Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Jean Briere"'
Autor:
Jean Briere
Publikováno v:
Bulletin de l'Académie Nationale de Médecine. 191:535-548
An increased platelet number in blood depends on a limited spectrum of causes, which aren't always simple to identify. Secondary thrombocytosis is a reactive process in relation with acute or chronic inflammatory diseases, or asplenia. The infrequent
Autor:
Devinder Gill, E. Van Den Neste, B. Glass, Dc Linch, Christian Gisselbrecht, Noel-Jean Milpied, Catherine Thieblemont, John Radford, Craig H. Moskowitz, Marek Trneny, David D.F. Ma, Ofer Shpilberg, Gilles Salles, Jean Briere, Nicolas Ketterer, Nicolas Mounier, Norbert Schmitz, Ulrich Dührsen
Publikováno v:
Bone marrow transplantation. 51(1)
Salvage chemotherapy followed by autologous stem cell transplantation (ASCT) is the standard second-line treatment for relapsed and refractory diffuse large B-cell lymphoma (DLBCL). However, the strategy is less clear in patients who require third-li
Autor:
Jean-François Bernard, Jean-Didier Rain, Jean-Jacques Kiladjian, Christine Chomienne, Jean Briere, Pierre Fenaux
Publikováno v:
Seminars in Thrombosis and Hemostasis. 32:417-421
Despite recent discoveries made in myeloproliferative disorders other than chronic myelogenous leukemia, which it is hoped will result in earlier diagnosis, and better evaluation and management of patients, hematological evolution to myelofibrosis, a
Autor:
Nicola Vahsen, Pier G. Mastroberardino, Frank Madeo, Nicholas Joza, Noelia Casares, Guido Kroemer, Silvia Engelhardt, Najet Debili, Silke Wissing, Hermann Schägger, Paule Bénit, Pierre Rustin, Olivier Feraud, Vladimir Lazar, Josef M. Penninger, Marie O. Pequignot, Jean Briere, Nathanael Larochette, Céline Candé, Mauro Piacentini
Publikováno v:
The EMBO Journal. 23:4679-4689
Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein that, after apoptosis induction, translocates to the nucleus where it participates in apoptotic chromatinolysis. Here, we show that human or mouse cells lacking AIF as a result of homolo
Publikováno v:
Pathologie Biologie. 52:289-293
Despite a prolonged survival of around 15 years linked to a prolonged complete remission induced by myelosuppression, myeloproliferative syndromes such as polycythemia vera (PV) and essential thrombosis (ET) remain at risk of lethal adverse affects s
Autor:
Yasmine Chait, Annie Bezeaud, Marie-Hélène Denninger, Dominique Valla, Marie-Claude Guillin, Jean Briere, Serge Erlinger, Sophie Hillaire, Nicole Casadevall
Publikováno v:
Hepatology. 31:587-591
According to a recent hypothesis, venous thrombosis results from the concurrence of several factors. This hypothesis was assessed in patients with portal or hepatic venous thrombosis by simultaneously investigating most of the currently identified pr
Autor:
Jean Briere, N. Elkassar
Publikováno v:
Baillière's Clinical Haematology. 11:787-801
Our current understanding of the pathogenesis of the myeloproliferative disorders is based on the assumption that they represent a clonal disorder resulting from transformation of a haematopoietic stem cell. Clonality assays exploit the fact that fem
Autor:
C. Gardin, Gilles Hetet, Jean Briere, Bernard Grandchamp, N. Elkassar, Jean-Jacques Kiladjian
Publikováno v:
Leukemia. 11:1821-1826
Essential thrombocythemia (ET) is a myeloproliferative disorder associated with megakaryocytic hyperplasia and thrombocytosis. In this disease, in vitro autonomous growth of megakaryocytic colonies has been demonstrated by various investigators. This
Publikováno v:
Baillière's Clinical Haematology. 10:65-88
Increased megakaryocyte (MK) proliferation in bone marrow is a feature common to the three Ph-negative myeloproliferative disorders (MPDs), i.e. essential thrombocythaemia (ET), polycythaemia vera (PV), and myelofibrosis with splenic myeloid metaplas
Publikováno v:
Journal of Laboratory and Clinical Medicine. 127:382-390
We have previously reported that platelet factor 4 (PF4) inhibits human erythroleukemic (HEL) cell growth in a dose-dependent fashion in vitro and that PF4 binds to HEL cells in a specific, saturable, and concentration-dependent manner. In this artic