Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Jennifer M, Hahn"'
Autor:
Stanley B. DeVore, Matthew Schuetz, Lauren Alvey, Henry Lujan, David E. Ochayon, Lindsey Williams, Wan Chi Chang, Alyssa Filuta, Brandy Ruff, Arjun Kothari, Jennifer M. Hahn, Eric Brandt, Latha Satish, Krishna Roskin, Andrew B. Herr, Jocelyn M. Biagini, Lisa J. Martin, Deniz Cagdas, Sevgi Keles, Joshua D. Milner, Dorothy M. Supp, Gurjit K. Khurana Hershey
Publikováno v:
Cell Reports, Vol 43, Iss 8, Pp 114589- (2024)
Summary: Caspase recruitment domain family member 14 (CARD14) and its variants are associated with both atopic dermatitis (AD) and psoriasis, but their mechanistic impact on skin barrier homeostasis is largely unknown. CARD14 is known to signal via N
Externí odkaz:
https://doaj.org/article/93643c0fb9c04c9683a5cbbebc4dcd2e
Autor:
Jennifer M. Hahn, BS, Kevin L. McFarland, MS, JD, Kelly A. Combs, BS, Heather M. Powell, PhD, Dorothy M. Supp, PhD
Publikováno v:
Plastic and Reconstructive Surgery, Global Open, Vol 10, Iss 11, p e4680 (2022)
Background:. Keloids are disfiguring, scar-like lesions that are challenging to treat, with low response rates to current interventions and frequent recurrence. It has been widely reported that keloids are characterized by myofibroblasts, specialized
Externí odkaz:
https://doaj.org/article/945b9d1902e34361a09a3cd3301df20b
Publikováno v:
STAR Protocols, Vol 3, Iss 1, Pp 101172- (2022)
Summary: Four types of primary cells—dermal fibroblasts, dermal microvascular endothelial cells, epidermal keratinocytes, and epidermal melanocytes—can be isolated simultaneously from a single human skin sample, without the use of xenogeneic muri
Externí odkaz:
https://doaj.org/article/9451912b5aa14ec2b82e27f13f0a5425
Autor:
Dorothy M. Supp, Jennifer M. Hahn, Kelly A. Combs, Kevin L. McFarland, Ann Schwentker, Raymond E. Boissy, Steven T. Boyce, Heather M. Powell, Anne W. Lucky
Publikováno v:
Cell Transplantation, Vol 28 (2019)
The blistering disease recessive dystrophic epidermolysis bullosa (RDEB) is caused by mutations in the gene encoding collagen VII (COL7), which forms anchoring fibrils that attach the epidermis to the dermis. Cutaneous gene therapy to restore COL7 ex
Externí odkaz:
https://doaj.org/article/abcfe7ae4538485ab5c75f5e748d3f6a
Autor:
Jennifer M Hahn, Kelly A Combs, Christopher M Lloyd, Kevin L McFarland, Steven T Boyce, Dorothy M Supp
Publikováno v:
PLoS ONE, Vol 14, Iss 3, p e0213325 (2019)
Engineered skin substitutes (ESS), prepared using primary human fibroblasts and keratinocytes with a biopolymer scaffold, were shown to provide stable closure of excised burns, but relatively little is known about innervation of ESS after grafting. T
Externí odkaz:
https://doaj.org/article/38c54458966343c997cba9612767d5e3
Autor:
Christopher M. Lloyd, Kelly A. Combs, Dorothy M. Supp, Viki B. Swope, Steven T. Boyce, Zalfa A. Abdel-Malek, Jennifer M. Hahn
Publikováno v:
Journal of Burn Care & Research. 41:751-760
Engineered skin substitutes (ESS) containing autologous fibroblasts and keratinocytes provide stable wound closure in patients with large, full-thickness burns, but are limited by hypopigmentation due to absence of added melanocytes. DNA damage cause
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e80aafb7f93118d2a619402c4c2e4e0
https://doi.org/10.1093/jbcr/iraa029
https://doi.org/10.1093/jbcr/iraa029
Autor:
Heather M. Powell, Raymond E. Boissy, Steven T. Boyce, Kelly A. Combs, Ann Schwentker, Anne W. Lucky, Kevin L. McFarland, Dorothy M. Supp, Jennifer M. Hahn
Publikováno v:
Cell Transplantation
Cell Transplantation, Vol 28 (2019)
Cell Transplantation, Vol 28 (2019)
The blistering disease recessive dystrophic epidermolysis bullosa (RDEB) is caused by mutations in the gene encoding collagen VII (COL7), which forms anchoring fibrils that attach the epidermis to the dermis. Cutaneous gene therapy to restore COL7 ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1be4157611d01656f47de936f1c136f7
https://doi.org/10.1177/0963689719857657
https://doi.org/10.1177/0963689719857657
Publikováno v:
Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair SocietyREFERENCES. 29(5)
Keloids are fibroproliferative lesions resulting from an abnormal wound healing process due to pathological mechanisms that remain incompletely understood. Keloids tend to occur more frequently in anterior versus posterior body regions (e.g., ears, f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2997e30a5e6d8c19c7b332850fe35a30
https://pubmed.ncbi.nlm.nih.gov/33811779
https://pubmed.ncbi.nlm.nih.gov/33811779
Publikováno v:
Journal of Burn Care & Research. 43:S145-S145
Introduction Keloids are disfiguring, fibroproliferative lesions that can result from an abnormal wound healing process. Keloids are characterized by excessive and abnormal deposition of extracellular matrix and unrestrained expansion beyond the orig
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8bc8f4d92d25e5c2a8127b72aaaa80ed
https://doi.org/10.1093/jbcr/irac012.238
https://doi.org/10.1093/jbcr/irac012.238
Autor:
Jennifer M. Hahn, Heather M. Powell, Britani N. Blackstone, Dorothy M. Supp, Kevin L. McFarland, Danielle M. DeBruler
Publikováno v:
Acta Biomaterialia. 80:247-257
Engineered skin (ES) offers many advantages over split-thickness skin autografts for the treatment of burn wounds. However, ES, both in vitro and after grafting, is often significantly weaker, less elastic and more compliant than normal human skin. B
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a3df8fd595c1ffb43b0404c7ba9cadb
https://doi.org/10.1016/j.actbio.2018.09.014
https://doi.org/10.1016/j.actbio.2018.09.014