Zobrazeno 1 - 10
of 66
pro vyhledávání: '"Jean Mérot"'
Autor:
Constance Delwarde, Romain Capoulade, Jean Mérot, Solena Le Scouarnec, Nabila Bouatia-Naji, Mengyao Yu, Olivier Huttin, Christine Selton-Suty, Jean-Marc Sellal, Nicolas Piriou, Jean-Jacques Schott, Christian Dina, Thierry Le Tourneau
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 10 (2023)
Mitral valve prolapse (MVP) is a common condition affecting 2–3% of the general population, and the most complex form of valve pathology, with a complication rate up to 10–15% per year in advanced stages. Complications include mitral regurgitatio
Externí odkaz:
https://doaj.org/article/04a49479f9784ecb80f4524ff2bcf9d1
Autor:
Damien Duval, Pauline Labbé, Léa Bureau, Thierry Le Tourneau, Russell A. Norris, Roger R. Markwald, Robert Levine, Jean-Jacques Schott, Jean Mérot
Publikováno v:
Journal of Cardiovascular Development and Disease, Vol 2, Iss 3, Pp 233-247 (2015)
Although the genetic basis of mitral valve prolapse (MVP) has now been clearly established, the molecular and cellular mechanisms involved in the pathological processes associated to a specific mutation often remain to be determined. The FLNA gene (e
Externí odkaz:
https://doaj.org/article/7fd05b35d0354b49b4832a66391294c7
Autor:
Fabien C Coyan, Fayal Abderemane-Ali, Mohamed Yassine Amarouch, Julien Piron, Jérôme Mordel, Céline S Nicolas, Marja Steenman, Jean Mérot, Céline Marionneau, Annick Thomas, Robert Brasseur, Isabelle Baró, Gildas Loussouarn
Publikováno v:
PLoS ONE, Vol 9, Iss 3, p e93255 (2014)
INTRODUCTION:Phosphatidylinositol-4,5-bisphosphate (PIP2) is a cofactor necessary for the activity of KCNQ1 channels. Some Long QT mutations of KCNQ1, including R243H, R539W and R555C have been shown to decrease KCNQ1 interaction with PIP2. A previou
Externí odkaz:
https://doaj.org/article/2457806624d04ecea1b5be87c18b48d5
Autor:
Marine Sallé, Pascal Aumond, Benjamin Le Vely, Guillaume Guimbretière, Cédric Lemay, Bertrand Cariou, Thierry Le Tourneau, Claire Toquet, Jean Mérot, Jean-Christian Roussel, Romain Capoulade
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 15:221
Autor:
Benjamin Le Vely, Constance Delwarde, Pascal Aumond, Marine Sallé, Mounia Elhannani, Thierry Le Tourneau, Claire Toquet, Jean Mérot, Elvira Mass, Romain Capoulade
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 15:222
Autor:
Constance Delwarde, Benjamin Le Vély, Amir H. Kayvanjoo, Pascal Aumond, Séverine Remy, Laurent Monassier, Claire Toquet, Jean-Jacques Schott, Thierry Le Tourneau, Elvira Mass, Jean Mérot, Romain Capoulade
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 14:204
Autor:
S Le Scouarnec, T. Le Tourneau, Jean-Jacques Schott, Nicolas Piriou, Jean Mérot, Caroline Cueff, R. Capoulade, Marie Marrec
Publikováno v:
European Heart Journal - Cardiovascular Imaging. 21
Funding Acknowledgements PHRCI mitral 2012 Background Filamin-A mitral valve prolapse/dystrophy (FLNA-MVP) phenotype associates moderate MVP and a paradoxical restricted motion in diastole. Purpose We aimed to assess the association of MVP with restr
Autor:
Amir Hossein Kayvanjoo, T. Le Tourneau, S Blandin, R. Capoulade, Claire Toquet, I Anegon, S Remy, B. Lauzier, Jean-Jacques Schott, Elvira Mass, Jean Mérot, P. Aumond, J Veziers, C. Delwarde
Publikováno v:
Archives of Cardiovascular Diseases Supplements. 14:60
Introduction Mitral Valve Prolapse (MVP) affects 3% of the population and is characterized by a heterogeneous mitral leaflet remodeling. The pathophysiological mechanisms involved in MVP development are not fully understood, the only therapeutic opti
Autor:
Maarit Hellman, Perttu Permi, Ulla Pentikäinen, Simon Lecointe, Tatu Haataja, Jean Mérot, Romain Capoulade
Publikováno v:
Biophysical Journal
Biophysical Journal, Biophysical Society, 2019, 117 (8), pp.1467-1475. ⟨10.1016/j.bpj.2019.08.032⟩
Biophys J
Biophysical Journal, Biophysical Society, 2019, 117 (8), pp.1467-1475. ⟨10.1016/j.bpj.2019.08.032⟩
Biophys J
Mitral valve diseases affect approximately 3% of the population and are the most common reasons for valvular surgery because no drug-based treatments exist. Inheritable genetic mutations have now been established as the cause of mitral valve insuffic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::305fa7a967fff21b0c0cbc2ac4265068
https://hal.archives-ouvertes.fr/hal-02332272
https://hal.archives-ouvertes.fr/hal-02332272
Autor:
Caroline Cueff, T. Le Tourneau, Nicolas Piriou, Jean Mérot, S Le Scouarnec, R. Capoulade, Jean-Jacques Schott
Publikováno v:
European Heart Journal. 40
Background Filamin-A mitral valve prolapse/dystrophy (FLNA-MVP) phenotype associates MVP and a paradoxical restricted motion in diastole. Purpose We aim to assess the association of mitral valve prolapse to restricted motion in diastole in MVP patien