Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Jason Winston"'
Autor:
Paul B. Perrin, Wanderimam Tuktur, David C. Wheeler, Jason Winston, Evanise Lexima, Briana Mezuk, Jacinda Fleming, Viktoryia A Kalesnikava, Joshua Montgomery, Tiffany L. Green
Publikováno v:
Psychosomatic Medicine. 82:658-668
Objective There are pronounced racial and socioeconomic disparities in type 2 diabetes. Although "stress" as a general phenomenon is hypothesized to contribute to these disparities, few studies have objective measures of stress reactivity in diverse
Autor:
Maria Jose Mejia Ruiz, Julie Ober Allen, Briana Mezuk, William Thornton, Jason Winston, Kim Nguyen, Jacinda Fleming, Jana Smith
Publikováno v:
Patient education and counseling. 105(5)
Objective To explore the perspectives of coaches and participant alumni of the YMCA Diabetes Control Program (DCP) to inform the development of a peer support component of the DCP for sustained diabetes self-management. Methods Coaches (n = 2) and al
Autor:
Jason Winston, Peter H. Watson, Stuart H. Yuspa, Barbara K. Vonderhaar, Michele Gunsior, Paul K. Goldsmith, Alif Dharamsi, Christopher Voscopoulos, Ronald Wolf, Melanie Olson
Publikováno v:
Cancer Letters. 277:101-107
Human S100A7 (psoriasin) is considered a marker for specific stages of breast cancer. hS100A15 is almost identical to hS100A7 and difficult to discriminate. We developed specific probes to distinguish hS100A7 and hS100A15, and demonstrate their diffe
Autor:
Joost J. Oppenheim, Hui-Fang Dong, O. M. Zack Howard, Rao Divi, Ronald Wolf, Paul K. Goldsmith, Michele Gunsior, Stuart H. Yuspa, Jason Winston, Karen M. Boeshans, Bijan Ahvazi, Christopher Voscopoulos, Triantafyllos Chavakis
Publikováno v:
The Journal of Immunology. 181:1499-1506
Human S100A7 (psoriasin) is overexpressed in inflammatory diseases. The recently discovered, co-evolved hS100A15 is almost identical in sequence and up-regulated with hS100A7 during cutaneous inflammation. The functional role of these closely related
Autor:
Jason Winston, Ronald Wolf
Publikováno v:
Journal of Dermatological Science. 67:205-207
Autor:
Christophe Cataisson, Ulrike Lichti, Stuart H. Yuspa, Triantafyllos Chavakis, Ronald Wolf, Jason Winston, Val Bliskovski, O. M. Zack Howard, Alif Dharamsi, Francesca Mascia, Lionel Feigenbaum, Thomas Ruzicka
Publikováno v:
Science Translational Medicine. 2
Psoriasis is a common complex genetic disease characterized by hyperplasia and inflammation in the skin; however, the relative contributions of epidermal cells and the immune system to disease pathogenesis remain unclear. Linkage studies have defined