Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Jennifer N Berry"'
Altered nicotine reward-associated behavior following α4 nAChR subunit deletion in ventral midbrain.
Autor:
Can Peng, Staci E Engle, Yijin Yan, Marcus M Weera, Jennifer N Berry, Matthew C Arvin, Guiqing Zhao, J Michael McIntosh, Julia A Chester, Ryan M Drenan
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0182142 (2017)
Nicotinic acetylcholine receptors containing α4 subunits (α4β2* nAChRs) are critical for nicotinic cholinergic transmission and the addictive action of nicotine. To identify specific activities of these receptors in the adult mouse brain, we coupl
Externí odkaz:
https://doaj.org/article/5a4fdee2b9f645bd89d000874dc2acce
Autor:
Melissa J. Evans, Jennifer N. Berry
Publikováno v:
The FASEB Journal. 35
Autor:
Staci E. Engle, Veronica J. Kim, J. Michael McIntosh, Can Peng, Rachael L. Neve, Ryan M. Drenan, Jennifer N. Berry, Yijin Yan
Publikováno v:
Eur J Neurosci
Nicotinic acetylcholine receptors (nAChRs), prototype members of the cys-loop ligand-gated ion channel family, are key mediators of cholinergic transmission in the central nervous system. Despite their importance, technical gaps exist in our ability
Publikováno v:
Pharmacology Biochemistry and Behavior. 208:173221
The increasingly popular combination of "energy drinks" containing high amounts of caffeine and alcohol has been shown to induce a stimulated, rather than sedated, state which may result in increased binge drinking and increased risk for alcohol-attr
Autor:
Terrance Chiang, Jiwon E. Ha, Jennifer N. Berry, Richard M. van Rijn, Mee Jung Ko, Meridith T. Robins
Publikováno v:
Frontiers in Behavioral Neuroscience
Frontiers in Behavioral Neuroscience, Vol 12 (2018)
Frontiers in Behavioral Neuroscience, Vol 12 (2018)
beta-arrestin 1 and 2 are highly expressed proteins involved in the desensitization of G protein-coupled receptor signaling which also regulate a variety of intracellular signaling pathways. Gene knockout studies suggest that the two isoforms are not
Publikováno v:
Alcohol (Fayetteville, N.Y.)
Chronic intermittent ethanol consumption is associated with neurodegeneration and cognitive deficits in preclinical laboratory animals and in the clinical population. While previous work suggests a role for neuroadaptations in the N-methyl-d-aspartat
Autor:
Anna R. Reynolds, Lynda J. Sharrett-Field, Jennifer N. Berry, Sydney R. Winchester, Mark A. Prendergast, Meredith A. Saunders
Chronic, intermittent ethanol (CIE) exposure is known to produce neuroadaptive alterations in excitatory neurotransmission that contribute to the development of dependence. Although activation of protein kinases (e.g., cyclic AMP [cAMP]-dependent pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ac2ad93e301531a88f85299771d027b
https://europepmc.org/articles/PMC6662204/
https://europepmc.org/articles/PMC6662204/
Autor:
Matthew C. Arvin, J. Michael McIntosh, Yijin Yan, Staci E. Engle, Guiqing Zhao, Ryan M. Drenan, Julia A. Chester, Marcus M. Weera, Can Peng, Jennifer N. Berry
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0182142 (2017)
PLoS ONE
PLoS ONE
Nicotinic acetylcholine receptors containing α4 subunits (α4β2* nAChRs) are critical for nicotinic cholinergic transmission and the addictive action of nicotine. To identify specific activities of these receptors in the adult mouse brain, we coupl
Autor:
Lynda J. Sharrett-Field, Tracy Butler, Jennifer N. Berry, Anna R. Reynolds, Mark A. Prendergast
Publikováno v:
Alcoholism: Clinical and Experimental Research. 37:1417-1423
Stress Often Precedes the development of alcohol dependence and is also a consequence of dependence, thereby perpetuating the cycle of dependence, withdrawal, and relapse to alcohol use (Heilig et al., 2010; Koob and Kreek, 2007). Indeed, many indivi
Publikováno v:
Neuroscience. 222:147-158
Excitotoxicity is the overexcitation of neurons due to the excessive activation of excitatory amino acid (EAA) receptors and likely contributes to central nervous system injury in neurodegenerative states, including traumatic brain injury (TBI), isch