Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Jeremy B. Bergsman"'
Autor:
Wenyan Chen, Jeremy B Bergsman, Xiaohua Wang, Gawain Gilkey, Carol-Renée Pierpoint, Erin A Daniel, Emmanuel M Awumey, Philippe Dauban, Robert H Dodd, Martial Ruat, Stephen M Smith
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8563 (2010)
BackgroundNerve terminal invasion by an axonal spike activates voltage-gated channels, triggering calcium entry, vesicle fusion, and release of neurotransmitter. Ion channels activated at the terminal shape the presynaptic spike and so regulate the m
Externí odkaz:
https://doaj.org/article/96c870ffcb884cbd998cf97f0db3a71c
Publikováno v:
Journal of Neuroscience Methods. 152:32-39
The use of fluorescent probes such as FM 1-43 or synapto-pHluorin to study the dynamic aspects of synaptic function has dramatically increased in recent years. The analysis of such experiments is both labor intensive and subject to potentially signif
Publikováno v:
Journal of Neurophysiology. 92:545-552
The calyx of Held is a giant nerve terminal that forms a synapse directly onto the principal cells of the medial nucleus of the trapezoid body (MNTB) in the mammalian auditory brain stem. This central synapse, which is involved in sound localization,
Autor:
Nobutoshi Harata, Richard W. Tsien, Richard H. Scheller, Jeremy B. Bergsman, Stephen M. Smith
Publikováno v:
Neuron. 41(2):243-256
Synaptic activity causes reductions in cleft [Ca(2+)] that may impact subsequent synaptic efficacy. Using modified patch-clamp techniques to record from single neocortical nerve terminals, we report that physiologically relevant reductions of extrace
Autor:
Richard W. Tsien, Jeremy B. Bergsman
Publikováno v:
The Journal of Neuroscience. 20:4368-4378
When the presynaptic membrane protein syntaxin is coexpressed inXenopusoocytes with N- or P/Q-type Ca2+channels, it promotes their inactivation (Bezprozvanny et al., 1995; Wiser et al., 1996, 1999; Degtiar et al., 2000) (I. B. Bezprozvanny, P. Zhong,
Autor:
Jeremy B. Bergsman, Philippe Dauban, Stephen M. Smith, Xiaohua Wang, Wenyan Chen, Robert H. Dodd, Carol Renée Pierpoint, Martial Ruat, Erin A. Daniel, Gawain M. Gilkey, Emmanuel M. Awumey
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8563 (2010)
PLoS ONE
PLoS ONE, Public Library of Science, 2010, 5 (1), pp.e8563. ⟨10.1371/journal.pone.0008563⟩
PLoS ONE, Public Library of Science, 2010, 5 (1), pp.e8563-. ⟨10.1371/journal.pone.0008563⟩
PLoS ONE
PLoS ONE, Public Library of Science, 2010, 5 (1), pp.e8563. ⟨10.1371/journal.pone.0008563⟩
PLoS ONE, Public Library of Science, 2010, 5 (1), pp.e8563-. ⟨10.1371/journal.pone.0008563⟩
International audience; BACKGROUND: Nerve terminal invasion by an axonal spike activates voltage-gated channels, triggering calcium entry, vesicle fusion, and release of neurotransmitter. Ion channels activated at the terminal shape the presynaptic s
Publikováno v:
Pharmacology of Ionic Channel Function: Activators and Inhibitors ISBN: 9783642630309
Voltage-gated Ca2+channels are members of a superfamily of voltage-gated ion channels which also includes Na+channels and K+channels. Ca2+channels transduce membrane potential changes to intracellular Ca2+-signals in a wide variety of cell types, inc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2da6173dc6deba1006fe3350f077d06f
https://doi.org/10.1007/978-3-642-57083-4_3
https://doi.org/10.1007/978-3-642-57083-4_3
Publikováno v:
Critical Care
Excessive and uncontrolled synaptic transmission resulting in increased glutamate release has been highlighted as responsible for neuronal death in a variety of neurological conditions including head trauma, stroke and status epilepticus. We are inte