Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Jedd B. Sereysky"'
Publikováno v:
Journal of Orthopaedic Research. 30:1327-1334
Accumulation of sub-rupture fatigue damage has been implicated in the development of tendinopathy. We previously developed an in vivo model of damage accumulation using the rat patellar tendon. Our model allows us to control the input loading paramet
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e7251c6dfb24b6fd06294b26e92a9023
https://doi.org/10.1002/jor.22059
https://doi.org/10.1002/jor.22059
Publikováno v:
Journal of Orthopaedic Research. 30:965-972
The purpose of this study was to develop and validate an in vivo mouse model of tendon fatigue and use this model to investigate and quantify the physical manifestations of fatigue damage in mouse tendon. Patellar tendons of C57BL/6J mice were fatigu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4656a4bfe79eb353d9544ee56f745156
https://doi.org/10.1002/jor.22012
https://doi.org/10.1002/jor.22012
Autor:
Vincent M. Wang, Jonathan P. Braman, Jelena Basta-Pljakic, Jonathan Y. Lee, Daniel J. Leong, Mitch B. Schaffler, Hui B. Sun, Yonghui Li, David T. Fung, Raymond A. Klug, Evan L. Flatow, Nelly Andarawis-Puri, Jedd B. Sereysky, Stephen J. Ros, Karl J. Jepsen
Publikováno v:
Journal of Orthopaedic Research. 28:1380-1386
Expression profiling of selected matrix remodeling genes was conducted to evaluate differences in molecular response to low-cycle (100) and high-cycle (7,200) sub-failure-fatigue loading of patellar tendons. Using our previously developed in vivo pat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::452329d259dbf4f1520839f9a56b9f43
https://doi.org/10.1002/jor.21132
https://doi.org/10.1002/jor.21132
Publikováno v:
International journal of experimental pathology. 94(4)
Tendinopathies are common muskoloskeletal injuries that lead to pain and disability. Development and pathogenesis of tendinopathy is attributed to progressive pathological changes to the structure, function, and biology of tendon. The nature of this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::502a04dbdf20fd21028a0bd340ddd239
https://pubmed.ncbi.nlm.nih.gov/23772908
https://pubmed.ncbi.nlm.nih.gov/23772908
Publikováno v:
Journal of biomechanics. 45(1)
Damage accumulation underlies tendinopathy. Animal models of overuse injuries do not typically control loads applied to the tendon. Our in vivo model in the rat patellar tendon allows direct control of the loading applied to the tendon. Despite this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef640c4cb9df7e1206f7f5664b5712c4
https://pubmed.ncbi.nlm.nih.gov/22055428
https://pubmed.ncbi.nlm.nih.gov/22055428
Tendon pathology is frequently sub-clinical prior to frank rupture, denoting the need for non-destructive methods of assessing disease presence and progression. Despite the lack of clinical presentation, previous studies have observed that distinct c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a818588b7932c0539c5023b0d120de5a
https://europepmc.org/articles/PMC2937080/
https://europepmc.org/articles/PMC2937080/
Autor:
Karl J. Jepsen, Damien M. Laudier, David T. Fung, Mitchell B. Schaffler, Rumana Huq, Evan L. Flatow, Jelena Basta-Pljakic, Jedd B. Sereysky
Publikováno v:
Annals of biomedical engineering. 38(5)
Conventional histologic methods provide valuable information regarding the physical nature of damage in fatigue-loaded tendons, limited to thin, two-dimensional sections. We introduce an imaging method that characterizes tendon microstructure three-d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c589d78e524fe40456518fa7f2043d7f
https://pubmed.ncbi.nlm.nih.gov/20232150
https://pubmed.ncbi.nlm.nih.gov/20232150
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 285(3)
Programmed electrical stimulation (PES) is a crucial aspect of the evaluation of the risk of arrhythmias in cardiac patients and provides a powerful tool for understanding the mechanisms of arrhythmia in experimental models. Whereas PES in the mouse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ebb34a2d4b310d5d10b9437b2c25da57
https://pubmed.ncbi.nlm.nih.gov/12750061
https://pubmed.ncbi.nlm.nih.gov/12750061