Zobrazeno 1 - 10
of 112
pro vyhledávání: '"Jeffrey D, Ritzenthaler"'
Autor:
Glenn W. Vicary, Jeffrey D. Ritzenthaler, Tanmay S. Panchabhai, Edilson Torres-González, Jesse Roman
Publikováno v:
Respiratory Research, Vol 18, Iss 1, Pp 1-12 (2017)
Abstract Background Tobacco-related chronic lung diseases are characterized by alterations in lung architecture leading to decreased lung function. Knowledge of the exact mechanisms involved in tobacco-induced tissue remodeling and inflammation remai
Externí odkaz:
https://doaj.org/article/36a411bfed48487c8200b57cf0d3ed3b
Supplementary Figure 5A-D from Nicotine Stimulates PPARβ/δ Expression in Human Lung Carcinoma Cells through Activation of PI3K/mTOR and Suppression of AP-2α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0912ad713ec5e730818d6322ce8e2cc1
https://doi.org/10.1158/0008-5472.22379268
https://doi.org/10.1158/0008-5472.22379268
Supplementary Figure 6A-C from Nicotine Stimulates PPARβ/δ Expression in Human Lung Carcinoma Cells through Activation of PI3K/mTOR and Suppression of AP-2α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f91c0421449f498d8c65bb4c35245e2
https://doi.org/10.1158/0008-5472.22379265.v1
https://doi.org/10.1158/0008-5472.22379265.v1
Supplementary Figure 2A-B from Nicotine Stimulates PPARβ/δ Expression in Human Lung Carcinoma Cells through Activation of PI3K/mTOR and Suppression of AP-2α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13af89cc9ca99812c633d2f6bc5f5860
https://doi.org/10.1158/0008-5472.22379277.v1
https://doi.org/10.1158/0008-5472.22379277.v1
We previously showed that nicotine stimulates non–small cell lung carcinoma (NSCLC) cell proliferation through nicotinic acetylcholine receptor (nAChR)–mediated signals. Activation of peroxisome proliferator–activated receptor β/δ (PPARβ/δ)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ead7501624713b67075862e40418ff85
https://doi.org/10.1158/0008-5472.c.6499194.v1
https://doi.org/10.1158/0008-5472.c.6499194.v1
Supplementary Figure 4 from Nicotine Stimulates PPARβ/δ Expression in Human Lung Carcinoma Cells through Activation of PI3K/mTOR and Suppression of AP-2α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41b5bfba1343b36ae3d8044295dcdb0f
https://doi.org/10.1158/0008-5472.22379271
https://doi.org/10.1158/0008-5472.22379271
Supplementary Figure 3 from Nicotine Stimulates PPARβ/δ Expression in Human Lung Carcinoma Cells through Activation of PI3K/mTOR and Suppression of AP-2α
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::545e1794d0640a2b8ee4876a55dc7af6
https://doi.org/10.1158/0008-5472.22379274
https://doi.org/10.1158/0008-5472.22379274
Autor:
Jeffrey D. Ritzenthaler, Edilson Torres-Gonzalez, Yuxuan Zheng, Igor N. Zelko, Victor van Berkel, David R. Nunley, Biniam Kidane, Andrew J. Halayko, Ross Summer, Walter H. Watson, Jesse Roman
Publikováno v:
Am J Physiol Lung Cell Mol Physiol
Increased senescence and expression of profibrotic genes in old lung fibroblasts contribute to disrepair responses. We reported that primary lung fibroblasts from old mice have lower expression and activity of the cystine transporter Slc7a11/xCT than
Autor:
Juliane I. Beier, Luping Guo, Swati Joshi-Barve, Jeffrey D. Ritzenthaler, Jesse Roman, Gavin E. Arteel
Publikováno v:
Annals of Hepatology, Vol 15, Iss 5, Pp 762-772 (2016)
Background. The regenerative capacity of the liver is critical for proper responses to injury. Fibrin extracellular matrix (ECM) deposition is a common response to insult and contributes to inflammatory liver injury. However, the role of this matrix
Externí odkaz:
https://doaj.org/article/ec5ba9c02e124c2eb11fb40091d20247
Publikováno v:
Redox Biology, Vol 8, Iss C, Pp 305-315 (2016)
Pulmonary fibrosis affects millions worldwide and, even though there has been a significant investment in understanding the processes involved in wound healing and maladaptive repair, a complete understanding of the mechanisms responsible for lung fi
Externí odkaz:
https://doaj.org/article/4b32acb5f59c4ee581afa90244f44bf7