Výsledky vyhledávání - "Jennifer Chaplin"

The biology of giant ostracods (Crustacea, Cyprididae), a review focusing on the Mytilocypridinae from Australian inland waters

Autor: Mahabubur Rahman, Jennifer Chaplin, Adrian Pinder

Publikováno v: Marine and Freshwater Research. 74:1-19

This study uses published and unpublished data to create a comprehensive and up-to-date synthesis of available information on a little-known group of invertebrates, namely, non-marine giant ostracods (cypridids ≥3 mm long). Approximately 8% of the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::5861b35ac3f2991bd8774ea6ba8f07cc
https://doi.org/10.1071/mf22092

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Phylogenetic assessment of the halophilic Australian gastropod Coxiella and South African Tomichia resolves taxonomic uncertainties, uncovers new species and supports a Gondwanan link

Autor: Angus D'Arcy Lawrie, Jennifer Chaplin, Lisa Kirkendale, Corey Whisson, Adrian Pinder, Musa C. Mlambo

Publikováno v: Molecular Phylogenetics and Evolution. 184:107810

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::10d9637a086e39b1a28aeaba2d3ef8d0
https://doi.org/10.1016/j.ympev.2023.107810

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miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome

Autor: Petra Rocic, Jennifer Chaplin, Robert A. Barrington, Erika Smith, Brenda Hutcheson, Russell Terry, Rebecca Hutcheson, Spencer D. Proctor, Rashmi Jadhav

Publikováno v: American Journal of Physiology-Heart and Circulatory Physiology. 308:H1323-H1335

Coronary collateral growth (CCG) is impaired in metabolic syndrome. microRNA-21 (miR-21) is a proproliferative and antiapoptotic miR, which we showed to be elevated in metabolic syndrome. Here we investigate whether impaired CCG in metabolic syndrome

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::263a62c15f79a95bb02adf1f4186a7f4
https://doi.org/10.1152/ajpheart.00654.2014

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MicroRNA-145 Restores Contractile Vascular Smooth Muscle Phenotype and Coronary Collateral Growth in the Metabolic Syndrome

Autor: Jennifer Chaplin, Russell Terry, Petra Rocic, Thomas M. Lincoln, Rebecca Hutcheson, Alla Musiyenko, James C. Russell, Erika Smith

Publikováno v: Arteriosclerosis, Thrombosis, and Vascular Biology. 33:727-736

Objective— Transient, repetitive occlusion stimulates coronary collateral growth (CCG) in normal animals. Vascular smooth muscle cells (VSMCs) switch to synthetic phenotype early in CCG, then return to contractile phenotype. CCG is impaired in the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17e8c2ee6eb00f23b44382967c9caa4a
https://doi.org/10.1161/atvbaha.112.301116

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miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome

Autor: Jennifer Chaplin, James C. Russell, Brenda Hutcheson, Spencer D. Proctor, Petra Rocic, Rebecca Hutcheson, Sarah A. Gebb, Faye Borthwick, Erika Smith, Rashmi Jadhav

Publikováno v: FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 28(9)

Inadequate cell proliferation is considered a major causative factor for impaired coronary collateral growth (CCG). Proangiogenic growth factors (GFs) stimulate cell proliferation, but their administration does not promote CCG in patients. These GFs

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::38e45fa716879e6429fd0d7202117e8f
https://pubmed.ncbi.nlm.nih.gov/24903275

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