Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Jay Ashokkumar Pandya"'
Autor:
Jay Ashokkumar Pandya, N Srikant, Karen Boaz, Nidhi Manaktala, Supriya Nikita Kapila, Shanmukha Raviteja Yinti
Publikováno v:
South Asian Journal of Cancer, Vol 3, Iss 3, Pp 159-162 (2014)
Introduction: Radiation, commonly employed as neoadjuvant, primary, and adjuvant therapy for head and neck cancer causes numerous epithelial and stromal changes, prominent among which is fibrosis with its early and late consequences. Very little is k
Externí odkaz:
https://doaj.org/article/03d07847112449469789b48b1c930d3d
Autor:
Supriya Nikita Kapila, Srikant Natarajan, Karen Boaz, Jay Ashokkumar Pandya, Shanmukha Raviteja Yinti
Publikováno v:
Journal of Clinical and Diagnostic Research, Vol 9, Iss 9, Pp ZC93-ZC97 (2015)
Introduction: Quicker decalcification is essential for faster diagnosis of hard tissue pathology. Heat and mechanical agitation are known to hasten decalcification. Aim: To compare the rate of decalcification, cellular and staining characteristic
Externí odkaz:
https://doaj.org/article/862ee0c3aff8450ea93d1b5d09edfdea
Publikováno v:
Journal of Cancer Research and Therapeutics, Vol 15, Iss 3, Pp 564-570 (2019)
Purpose: Epithelial cells exposed to carcinogens and genetic damage, once surpass reversible cell damage, either undergo apoptosis or transform into malignancy, chiefly oral squamous cell carcinoma (OSCC). Progressive accumulation of genetic errors i
Autor:
Karen Boaz, Nandita Kp, Srikant Natarajan, Jay Ashokkumar Pandya, Amitha J Lewis, Nidhi Manaktala
Publikováno v:
Journal of Cancer Research and Therapeutics, Vol 14, Iss 3, Pp 666-670 (2018)
Purpose: Oral epithelial dysplasia (OED) occurs on exposure of epithelial cells to carcinogens and genetic alteration. Once the reversible cell damage is surpassed, cells either undergo apoptosis or transform into malignancy, chiefly oral squamous ce
Publikováno v:
Journal of cancer research and therapeutics. 15(3)
Epithelial cells exposed to carcinogens and genetic damage, once surpass reversible cell damage, either undergo apoptosis or transform into malignancy, chiefly oral squamous cell carcinoma (OSCC). Progressive accumulation of genetic errors in TP53 re
Autor:
Jay Ashokkumar, Pandya, Karen, Boaz, Srikant, Natarajan, Nidhi, Manaktala, K P, Nandita, Amitha J, Lewis
Publikováno v:
Journal of cancer research and therapeutics. 14(3)
Oral epithelial dysplasia (OED) occurs on exposure of epithelial cells to carcinogens and genetic alteration. Once the reversible cell damage is surpassed, cells either undergo apoptosis or transform into malignancy, chiefly oral squamous cell carcin
Analysis of reticulin fiber pattern in lymph nodes with metastasis from oral squamous cell carcinoma
Autor:
Shanmukha Raviteja Yinti, Amitha J Lewis, Jay Ashokkumar Pandya, Supriya Nikita Kapila, Karen Boaz, Srikant Natarajan
Publikováno v:
Dental Hypotheses, Vol 6, Iss 3, Pp 104-108 (2015)
Introduction: The aim of the study was to determine the susceptibility of lymph nodes to metastasis in oral squamous cell carcinoma (OSCC) by analyzing the alterations in reticulin fiber pattern. Materials and Methods: This retrospective study on 30
Autor:
Karen Boaz, Supriya Nikita Kapila, Nidhi Manaktala, Jay Ashokkumar Pandya, Shanmukha Raviteja Yinti, Srikant N
Publikováno v:
South Asian Journal of Cancer
South Asian Journal of Cancer, Vol 3, Iss 3, Pp 159-162 (2014)
South Asian Journal of Cancer, Vol 3, Iss 3, Pp 159-162 (2014)
Introduction: Radiation, commonly employed as neoadjuvant, primary, and adjuvant therapy for head and neck cancer causes numerous epithelial and stromal changes, prominent among which is fibrosis with its early and late consequences. Very little is k
Autor:
Srikant Natarajan, Karen Boaz, Jay Ashokkumar Pandya, Shanmukha Raviteja Yinti, Supriya Nikita Kapila
Publikováno v:
Journal of Clinical and Diagnostic Research, Vol 9, Iss 9, Pp ZC93-ZC97 (2015)
Introduction: Quicker decalcification is essential for faster diagnosis of hard tissue pathology. Heat and mechanical agitation are known to hasten decalcification. Aim: To compare the rate of decalcification, cellular and staining characteristics of