Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Jessica E Owen"'
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-14 (2021)
Abstract Corpora amylacea (CoA) are spherical aggregates of glucose polymers and proteins within the periventricular, perivascular and subpial regions of the cerebral cortex and the hippocampal cornu ammonis (CA) subfields. The present study quantifi
Externí odkaz:
https://doaj.org/article/b7703b2aae104229b8217ea97adf29b9
Autor:
Jessica E. Owen, Sigrid C. Veasey
Publikováno v:
Neurobiology of Disease, Vol 139, Iss , Pp 104820- (2020)
Chronic short sleep or extended wake periods are commonly observed in most industrialized countries. Previously neurobehavioral impairment following sleep loss was considered to be a readily reversible occurrence, normalized upon recovery sleep. Rece
Externí odkaz:
https://doaj.org/article/b900a935f6aa48a9b0ed56a19bd12a35
Autor:
Guanxia Zhan, Sigrid C. Veasey, Polina Fenik, Yan Zhu, Cathy Liu, Jessica E Owen, Patrick Bell
Publikováno v:
Sleep
Chronic short sleep (CSS) is prevalent in modern societies and has been proposed as a risk factor for Alzheimer’s disease (AD). In support, short-term sleep loss acutely increases levels of amyloid β (Aβ) and tau in wild type (WT) mice and humans
Autor:
Isleifur Olafsson, Stephen R. Robinson, Elizabeth Cook, Bryndis Benediktsdottir, Thorarinn Gislason, Jessica E. Owen
Publikováno v:
Sleep. 44
Obstructive sleep apnea (OSA) involves intermittent cessations of breathing during sleep. People with OSA can experience memory deficits and have reduced hippocampal volume; these features are also characteristic of Alzheimer’s disease (AD), where
Publikováno v:
Sleep. 42
Obstructive sleep apnea (OSA) is commonly associated with memory impairments. Although MRI studies have found volumetric differences in the hippocampus of people with OSA compared with controls, MRI lacks the spatial resolution to detect changes in t
Publikováno v:
Sleep. 42:A120-A121
Publikováno v:
Neurochemical Research. 39:693-699
Hemin, the degradation product of hemoglobin, contributes to the neurodegeneration that occurs in the weeks following a hemorrhagic stroke. The breakdown of hemin in cells releases redox-active iron that can facilitate the production of toxic hydroxy
Publikováno v:
Sleep. 41:A114-A114
Publikováno v:
Neurochemical research. 41(1-2)
Hemin is a breakdown product of the blood protein, hemoglobin and is responsible for much of the secondary damage caused following a hemorrhagic stroke. Hemin is toxic to cultured astrocytes and it is thought that this toxicity is due to iron that is
Publikováno v:
Sleep. 40:A100-A100