Zobrazeno 1 - 10
of 93
pro vyhledávání: '"Jeffrey S. Gilbert"'
Autor:
Connor F. Laule, Cameron R. Wing, Evan J. Odean, Jacob A. Wilcox, Jeffrey S. Gilbert, Jean F. Regal
Publikováno v:
Journal of Immunotoxicology, Vol 14, Iss 1, Pp 235-240 (2017)
Preeclampsia is a pregnancy-specific condition manifested by new-onset maternal hypertension with systemic inflammation, including increased innate immune system complement activation. While exact pathophysiology is unknown, evidence suggests that in
Externí odkaz:
https://doaj.org/article/7d2f0ada378d46308c646fbef8f641a0
Autor:
Kendra J. Towner, Cameron R. Wing, Sherry D. Fleming, Connor F. Laule, Kate M. Root, Jean F. Regal, Evan Odean, Jeffrey S. Gilbert, Cassandra M Hamm
Publikováno v:
Am J Physiol Heart Circ Physiol
Preeclampsia is a prevalent pregnancy complication characterized by new-onset maternal hypertension and inflammation, with placental ischemia as the initiating event. Studies of others have provided evidence for the importance of lymphocytes in place
Publikováno v:
Am J Physiol Heart Circ Physiol
Autor:
Lynne T. Bemis, Sherry D. Fleming, Kate M. Root, Cameron R. Wing, Jenna M. Lund, Jean F. Regal, Jeffrey S. Gilbert, Luke McCutcheon
Publikováno v:
Mol Immunol
Preeclampsia is characterized by new onset hypertension and fetal growth restriction and is associated with aberrant activation of the innate immune complement system and stressed or ischemic placenta. Previous studies have suggested a role for both
Autor:
Jeffrey S. Gilbert, Noel Fernando Nieto, Jean F. Regal, Megan E. Strehlke, Cameron R. Wing, Jenna M. Peterson, Lynne T. Bemis, Sherry D. Fleming, Jordan E. Parker
Publikováno v:
Molecular Immunology. 78:38-47
Preeclampsia is characterized by development of hypertension during pregnancy and reduced placental perfusion. Previous studies in a rat model of placental ischemia-induced hypertension demonstrated that inhibiting complement activation attenuated in
Autor:
Evan Odean, Sherry D. Fleming, Jeffrey S. Gilbert, Cameron R. Wing, Connor F. Laule, Jean F. Regal
Publikováno v:
The FASEB Journal. 32
Publikováno v:
Hypertension. 70
Preeclampsia is characterized by hypertension, reduced placental perfusion, intrauterine growth restriction and increased activation of complement, part of innate immunity. Using the Reduced Uterine Perfusion Pressure (RUPP) model of placental ischem
Autor:
Ashley J. Bauer, Ronald R. Regal, Jenna M. Peterson, Jeffrey S. Gilbert, Jean F. Regal, Jonathan W. Opacich, Alex C. Loeks-Johnson, Kathryn E Lillegard, Barbara J. Elmquist
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 351:344-351
Early-onset pre-eclampsia is characterized by decreased placental perfusion, new-onset hypertension, angiogenic imbalance, and endothelial dysfunction associated with excessive activation of the innate immune complement system. Although our previous
Autor:
Henry Marsh, Sarah J. Lojovich, Ashley J. Bauer, Alex C. Johnson, Jeffrey S. Gilbert, Kathryn E Lillegard, Jean F. Regal
Publikováno v:
Molecular Immunology. 56:91-97
Preeclampsia is a major obstetric problem defined by new-onset hypertension and proteinuria associated with compromised placental perfusion. Although activation of the complement system is increased in preeclampsia compared to normal pregnancy, it re
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 304:H1159-H1165
Previous studies suggest restoration of angiogenic balance can lower blood pressure and improve vascular endothelium function in models of preeclampsia. Our laboratory has recently reported exercise training mitigates hypertension in an animal model