Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Javier G Villamil-Ortiz"'
Publikováno v:
Actualidades Biológicas, Vol 34, Iss 96, Pp 172-172 (2012)
Externí odkaz:
https://doaj.org/article/3268a54764c24dfd92a49d9dae0013f8
Publikováno v:
Biochimica et biophysica acta. Molecular basis of disease. 1864(11)
Inflammation has been suggested to play early and perhaps causative roles in Alzheimer's disease (AD) pathogenesis possibly in part by the overactivation of the aspartic acid protease named β-site amyloid precursor protein (APP) cleaving enzyme 1 (B
Autor:
Alvaro Barrera-Ocampo, Francisco Lopera, Julián D. Arias-Londoño, Javier G. Villamil-Ortiz, Gloria Patricia Cardona-Gómez, Andrés Villegas
Publikováno v:
Journal of Alzheimer's disease : JAD. 61(1)
Lipids are considered important factors in the pathogenesis of Alzheimer's disease (AD). In this study, we realized a comparative analysis of the phospholipid profile and phospholipid composition of the temporal cortex from E280A-familiar AD (FAD), s
Autor:
Diego Piedrahita, Claudia M. Velásquez-Rodríguez, Javier G. Villamil-Ortiz, Julián D. Arias-Londoño, Gloria Patricia Cardona-Gómez, Alvaro Barrera-Ocampo
Publikováno v:
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Repositorio UdeA
Universidad de Antioquia
instacron:Universidad de Antioquia
Frontiers in Cellular Neuroscience, Vol 10 (2016)
Repositorio UdeA
Universidad de Antioquia
instacron:Universidad de Antioquia
β-amyloid (Aβ) is produced by the β-secretase 1 (BACE1)-mediated enzymatic cleavage of the amyloid precursor protein through the amyloidogenic pathway, making BACE1 a therapeutic target against Alzheimer’s disease (AD). Alterations in lipid meta
Publikováno v:
Repositorio UdeA
Universidad de Antioquia
instacron:Universidad de Antioquia
Frontiers in Aging Neuroscience
Frontiers in Aging Neuroscience, Vol 7 (2015)
Universidad de Antioquia
instacron:Universidad de Antioquia
Frontiers in Aging Neuroscience
Frontiers in Aging Neuroscience, Vol 7 (2015)
RESUMEN: La enfermedad de Alzheimer (AD) y la isquemia cerebral (CI) son neuropatologías que se caracterizan por agregados de proteína tau, un sello distintivo del trastorno cognitivo y la demencia. La acumulación de proteínas puede ser inducida
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9686d2f174f826a85b5a828b0f3672ca