Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Jennifer M, Felton"'
Aryl hydrocarbon receptor and IL-13 signaling crosstalk in human keratinocytes and atopic dermatitis
Autor:
Steven P. Proper, Alexander T. Dwyer, Andrews Appiagyei, Jennifer M. Felton, Netali Ben-Baruch Morgenstern, Justin M. Marlman, Michael Kotliar, Artem Barski, Ty D. Troutman, Marc E. Rothenberg, Tesfaye B. Mersha, Nurit P. Azouz
Publikováno v:
Frontiers in Allergy, Vol 5 (2024)
IntroductionAtopic dermatitis (AD) is an allergic skin disease mediated by skin barrier impairment and IL-13-driven immune response. Activation of the aryl hydrocarbon receptor (AHR) has shown promise in early clinical trials for AD; however, the mec
Externí odkaz:
https://doaj.org/article/250f10865655442983c6157c3f1b2cd5
Autor:
Netali Ben-Baruch Morgenstern, Adina Y. Ballaban, Ting Wen, Tetsuo Shoda, Julie M. Caldwell, Kara Kliewer, Jennifer M. Felton, J. Pablo Abonia, Vincent A. Mukkada, Philip E. Putnam, Scott M. Bolton, Daniel F. Dwyer, Nora A. Barrett, Marc E. Rothenberg
Publikováno v:
J Allergy Clin Immunol
BACKGROUND: Mast cells (MCs) are pleiotropic cells that accumulate in the esophagus of patients with eosinophilic esophagitis (EoE) and are thought to contribute to disease pathogenesis, yet their properties and functions in this organ are largely un
Autor:
Netali Ben-Baruch Morgenstern, Tetsuo Shoda, Yrina Rochman, Julie M. Caldwell, Margaret H. Collins, Vincent Mukkada, Philip E. Putnam, Scott M. Bolton, Jennifer M. Felton, Mark Rochman, Cristin Murray-Petzold, Kara L. Kliewer, Marc E. Rothenberg
Publikováno v:
Journal of Allergy and Clinical Immunology.
Autor:
Tetsuo Shoda, Ting Wen, Julie M. Caldwell, Netali Ben-Baruch Morgenstern, Garrett A. Osswald, Mark Rochman, Lydia E. Mack, Jennifer M. Felton, J. Pablo Abonia, Nicoleta C. Arva, Dan Atkins, Peter A. Bonis, Kelley E. Capocelli, Margaret H. Collins, Evan S. Dellon, Gary W. Falk, Nirmala Gonsalves, Sandeep K. Gupta, Ikuo Hirano, John Leung, Paul A. Menard-Katcher, Vincent A. Mukkada, Philip E. Putnam, Amanda K. Rudman Spergel, Jonathan M. Spergel, Joshua B. Wechsler, Guang-Yu Yang, Seema S. Aceves, Glenn T. Furuta, Marc E. Rothenberg, Seema Aceves, Samuel Almonte, Rachel Andrews, Ashley Arrington, Nicoleta Arva, Fred Atkins, Dominique Bailey, Alexis Berry, Bridget Besl, Scott Bolton, Peter Bonis, Wendy Book, Kimberly Bray, Teresa Brown, Cassandra Burger, Deirdre Burke, Jonathon Cahoon, Kelley Capocelli, Mirna Chehade, Margaret Collins, Carla Davis, Evan Dellon, Maureen DeMarshall, Lauren DiTommaso, Ranjan Dohil, Michael Eby, Gary Falk, David Fleischer, Heather Foote, Kelci Foss, Joel Friedlander, Patricia Fulkerson, Glenn Furuta, Debra Geno, Thomas Greuter, Sandeep Gupta, Frank Hamilton, Kirk Harris, Jennifer Harris, Girish Hiremath, Nicole Holland-Thomas, Lea Jacinto, Amir Kagalwalla, Timothy Kaseta, David Katzka, Kaitlin Keeley, Emad Khosh-Hemmat, Paneez Khoury, Eileen King, Kara Kliewer, Amy Klion, Jennifer Knowles, Kendra Kocher, Ellyn Kodroff, Jeffrey Krischer, Shay Kyle, Meredith Levy, Chris Liacouras, Denise Mack, Lisa Martin, Ellen Martin, Talaya McCright-Gill, Paul Menard-Katcher, Calies Menard-Katcher, Gabriela Mendoza, Melissa Mingler, Mike Minnicozzi, Amanda Muir, Vincent Mukkada, Cristin MurrayPetzold, Robert Newbury, Quan Nhu, Oghenekpaobor (Joel) Oyibo, Allisa Paliana, Zhaoxing Pan, Robbie Pesek, Kathryn Peterson, Heidi Poppendeck, Philip Putnam, Fabian Rivera, Marc Rothenberg, Amanda Rudman Spergel, Kathleen Sable, Alain Schoepfer, Melissa Scott, Rachel Sheridan, Selma Sinanovic, Jonathan Spergel, MaryJo Strobel, Kiki Sun, Amy Tasco, Crystal Tholen, Katherine Thompson, Tiffany Tomkinson, Daisy Tran, Alexandra Tylicki, Tiina Urv, Mei-Lun Wang, Joshua Wechsler, Barry Wershil, Lisa Wheatley, Leah Wilkey, Angelika Zalewski, Amy Zicarelli
Publikováno v:
Gastroenterology. 162:439-453
Eosinophilic esophagitis (EoE) can progress to fibrostenosis by unclear mechanisms. Herein, we investigated gene dysregulation in fibrostenotic EoE, its association with clinical parameters and specific pathways, and the functional consequences.Esoph
Autor:
Jennifer M Felton, Mark Rochman, Chandrashekhar Pasare, Marc E. Rothenberg, Aleksey Porollo, Lydia E Mack, Yrina Rochman, Julie M. Caldwell, Jeff E. Habel, Michael Brusilovsky
Publikováno v:
Nature Immunology. 22:1316-1326
Environmental allergens, including fungi, insects and mites, trigger type 2 immunity; however, the innate sensing mechanisms and initial signaling events remain unclear. Herein, we demonstrate that allergens trigger RIPK1–caspase 8 ripoptosome acti
Autor:
Marc E. Rothenberg, Artem Barski, Matthew T. Weirauch, Jennifer M Felton, Kevin Ernst, Lee Edsall, Patricia C. Fulkerson, Benjamin Wronowski, Sreeja Parameswaran, Sushmitha Vallabh, Astha Malik, Michael Kotliar
Publikováno v:
The Journal of Immunology. 207:1044-1054
Eosinophils develop in the bone marrow from hematopoietic progenitors into mature cells capable of a plethora of immunomodulatory roles via the choreographed process of eosinophilopoiesis. However, the gene regulatory elements and transcription facto
Autor:
Calum T. Robb, You Zhou, Jennifer M. Felton, Birong Zhang, Marie Goepp, Privjyot Jheeta, Danielle J. Smyth, Rodger Duffin, Sonja Vermeren, Richard M. Breyer, Shuh Narumiya, Henry J. McSorley, Rick M. Maizels, Jürgen K. J. Schwarze, Adriano G. Rossi, Chengcan Yao
Publikováno v:
Robb, C T, Zhou, Y, Felton, J M, Zhang, B, Goepp, M, Jheeta, P, Smyth, D J, Duffin, R, Vermeren, S, Breyer, R M, Narumiya, S, McSorley, H J, Maizels, R M, Schwarze, J K J, Rossi, A G & Yao, C 2022, ' Metabolic regulation by prostaglandin E2 impairs lung group 2 innate lymphoid cell responses ', Allergy . https://doi.org/10.1111/all.15541
BACKGROUND: Group 2 innate lymphoid cells (ILC2s) play a critical role in asthma pathogenesis. Non-steroidal anti-inflammatory drug (NSAID)-exacerbated respiratory disease (NERD) is associated with reduced signaling via EP2, a receptor for prostaglan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::746c36722ff2f84466ab63ada2f62274
https://www.pure.ed.ac.uk/ws/files/300635753/Robb_et_al_Accepted_manuscript.docx
https://www.pure.ed.ac.uk/ws/files/300635753/Robb_et_al_Accepted_manuscript.docx
Autor:
Calum T, Robb, You, Zhou, Jennifer M, Felton, Birong, Zhang, Marie, Goepp, Privjyot, Jheeta, Danielle J, Smyth, Rodger, Duffin, Sonja, Vermeren, Richard M, Breyer, Shuh, Narumiya, Henry J, McSorley, Rick M, Maizels, Jürgen K J, Schwarze, Adriano G, Rossi, Chengcan, Yao
Publikováno v:
AllergyREFERENCES.
Group 2 innate lymphoid cells (ILC2s) play a critical role in asthma pathogenesis. Non-steroidal anti-inflammatory drug (NSAID)-exacerbated respiratory disease (NERD) is associated with reduced signaling via EP2, a receptor for prostaglandin EThe rol
Autor:
Calum T. Robb, You Zhou, Jennifer M. Felton, Birong Zhang, Marie Goepp, Privjyot Jheeta, Danielle J. Smyth, Richard M. Breyer, Shuh Narumiya, Henry J. McSorley, Rick Maizels, Jürgen Schwarze, Adriano G. Rossi, Chengcan Yao
Background: Group 2 innate lymphoid cells (ILC2s) play a critical role in asthma pathogenesis. Non-steroidal anti-inflammatory drug (NSAID)-exacerbated respiratory disease (NERD) is associated with reduced signaling via EP2, a receptor for prostaglan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d97a7b152ef277c7a3a10e23e8507770
https://doi.org/10.22541/au.164068294.41852173/v1
https://doi.org/10.22541/au.164068294.41852173/v1
Autor:
Calum T. Robb, You Zhou, Jennifer M. Felton, Birong Zhang, Marie Goepp, Privjyot Jheeta, Danielle J. Smyth, Richard M. Breyer, Shuh Narumiya, Henry J. McSorley, Rick M. Maizels, Jürgen K.J. Schwarze, Adriano G. Rossi, Chengcan Yao
Group 2 innate lymphoid cells (ILC2s) play a critical role in asthma pathogenesis. Non-steroidal anti-inflammatory drug (NSAID)-exacerbated respiratory disease (NERD) is associated with reduced signaling via EP2, a receptor for prostaglandin E2 (PGE2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f4816783f877255fef5b70a4c85e419b
https://doi.org/10.1101/2021.12.23.474031
https://doi.org/10.1101/2021.12.23.474031