Zobrazeno 1 - 10
of 199
pro vyhledávání: '"Jean Sébastien Silvestre"'
Autor:
Philippe Foubert, Claire Squiban, Valérie Holler, Valérie Buard, Carole Dean, Bernard I. Levy, Marc Benderitter, Jean Sébastien Silvestre, Gérard Tobelem, Radia Tamarat
Publikováno v:
Cell Transplantation, Vol 24 (2015)
Endothelial progenitor cell (EPC) transplantation has beneficial effects for therapeutic neovascularization. We therefore assessed the effect of a therapeutic strategy based on EPC administation in the healing of radiation-induced damage. To improve
Externí odkaz:
https://doaj.org/article/b6fd1fa60a3e42ceb4886275b12546ed
Autor:
Manon Desgres, Bruna Lima Correa, Lorena Petrusca, Gwennhael Autret, Chloé Pezzana, Céline Marigny, Chloé Guillas, Valérie Bellamy, José Vilar, Marie-Cécile Perier, Florent Dingli, Damarys Loew, Camille Humbert, Jérôme Larghero, Guillaume Churlaud, Nisa Renault, Pierre Croisille, Albert Hagège, Jean-Sébastien Silvestre, Philippe Menasché
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 10 (2023)
BackgroundCurrent treatments of chemotherapy-induced cardiomyopathy (CCM) are of limited efficacy. We assessed whether repeated intravenous injections of human extracellular vesicles from cardiac progenitor cells (EV-CPC) could represent a new therap
Externí odkaz:
https://doaj.org/article/5374d51a15114ea6a6d1bd5f0f274719
Comment créer des vaisseaux de bonne qualité dans un territoire en souffrance et comment restaurer la perfusion sanguine dans un territoire ischémique? De nombreux types cellulaires initient des multitudes de mécanismes moléculaires qui particip
Autor:
Jean-Sébastien Silvestre
Publikováno v:
Cardiovascular Research. 119:1097-1099
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d8ba3fc0523fd51d937e6f9fc8a3e1bd
https://doi.org/10.1093/cvr/cvad051
https://doi.org/10.1093/cvr/cvad051
Autor:
Emilie Simonnet, Sabrina Martin, José Vilar, Emilie Vessieres, Sonia Taib, Virginie Monceau, Luc Pardanaud, Nadine Bouby, Anne Eichmann, Jean-Sébastien Silvestre, Daniel Henrion, Isabelle Brunet
The autonomic sympathetic nervous system innervates peripheral resistance arteries, thereby controlling arterial diameter and modulating blood supply to organs and arterial tone. Despite its fundamental role in blood flow regulation and adaptive resp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::be6d587d26e8933dc0aaedf8e6352902
https://doi.org/10.1101/2023.02.03.526852
https://doi.org/10.1101/2023.02.03.526852
Autor:
Pierre Nivoit, Thomas Mathivet, Junxi Wu, Yann Salemkour, Devanarayanan Siva Sankar, Véronique Baudrie, Jennifer Bourreau, Anne-Laure Guihot, Emilie Vessières, Mathilde Lemitre, Cinzia Bocca, Jérémie Teillon, Morgane Le Gall, Anna Chipont, Estelle Robidel, Neeraj Dhaun, Eric Camerer, Pascal Reynier, Etienne Roux, Thierry Couffinhal, Patrick Hadoke, Jean-sébastien Silvestre, Xavier Guillonneau, Philippe Bonnin, Daniel Henrion, Jörn Dengjel, Pierre-Louis Tharaux, Olivia Lenoir
Dysregulated autophagy is associated with cardiovascular and metabolic diseases, where impaired flow-mediated endothelial cell responses promote cardiovascular risk. The mechanism by which the autophagy machinery regulates endothelial functions is co
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c237327fa47b8078f174921ac2ebec32
https://doi.org/10.21203/rs.3.rs-2276541/v1
https://doi.org/10.21203/rs.3.rs-2276541/v1
Autor:
Simon Bond, Joseph Cheriyan, Jean-Sébastien Silvestre, Brian Y.H. Lam, Yanyi Sun, James H.F. Rudd, Yuning Lu, Marcella Ma, Ziad Mallat, Meritxell Nus, James Harrison, Rouchelle Sriranjan, Andrew S. Sage, Xian Yu, Stephen A. Newland, Tian X. Zhao, Xiang Cheng
Publikováno v:
Journal of the American College of Cardiology. 78:1127-1142
BACKGROUND: Innate lymphoid cells type 2 (ILC2s) play critical homeostatic functions in peripheral tissues. ILC2s reside in perivascular niches and limit atherosclerosis development. OBJECTIVES: ILC2s also reside in the pericardium but their role in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b07755d03b10bcf70102a869a933313
https://doi.org/10.1016/j.jacc.2021.07.018
https://doi.org/10.1016/j.jacc.2021.07.018
Autor:
Yujiao ZHANG, Marie Vandestienne, Jean-Rémi Lavillegrand, Jeremie Joffre, Icia Santos-Zas, Aonghus Lavelle, Xiadan Zhong, Wilfried Le Goff, Maryse Guerin, Olivia Lenoir, Ludivine Laurans, Patrick Bruneval, Coralie Guérin, Marc Diedisheim, Melanie Migaud, Anne Puel, Fanny Lanternier, Jean-Laurent Casanova, Clement Cochain, Alma Zernecke, Antoine-Emmanuel Saliba, Jean-sébastien Silvestre, Alain Tedgui, Ziad Mallat, Soraya Taleb, Cecile Vindis, Stephane Camus, Harry Sokol, Hafid Ait-Oufella
Macrophage-mediated innate immune responses contribute to the initiation, progression and complications of atherosclerosis. However, the underlying pathways linking activation of macrophages to atherosclerotic plaque develoment are still poorly under
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::afd45fa10f871af865755747d3bb3529
https://doi.org/10.21203/rs.3.rs-1834549/v2
https://doi.org/10.21203/rs.3.rs-1834549/v2
Autor:
Clément Delacroix, Alexandra Achab-Ali, Paul Alayrac, Marine Gandon-Renard, Fatou Dramé, David Sassoon, Jean-Sébastien Silvestre, Jean-Sébastien Hulot
BackgroundIntegrins are surface receptors that bind to extracellular matrix ligands and regulate cellular function through mechanical stress-initiated signal transduction. Integrin alpha V (or CD51) is implicated in myocardial fibrosis and anti-CD51
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0cb7c01f653c6665a473e1779167c49e
https://doi.org/10.1101/2022.10.10.511547
https://doi.org/10.1101/2022.10.10.511547
Publikováno v:
médecine/sciences
médecine/sciences, 2022, 38 (10), pp.766-768. ⟨10.1051/medsci/2022119⟩
médecine/sciences, 2022, 38 (10), pp.766-768. ⟨10.1051/medsci/2022119⟩
International audience; No abstract available
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::109e0024bb8f475442bbcd66764fc410
https://hal.science/hal-03811510
https://hal.science/hal-03811510