Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Jason S. Groshong"'
Autor:
Bhupinder P.S. Vohra, Jason S. Groshong, Roberto Zayas, Robert L. Wollmann, Christopher M. Gomez
Publikováno v:
Neurobiology of Disease, Vol 23, Iss 2, Pp 462-470 (2006)
In the slow-channel syndrome (SCS) mutant acetylcholine receptors elicit calcium overload and myonuclear degeneration at the neuromuscular junction (NMJ), without muscle fiber death. Activated caspases are present at SCS motor endplates. We hypothesi
Externí odkaz:
https://doaj.org/article/15d370629a8b4762bd00c087599455cd
Autor:
Kate Kosmac, R. Grace Walton, Beibei Zhu, Brian S. Finlin, Philip A. Kern, Jyothi Mula, Jason S. Groshong, Bailey D. Peck, Charlotte A. Peterson, Christopher S. Fry
Publikováno v:
Scientific Reports, Vol 9, Iss 1, Pp 1-14 (2019)
Scientific Reports
Scientific Reports
Skeletal muscle macrophages participate in repair and regeneration following injury. However, their role in physiological adaptations to exercise is unexplored. We determined whether endurance exercise training (EET) alters macrophage content and cha
We present a robust, fresh-frozen approach to immunohistochemistry (IHC), without committing the tissue to IHC via fixation and cryopreservation while maintaining long-term storage, using LiCor-based infrared (IR) quantification for sensitive assessm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::095e7938ef1715564bfd53c7a371c412
https://doi.org/10.20944/preprints202012.0641.v1
https://doi.org/10.20944/preprints202012.0641.v1
Publikováno v:
J Chem Neuroanat
DNSP-11 antibody signal was investigated in perfusion fixated Fischer 344 rat brains by immunohistochemistry with a custom, affinity purified polyclonal antibody. The DNSP-11-antibody signal was differentially localized from the mature GDNF protein b
Autor:
Lisa J. Robinson, Vasu Punj, Jason S. Groshong, Preet M. Chaudhary, Hittu Matta, Anwaar Ahmad, Sandra Schamus, Parkash S. Gill
Publikováno v:
Cancer Biology & Therapy. 10:1033-1040
Primary effusion lymphoma (PEL) is an aggressive form of lymphoma that is associated with infection by Kaposi's sarcoma-associated herpesvirus (KSHV). One of the KSHV genes expressed in PEL cells is K13, a potent activator of the NF-κB pathway. K13
Autor:
Christopher S. Fry, Jason S. Groshong, Sami L. Michaelis, Charlotte A. Peterson, R. Grace Walton, Brian S. Finlin, Kevin A. Murach, Philip A. Kern
Publikováno v:
Physiological Reports
This investigation evaluated whether moderate‐intensity cycle ergometer training affects satellite cell and molecular responses to acute maximal concentric/eccentric resistance exercise in middle‐aged women. Baseline and 72 h postresistance exerc
Autor:
Yanqiang Yang, Hittu Matta, Han Yi, Preet M. Chaudhary, Jason S. Groshong, Ramakrishnan Gopalakrishnan
Publikováno v:
The Journal of biological chemistry. 286(32)
Myeloma cells are dependent on IL6 for their survival and proliferation during the early stages of disease, and independence from IL6 is associated with disease progression. The role of the NF-κB pathway in the IL6-independent growth of myeloma cell
Autor:
Richard J. Miller, Bhupinder P. S. Vohra, Elena Kudryashova, Bula J. Bhattacharyya, Roberto Zayas, Melissa J. Spencer, Jason S. Groshong, Robert L. Wollmann, Christopher M. Gomez
Publikováno v:
The Journal of clinical investigation. 117(10)
The slow-channel myasthenic syndrome (SCS) is a hereditary disorder of the acetylcholine receptor (AChR) of the neuromuscular junction (NMJ) that leads to prolonged AChR channel opening, Ca(2+) overload, and degeneration of the NMJ. We used an SCS tr
Autor:
Robert L. Wollmann, Bhupinder P. S. Vohra, Christopher M. Gomez, Jason S. Groshong, Roberto Zayas
Publikováno v:
Neurobiology of Disease, Vol 23, Iss 2, Pp 462-470 (2006)
In the slow-channel syndrome (SCS) mutant acetylcholine receptors elicit calcium overload and myonuclear degeneration at the neuromuscular junction (NMJ), without muscle fiber death. Activated caspases are present at SCS motor endplates. We hypothesi
Publikováno v:
Cell calcium. 41(4)
Strict control of calcium entry through excitatory synaptic receptors is important for shaping synaptic responses, gene expression, and cell survival. Disruption of this control may lead to pathological accumulation of Ca2+. The slow-channel congenit