Zobrazeno 1 - 10
of 43
pro vyhledávání: '"Jayashree Shanker"'
Autor:
Saikat Kanjilal, Jayashree Shanker, Veena S Rao, Natesha B Khadrinarasimhaih, Manjari Mukherjee, et al
Publikováno v:
Vascular Health and Risk Management, Vol Volume 4, Pp 189-197 (2008)
Saikat Kanjilal3, Jayashree Shanker1, Veena S Rao2, Natesha B Khadrinarasimhaih2, Manjari Mukherjee4, Shamanna S Iyengar3, Vijay V Kakkar1,51Mary and Garry Weston Functional Genomics Unit, Thrombosis Research Institute, Bangalore, India; 2Tata Proteo
Externí odkaz:
https://doaj.org/article/b054bef1660d40e980c3e2c9353fa52f
Autor:
Jayashree Shanker, Saikat Kanjilal, Veena S Rao, Ganapathy Perumal, Natesha B Khadrinarasimhiah, et al
Publikováno v:
Vascular Health and Risk Management, Vol Volume 3, Pp 1063-1068 (2008)
Jayashree Shanker1, Saikat Kanjilal3, Veena S Rao2, Ganapathy Perumal1, Natesha B Khadrinarasimhiah2, Manjari Mukherjee4, Shamanna S Iyengar3, Vijay V Kakkar51Mary and Garry Weston Functional Genomics Unit, Thrombosis Research Institute, Bangalore, I
Externí odkaz:
https://doaj.org/article/b637ee197ea3414ab4958cd4bdf9a9b8
Publikováno v:
PLoS ONE, Vol 9, Iss 4, p e94328 (2014)
Network analysis is a novel method to understand the complex pathogenesis of inflammation-driven atherosclerosis. Using this approach, we attempted to identify key inflammatory genes and their core transcriptional regulators in coronary artery diseas
Externí odkaz:
https://doaj.org/article/3a0b9c7a3b0342bbb66ca92e6f2e013e
Autor:
Rajani Kanth Vangala, Vandana Ravindran, Madan Ghatge, Jayashree Shanker, Prathima Arvind, Hima Bindu, Meghala Shekar, Veena S Rao
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e57193 (2013)
Patients with cardiovascular disease show a panel of differentially regulated serum biomarkers indicative of modulation of several pathways from disease onset to progression. Few of these biomarkers have been proposed for multimarker risk prediction
Externí odkaz:
https://doaj.org/article/37bffc5b4e9a4e1d9e293259b8a961b6
Autor:
Veena S. Rao, Natesha B. Kadarinarasimhiah, Shibu John, Sridhara Hebbagodi, Jayashree Shanker, Vijay V. Kakkar
Publikováno v:
International Journal of Vascular Medicine, Vol 2010 (2010)
Inflammation plays a pivotal role in all stages of atherosclerosis. Numerous inflammatory, lipid, and cytokines markers have been associated with coronary artery disease (CAD) risk but data directly comparing their predictive value are limited. Studi
Externí odkaz:
https://doaj.org/article/af73099306d64c1ba13fc700e6924455
Publikováno v:
The Indian Journal of Medical Research
Indian Journal of Medical Research, Vol 145, Iss 6, Pp 777-785 (2017)
Indian Journal of Medical Research, Vol 145, Iss 6, Pp 777-785 (2017)
Background & objectives: Metabolic syndrome (MetS) increases the likelihood of developing coronary artery disease (CAD), and inflammation is involved in the pathogenesis of both these conditions. The present work was conducted to examine the relative
Autor:
ARVIND, PRATHIMA1, JAYASHREE, SHANKER1 Jayashreeshanker@triindia.org.in, JAMBUNATHAN, SRIKARTHIKA1, NAIR, JINY1, KAKKAR, VIJAY V.1
Publikováno v:
Journal of Genetics. Dec2015, Vol. 94 Issue 4, p601-610. 10p.
Autor:
Jayashree Shanker, Vijay V. Kakkar
Publikováno v:
International Journal of Cardiology. 214:97-106
The merits and demerits of classical risk factors in coronary artery disease (CAD) are widely debated. We analyzed the role of conventional (age, gender, diabetes, hypertension, smoking) and non-conventional risk factors (anthropometrics, fasting blo
Publikováno v:
Journal of Genetics. Sep2015, Vol. 94 Issue 3, p539-549. 11p.
Autor:
Neha Gupta, Prathima Arvind, Velu Nair, Bhuvnesh Kumar, Tathagata Chatterjee, Babita Kumari, Jiny Nair, Mohammad Z. Ashraf, Anita Sahu, Prabhash Kumar Jha, Manish Sharma, Tarun Tyagi, Jayashree Shanker, Nitin Bajaj, Amit Prabhakar
Publikováno v:
Thrombosis and haemostasis. 118(7)
Venous thromboembolism (VTE), a multi-factorial disease, is the third most common cardiovascular disease. Established genetic and acquired risk factors are responsible for the onset of VTE. High altitude (HA) also poses as an additional risk factor,