Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Jeong Im Woo"'
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-10 (2024)
Abstract Aqueous humor (AH) and blood levels of transforming growth factor β (TGFβ) are elevated in idiopathic primary open angle glaucoma (POAG) representing a disease biomarker of unclear status and function. Tsk mice display a POAG phenotype and
Externí odkaz:
https://doaj.org/article/3b1403046cc34ea593baad32b5d4a96b
Autor:
MinHee K. Ko, Jeong-Im Woo, Jose M. Gonzalez, Gayeoun Kim, Lynn Sakai, Janos Peti-Peterdi, Jonathan A. Kelber, Young-Kwon Hong, James C. Tan
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-16 (2022)
Abstract Primary open angle glaucoma (POAG) features an optic neuropathy, elevated aqueous humor (AH) TGFβ2, and major risk factors of central corneal thickness (CCT), increasing age and intraocular pressure (IOP). We examined Tight skin (Tsk) mice
Externí odkaz:
https://doaj.org/article/945bd2ddee8c4c2ea2e4d2b60fbf6cd4
Autor:
Dongchun Liang, Jeong-Im Woo, Hui Shao, Willi K Born, Rebecca L O'Brien, Henry J Kaplan, Deming Sun
Publikováno v:
PLoS ONE, Vol 13, Iss 5, p e0197189 (2018)
Whether γδ T cells inhibit or enhance the Foxp3 T cell response depends upon their activation status. The critical enhancing effector in the supernatant is adenosine. Activated γδ T cells express adenosine receptors at high levels, which enables
Externí odkaz:
https://doaj.org/article/160b4676efa74df4a6b0b6524420edf6
Autor:
MinHee K. Ko, Jeong-Im Woo, Jose M. Gonzalez, Gayeoun Kim, Lynn Sakai, Janos Peti-Peterdi, Jonathan A. Kelber, Young-Kwon Hong, James C. Tan
Publikováno v:
Scientific reports. 12(1)
Primary open angle glaucoma (POAG) features an optic neuropathy, elevated aqueous humor (AH) TGFβ2, and major risk factors of central corneal thickness (CCT), increasing age and intraocular pressure (IOP). We examined Tight skin (Tsk) mice to see if
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::849afc6fcd15e3e1febfb9454b086396
https://pubmed.ncbi.nlm.nih.gov/35739142
https://pubmed.ncbi.nlm.nih.gov/35739142
Publikováno v:
PLoS ONE, Vol 9, Iss 3, p e90933 (2014)
Middle ear infection, otitis media (OM), is clinically important due to the high incidence in children and its impact on the development of language and motor coordination. Previously, we have demonstrated that the human middle ear epithelial cells u
Externí odkaz:
https://doaj.org/article/f67575c28faa47168576e1db92bd4437
Publikováno v:
The Annals of otology, rhinology, and laryngology. 128(6_suppl)
Objectives: Inflammation is crucial for the pathogenesis of acquired sensorineural hearing loss, but the precise mechanism involved remains elusive. Among a number of inflammatory mediators, tumor necrosis factor-alpha (TNF-α) plays a pivotal role i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36bfc2aa75930ae2a32824601ddb6801
https://pubmed.ncbi.nlm.nih.gov/31092034
https://pubmed.ncbi.nlm.nih.gov/31092034
Publikováno v:
Annals of Otology, Rhinology & Laryngology; 2019 Supplement, Vol. 128, p8S-15S, 8p
Publikováno v:
BMC Infectious Diseases. 2010, Vol. 10 Issue 1, p314-323. 10p.
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950), vol 194, iss 8
Cochlear inflammatory diseases, such as tympanogenic labyrinthitis, are associated with acquired sensorineural hearing loss. Although otitis media is extremely frequent in children, tympanogenic labyrinthitis is not commonly observed, which suggests
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d3a3a7f972ea7918a40465edd0d19e9
https://europepmc.org/articles/PMC4390512/
https://europepmc.org/articles/PMC4390512/
Publikováno v:
Innate immunity. 21(2)
Otitis media (OM), one of the most prevalent diseases in young children, is clinically important owing to its high incidence in children and its potential impact on language development and motor coordination. OM is the most common reason for the pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eaa3610bbef06ab639e40b7200c15030
https://pubmed.ncbi.nlm.nih.gov/24842664
https://pubmed.ncbi.nlm.nih.gov/24842664