proteasome and autophagy-lysosome systems in glucocorticoid-induced muscle cell atrophy
Autor: Tomás García-Caballero, Yolanda Pazos, Carlos S. Mosteiro, Icía Santos-Zas, Uxía Gurriarán-Rodríguez, Jessica González-Sánchez, Tania Cid-Díaz, Jesus P. Camiña, Xesús Casabiell, Vincent Mouly
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle. 8:974-990
Background: Many pathological states characterized by muscle atrophy are associated with an increase in circulating glucocorticoids and poor patient prognosis, making it an important target for treatment. The development of treatments for glucocortic
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::e2db2279f82ed1f78ad721b08eb56b74
https://doi.org/10.1002/jcsm.12222
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7
Publisher Correction: Obestatin controls skeletal muscle fiber-type determination
Autor: José Luis Relova, Tania Cid-Díaz, Carlos Seoane-Mosteiro, Uxía Gurriarán-Rodríguez, Begoña Porteiro, Rosalía Gallego, Jesus P. Camiña, Jessica González-Sánchez, Rubén Nogueiras, Xesús Casabiell, Icía Santos-Zas, Yolanda Pazos, Vincent Mouly
Publikováno v: Scientific Reports
Scientific Reports, Vol 8, Iss 1, Pp 1-1 (2018)
Obestatin/GPR39 signaling stimulates skeletal muscle growth and repair by inducing both G-protein-dependent and -independent mechanisms linking the activated GPR39 receptor with distinct sets of accessory and effector proteins. In this work, we descr
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1a01b3e45c3b9626d6b6dd5d73988be5
http://europepmc.org/articles/PMC6294773
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8
β-Arrestin scaffolds and signaling elements essential for the obestatin/GPR39 system that determine the myogenic program in human myoblast cells
Autor: Carlos S. Mosteiro, Tania Cid-Díaz, Rosalía Gallego, Yolanda Pazos, Monica Bouzo-Lorenzo, Xesús Casabiell, Icía Santos-Zas, Vincent Mouly, José Señarís, Felipe F. Casanueva, Uxía Gurriarán-Rodríguez, Jesus P. Camiña, Gabriela Figueroa, Jessica González-Sánchez
Publikováno v: Cellular and Molecular Life Sciences. 73:617-635
Obestatin/GPR39 signaling stimulates skeletal muscle repair by inducing the expansion of satellite stem cells as well as myofiber hypertrophy. Here, we describe that the obestatin/GPR39 system acts as autocrine/paracrine factor on human myogenesis. O
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::354467b645ecf57c04772737df22d166
https://doi.org/10.1007/s00018-015-1994-z
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9
Improvement of Duchenne muscular dystrophy phenotype following obestatin treatment
Autor: Jessica, González-Sánchez, Agustín, Sánchez-Temprano, Tania, Cid-Díaz, Regina, Pabst-Fernández, Carlos S, Mosteiro, Rosalía, Gallego, Ruben, Nogueiras, Xesús, Casabiell, Gillian S, Butler-Browne, Vincent, Mouly, José Luis, Relova, Yolanda, Pazos, Jesús P, Camiña
Publikováno v: Journal of Cachexia, Sarcopenia and Muscle
Background This study was performed to test the therapeutic potential of obestatin, an autocrine anabolic factor regulating skeletal muscle repair, to ameliorate the Duchenne muscular dystrophy (DMD) phenotype. Methods and results Using a multidiscip
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::656849c341aea1be43d95eccab302e27
https://pubmed.ncbi.nlm.nih.gov/30216693
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10
Obestatin controls skeletal muscle fiber-type determination
Autor: Begoña Porteiro, José Luis Relova, Jesus P. Camiña, Yolanda Pazos, Vincent Mouly, Xesús Casabiell, Carlos Seoane-Mosteiro, Rosalía Gallego, Uxía Gurriarán-Rodríguez, Tania Cid-Díaz, Rubén Nogueiras, Icía Santos-Zas, Jessica González-Sánchez
Publikováno v: Scientific Reports
Scientific Reports, Nature Publishing Group, 2017, 7, pp.2137. ⟨10.1038/s41598-017-02337-4⟩
Scientific Reports, Vol 7, Iss 1, Pp 1-14 (2017)
Scientific Reports, 2017, 7, pp.2137. ⟨10.1038/s41598-017-02337-4⟩
International audience; Obestatin/GPR39 signaling stimulates skeletal muscle growth and repair by inducing both G-protein-dependent and-independent mechanisms linking the activated GPR39 receptor with distinct sets of accessory and effector proteins.
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::68eb93eb8298ef2251eddcf8bab1808d
https://hal.sorbonne-universite.fr/hal-01534535/document
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