Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Jens Kockskaemper"'
Autor:
Andreas Gardemann, C H Hartmann, Andreas Goette, Carmen Wolke, Ravi Kumar Chilukoti, Uwe Lendeckel, Alicja Bukowska, Florentina Pluteanu, Jens Kockskaemper
Publikováno v:
EP Europace. 20:i18-i18
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f0a2997b3e36bcbb14cb2a4ee762292f
https://doi.org/10.1093/europace/euy015.042
https://doi.org/10.1093/europace/euy015.042
Publikováno v:
SPIE Proceedings.
Excitation-contraction (e-c) coupling in atrial myocytes was studied with fast confocal microscopy and simultaneous Ca2+-current measurements. Cat atrial myocytes lack transverse tubules and contain junctional (j-SR) and non- junctional SR (nj-SR) wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dcc2a1f6025694d133d60a7ef8a8bbbc
https://doi.org/10.1117/12.472112
https://doi.org/10.1117/12.472112
Publikováno v:
ResearcherID
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1364a706762d3543658a8975801a20ce
https://doi.org/10.1093/eurheartj/eht307.32
https://doi.org/10.1093/eurheartj/eht307.32
Autor:
Alicja Bukowska, Uwe Lendeckel, T. Kiess, Andreas Goette, C. Wolke, Florentina Pluteanu, Ravi Kumar Chilukoti, C. Sack, Judit Preisenberger, Jens Kockskaemper
Publikováno v:
European Heart Journal. 34:P5040-P5040
Purpose: Hypertension induces cardiac remodelling at the structural and functional level. Endothelin-1 (ET-1) may contribute to cardiac remodelling. It is not known, however, whether atrial ET-1 signalling is altered in hypertension. Here we tested t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::68175fb663a9c19bc35cff3b807dc920
https://doi.org/10.1093/eurheartj/eht310.p5040
https://doi.org/10.1093/eurheartj/eht310.p5040
Autor:
Jens Kockskaemper, A. Hartmann, Alicja Bukowska, Ravi Kumar Chilukoti, C. Wolke, Florentina Pluteanu, Uwe Lendeckel, Andreas Goette
Publikováno v:
European Heart Journal. 34:P4989-P4989
Purpose: Arterial hypertension and age are the most predominant factors causing atrial fibrillation (AF). The exact proarrhythmogenic pathways involved are still unknown. The present study was designed to examine changes in the composition of atrial
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b55163c6a3b62e06e6cad43f620622d8
https://doi.org/10.1093/eurheartj/eht310.p4989
https://doi.org/10.1093/eurheartj/eht310.p4989
Autor:
Simon Sedej, Michael Sacherer, Jens Kockskaemper, Frank R. Heinzel, Marc A. Vos, Burkert Pieske, Philipp Gronau
Publikováno v:
CIÊNCIAVITAE
ResearcherID
ResearcherID
Diastolic Ca2+ leak from the sarcoplasmic reticulum (SR) via the ryanodine receptor (RyR2) contributes to arrhythmias. Protein kinase A (PKA) and Ca2+/Calmodulin- dependent protein kinase II (CaMKII) have been involved in SR Ca2+ leak by altering RyR
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad7549b409f84c0145747e306f9a8b19
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000306288601400&KeyUID=WOS:000306288601400
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000306288601400&KeyUID=WOS:000306288601400
Autor:
Simon Sedej, Stefanie Walther, Egbert Bisping, Jens Kockskaemper, Silvia G. Priori, Carlo Napolitano, Burkert Pieske, Albrecht Schmidt, Marco Denegri, Frank R. Heinzel
Publikováno v:
CIÊNCIAVITAE
Web of Science
Publons
ResearcherID
Web of Science
Publons
ResearcherID
Elevated spontaneous Ca2+ release from the sarcoplasmic reticulum (SR) Ca2+ release channel (ryanodine receptor-RyR2) due to a gain-of-function of the RyR2 defect contributes to contractile dysfunction and arrhythmias in heart failure (HF). However,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29c325a96b5a47613c2461b6368d7b85
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000306288602342&KeyUID=WOS:000306288602342
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=ORCID&SrcApp=OrcidOrg&DestLinkType=FullRecord&DestApp=WOS_CPL&KeyUT=WOS:000306288602342&KeyUID=WOS:000306288602342
Publikováno v:
ResearcherID
CIÊNCIAVITAE
CIÊNCIAVITAE
A hallmark of heart failure is impaired Ca2+ handling of cardiomyocytes. We previously showed that specific alterations in nuclear Ca2+ handling precede changes in cytoplasmic Ca2+ handling as heart failure progresses. However, a direct link between
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cc6915e572134266bd5275c8e55c9a57