Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Jean-François Séité"'
Publikováno v:
Arthritis & Rheumatology. 67:595-603
Autor:
Achouak Achour, Audrey Mohr, Quentin Simon, Ibtissem Ghedira, Boutahar Bendaoud, Pierre Youinou, Christophe Jamin, Jean-François Séité, Jacques-Olivier Pers
Publikováno v:
Journal of Allergy and Clinical Immunology
Journal of Allergy and Clinical Immunology, Elsevier, 2017, ⟨10.1016/j.jaci.2016.09.042⟩
Journal of Allergy and Clinical Immunology, Elsevier, 2017, ⟨10.1016/j.jaci.2016.09.042⟩
Background Follicular helper T (T FH ) cells support terminal B-cell differentiation. Human regulatory B (Breg) cells modulate cellular responses, but their control of T FH cell–dependent humoral immune responses is unknown. Objective We sought to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b87e4d9b9c3756e77c22d1181feb208
https://hal.archives-ouvertes.fr/hal-01492613
https://hal.archives-ouvertes.fr/hal-01492613
Publikováno v:
Autoimmunity Reviews. 7:435-439
Intravenous immunoglobulins (IVIg) consist of IgG concentrated from pooled plasma of numerous donors. Their contents is variable, depending in part on the method of preparation. Natural antibodies and natural autoantibodies are prominent in these pre
Autor:
Achouak, Achour, Quentin, Simon, Audrey, Mohr, Jean-François, Séité, Pierre, Youinou, Boutahar, Bendaoud, Ibtissem, Ghedira, Jacques-Olivier, Pers, Christophe, Jamin
Publikováno v:
The Journal of allergy and clinical immunology. 140(1)
Follicular helper T (TWe sought to assess the role of Breg cells on THuman T cells were polyclonally stimulated in the presence of IL-12 and IL-21 to generate TB-cell lymphoma 6, IL-21, inducible costimulator, CXCR5, and programmed cell death protein
Autor:
Christophe Jamin, Divi Cornec, Thomas Guerrier, Jean-François Séité, Pierre Youinou, Sophie Hillion
Publikováno v:
Journal of Autoimmunity
Journal of Autoimmunity, Elsevier, 2011, 37 (3), pp.190-7. ⟨10.1016/j.jaut.2011.05.014⟩
Journal of Autoimmunity, Elsevier, 2011, 37 (3), pp.190-7. ⟨10.1016/j.jaut.2011.05.014⟩
International audience; One way for intravenous Ig (IVIg) to affect responses of the B cells might be to operate through their TLR7 and TLR9. We confirm the ability of TLR agonists to induce CD25 expression in B cells. For this to occur, sialylated F
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cbcd45e670a36c61c5e6b97877834b20
https://hal.univ-brest.fr/hal-00771231
https://hal.univ-brest.fr/hal-00771231
Autor:
Pierre Youinou, Yves Renaudineau, Rizgar A. Mageed, Divi Cornec, Sophie Hillion, Jean-François Séité
Publikováno v:
Blood
Blood, American Society of Hematology, 2010, 116 (10), pp.1698-704. ⟨10.1182/blood-2009-12-261461⟩
Blood, American Society of Hematology, 2010, 116 (10), pp.1698-704. ⟨10.1182/blood-2009-12-261461⟩
Among various mechanisms for interactions with B cells, intravenous immunoglobulin (IVIg) may operate through the insertion of its Fc part into the Fc-γ receptor, or the binding of its sialic acid (SA)–bearing glycans to the negatively regulating
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f48e5e21038672a62b0398d76010bcec
https://hal.archives-ouvertes.fr/hal-01150519/file/JFSDC.pdf
https://hal.archives-ouvertes.fr/hal-01150519/file/JFSDC.pdf
Intravenous immunoglobulin induces a functional silencing program similar to anergy in human B cells
Publikováno v:
Journal of Allergy and Clinical Immunology
Journal of Allergy and Clinical Immunology, Elsevier, 2014, 133 (1), pp.181-188.e9. ⟨10.1016/j.jaci.2013.08.042⟩
Journal of Allergy and Clinical Immunology, Elsevier, 2014, 133 (1), pp.181-188.e9. ⟨10.1016/j.jaci.2013.08.042⟩
International audience; BACKGROUND: Chronic inflammatory and autoimmune diseases are largely due to inappropriate response of hyperactive or autoreactive B cells. These autoreactive B cells can evade central tolerance checkpoints and migrate to the p
Publikováno v:
Annals of the Rheumatic Diseases. 69:A48-A48
Although widely used to treat inflammatory diseases, the mechanism by which intravenous immunoglobulin (IVIg) modulates immune function is not fully understood. B cells from the blood of healthy volunteers and tonsils …
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