Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jenna C. Glatzer"'
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Abstract Neurons of the cochleovestibular ganglion (CVG) transmit hearing and balance information to the brain. During development, a select population of early otic progenitors express NEUROG1, delaminate from the otocyst, and coalesce to form the n
Externí odkaz:
https://doaj.org/article/6749d50a70094a27bf463aab0b4541e3
Astroglia are the most abundant glia cell in the central nervous system, playing essential roles in maintaining homeostasis. Key functions of astroglia include, but are not limited to, neurotransmitter recycling, ion buffering, immune modulation, neu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::831c133554d0fd6a73d9e50feea3b293
https://europepmc.org/articles/PMC6444185/
https://europepmc.org/articles/PMC6444185/
Autor:
Courtney C. Kellogg, Walter C. Low, Aleta R. Steevens, Jenna C. Glatzer, Peter A. Santi, Amy E. Kiernan
Publikováno v:
Development
The transcription factor sex determining region Y-box 2 (SOX2) is required for the formation of hair cells and supporting cells in the inner ear and is a widely used sensory marker. Paradoxically, we demonstrate via fate mapping that, initially, SOX2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2944103097a0b6c53b496bdb024e5584
https://pubmed.ncbi.nlm.nih.gov/31152002
https://pubmed.ncbi.nlm.nih.gov/31152002
Publikováno v:
Journal of neurogenetics. 31(1-2)
Astroglia are a morphologically diverse and highly abundant cell type in the CNS. Despite these obvious observations, astroglia still remain largely uncharacterized at the cellular and molecular level. In disease contexts such as amyotrophic lateral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::307488a3349253d630cc87ddc17eb2cf
https://pubmed.ncbi.nlm.nih.gov/28019127
https://pubmed.ncbi.nlm.nih.gov/28019127
Autor:
Jenna C. Glatzer, Jennifer Stocksdale, Laura P.W. Ranum, Wenzhen Duan, Nicolas Arbez, Ke Zhang, Jonathan C. Grima, Qi Peng, Juan C. Troncoso, Jeffrey D. Rothstein, Christopher A. Ross, Leslie M. Thompson, Kathleen C. Cunningham, J. Gavin Daigle, Harsh Wadhwa, Ishrat Ahmed, Charlene Geater, Solomon H. Snyder, Eva L. Morozko, Jacqueline T. Pham, Olga Pletnikova, Joseph Ochaba, Thomas E. Lloyd
Publikováno v:
Neuron, vol 94, iss 1
Huntington's disease (HD) is caused by an expanded CAG repeat in the Huntingtin (HTT) gene. The mechanism(s) by which mutant HTT (mHTT) causes disease is unclear. Nucleocytoplasmic transport, the trafficking of macromolecules between the nucleus and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::339f602c08d978be2345257b2d829e9d
https://doi.org/10.1016/j.neuron.2017.03.023
https://doi.org/10.1016/j.neuron.2017.03.023