Normalized Hand Grip and Back Muscle Strength as Risk Factors for Incident Type 2 Diabetes Mellitus: 16 Years of Follow-Up in a Population-Based Cohort Study

Autor: Jeon YJ, Lee SK, Shin C

Publikováno v: Diabetes, Metabolic Syndrome and Obesity, Vol Volume 14, Pp 741-750 (2021)

Yoo-Jeong Jeon,1 Seung Ku Lee,2,3 Chol Shin2– 5 1Department of Sports & Health Science, Hanbat National University, Daejeon, Korea; 2Institute of Human Genomic Study, College of Medicine, Korea University Ansan Hospital, Ansan-si, Gyeonggi-do, Repu

Externí odkaz: https://doaj.org/article/0c595c36bf32472f943152df3000d46c

Association between eNOS polymorphisms and risk of coronary artery disease in a Korean population: a meta-analysis

Autor: Kim Oj, Sung Jh, Kim Sh, Moon Jy, Lim Sw, Lee Be, Jeon Yj, Nam Keun Kim, Kim Jo, Kim Ij, Cha Dh

Publikováno v: Genetics and Molecular Research. 14:16508-16520

Coronary artery disease (CAD), a multifactorial disease, is a common cause of mortality in humans. Polymorphisms in the endothelial nitric oxide synthase (eNOS) gene (-786T>C, 4a4b, and 894G>T) have been previously associated with increased CAD risk.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d7d88051bcd20b096d874eb7364373f9
https://doi.org/10.4238/2015.december.9.23

EGFR and MET induce tumorigenesis and gefitinib resistance in lung cancers by controlling microRNA expression

Autor: Garofalo M1, Romano G, Di Leva G, Nuovo G, Jeon YJ, Ngankeu A, Sun J, Lovat F, Alder H, Condorelli G, Engelman JA, Ono M, Rho JK, Cascione L, Volinia S, Nephew KP, Croce CM.

Publikováno v: Nature medicine
(2011).
info:cnr-pdr/source/autori:Garofalo M1, Romano G, Di Leva G, Nuovo G, Jeon YJ, Ngankeu A, Sun J, Lovat F, Alder H, Condorelli G, Engelman JA, Ono M, Rho JK, Cascione L, Volinia S, Nephew KP, Croce CM./titolo:EGFR and MET induce tumorigenesis and gefitinib resistance in lung cancers by controlling microRNA expression/doi:/rivista:Nature medicine (Print)/anno:2011/pagina_da:/pagina_a:/intervallo_pagine:/volume

The involvement of the MET oncogene in de novo and acquired resistance of non-small cell lung cancers (NSCLCs) to tyrosine kinase inhibitors (TKIs) has previously been reported, but the precise mechanism by which MET overexpression contributes to TKI

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::96094bcd487c8711cb718cb788079e81
http://www.cnr.it/prodotto/i/340724

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