Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Jeffrey A. Canter"'
Autor:
Jeffrey A. Canter, Carol Lyons, Amy Saupe, Yuki Bradford, Nicole Comstock, Katie Garman, Timothy F. Jones, Rajal K. Mody, Sarah L. Lathrop, Lana M. Olson, Asha R. Kallianpur, Amanda Palmer, Katie Wymore
Publikováno v:
The Journal of Infectious Diseases. 217:1000-1010
Background Postdiarrheal hemolytic-uremic syndrome (D+HUS) following Shiga toxin-producing Escherichia coli (STEC) infection is a serious condition lacking specific treatment. Host immune dysregulation and genetic susceptibility to complement hyperac
Autor:
Jeffrey A Canter, Lana M Olson, Kylee Spencer, Nathalie Schnetz-Boutaud, Brent Anderson, Michael A Hauser, Silke Schmidt, Eric A Postel, Anita Agarwal, Margaret A Pericak-Vance, Paul Sternberg, Jonathan L Haines
Publikováno v:
PLoS ONE, Vol 3, Iss 5, p e2091 (2008)
The objective of this study was to determine if MTND2*LHON4917G (4917G), a specific non-synonymous polymorphism in the mitochondrial genome previously associated with neurodegenerative phenotypes, is associated with increased risk for age-related mac
Externí odkaz:
https://doaj.org/article/2006c475988f4c4e8018aaaafa4d5623
Autor:
Benjamin J, Grady, David C, Samuels, Gregory K, Robbins, Doug, Selph, Jeffrey A, Canter, Richard B, Pollard, David W, Haas, Robert, Shafer, Spyros A, Kalams, Deborah G, Murdock, Marylyn D, Ritchie, Todd, Hulgan, Martin, Hirsch
Publikováno v:
JAIDS Journal of Acquired Immune Deficiency Syndromes. 58:363-370
Mitochondrial DNA (mtDNA) variation has been associated with time to progression to AIDS and adverse effects from antiretroviral therapy (ART). In this study, full mitochondrial DNA (mtDNA) sequence data from US-based adult participants in the AIDS C
Autor:
Marylyn D. Ritchie, Richard Haubrich, David W. Haas, Grace A. McComsey, Pablo Tebas, Jeffrey A. Canter, Todd Hulgan, Sharon A. Riddler
Publikováno v:
AIDS. 25:37-47
Mitochondrial DNA (mtDNA) influences metabolic diseases and perhaps antiretroviral therapy (ART) complications. We explored associations between European mtDNA haplogroups and metabolic changes among A5142 participants.Seven hundred and fifty-seven A
Publikováno v:
Journal of Orthopaedic Trauma. 24:530-533
Objectives: Heterotopic Ossification (HO) is a significant complication after trauma occurring in 12% to 25% of fractures. Although clinical predictors have been studied to determine the likelihood of developing HO, the results have been inconsistent
Autor:
Kimberly G. Norman, Ginger L. Milne, Jeffrey A. Canter, Jason D. Morrow, Mingjian Shi, James E. Sligh
Publikováno v:
Mitochondrion. 10:94-101
Transplant recipients have an elevated risk of skin cancer, with a 65- to 250-fold increase in squamous cell carcinoma. Usage of the immunosuppressant cyclosporine A (CsA) is associated with the development of skin cancer. We hypothesized that the in
Autor:
Jeffrey A. Canter, Maria Styczyńska, Maria Barcikowska, Cezary Zekanowski, Aleksandra Maruszak
Publikováno v:
Neurobiology of Aging. 30:1749-1755
We evaluated the involvement of the major Caucasian-specific mitochondrial haplogroups (H, I, J, K, T, U, V, W and X), haplogroup clusters (HV, UK, TJ, IWX) and two functional mtSNPs (4216, 4917) in the pathogenesis of Alzheimer's disease (AD) in the
Autor:
Jeffrey A. Canter, Judith M. Jenkins, Jason H. Moore, Anna E. Williams, Patrick R. Norris, John A. Morris
Publikováno v:
Annals of Surgery. 250:524-530
Objective: Personalized medicine merges genetics, physiology, and patient outcome. Loss of physiologic complexity (heart rate [HR] variability) is a bedside biomarker for autonomic nervous system (ANS) dysfunction. We hypothesized that variability in
Autor:
Jeffrey A. Canter, John A. Morris, Anna E. Williams, Patrick R. Norris, Jason H. Moore, Judith M. Jenkins
Publikováno v:
Journal of the American College of Surgeons. 208:663-668
Background We have previously demonstrated an unappreciated link between the autonomic nervous system and mortality, heart rate variability, and physiologic complexity. Study Design Genetic variation in adrenergic receptors or key enzymes in catechol
Autor:
John A. Morris, Jeffrey A. Canter, Anna E. Williams, Brent S. McNew, Judith M. Jenkins, Marshall L. Summar, Patrick R. Norris, Bryan A. Cotton, Cedric Francois, Paul Olson, Jason H. Moore
Publikováno v:
Journal of Trauma: Injury, Infection & Critical Care. 66:1265-1272
Background: Trauma is a disease of inflammation. Complement Component 2 (C2) is a protease involved in activation of complement through the classical pathway and has been implicated in a variety of chronic inflammatory diseases. We hypothesized that